Safety and efficacy of panitumumab in combination with trifluridine/tipiracil for pre-treated patients with unresectable, metastatic colorectal cancer with wild-type RAS: The phase 1/2 APOLLON study.


Journal

International journal of clinical oncology
ISSN: 1437-7772
Titre abrégé: Int J Clin Oncol
Pays: Japan
ID NLM: 9616295

Informations de publication

Date de publication:
Jul 2021
Historique:
received: 09 10 2020
accepted: 17 03 2021
pubmed: 1 5 2021
medline: 23 6 2021
entrez: 30 4 2021
Statut: ppublish

Résumé

We aimed to assess the safety and efficacy of combination treatment with panitumumab plus trifluridine/tipiracil (FTD/TPI) in patients with wild-type RAS metastatic colorectal cancer (mCRC) who were refractory/intolerant to standard therapies other than anti-epidermal growth factor receptor therapy. APOLLON was an open-label, multicentre, phase 1/2 trial. In the phase 1 part, 3 + 3 de-escalation design was used to investigate the recommended phase 2 dose (RP2D); all patients in the phase 2 part received the RP2D. The primary endpoint was investigator-assessed progression-free survival (PFS) rate at 6 months. Secondary endpoints included PFS, overall survival (OS), overall response rate (ORR), disease control rate (DCR), time to treatment failure (TTF), and safety. Fifty-six patients were enrolled (phase 1, n = 7; phase 2, n = 49) at 25 Japanese centres. No dose-limiting toxicities were observed in patients receiving panitumumab (6 mg/kg every 2 weeks) plus FTD/TPI (35 mg/m Panitumumab plus FTD/TPI exhibited favourable anti-tumour activity with a manageable safety profile and may be a therapeutic option for pre-treated mCRC patients.

Sections du résumé

BACKGROUND BACKGROUND
We aimed to assess the safety and efficacy of combination treatment with panitumumab plus trifluridine/tipiracil (FTD/TPI) in patients with wild-type RAS metastatic colorectal cancer (mCRC) who were refractory/intolerant to standard therapies other than anti-epidermal growth factor receptor therapy.
METHODS METHODS
APOLLON was an open-label, multicentre, phase 1/2 trial. In the phase 1 part, 3 + 3 de-escalation design was used to investigate the recommended phase 2 dose (RP2D); all patients in the phase 2 part received the RP2D. The primary endpoint was investigator-assessed progression-free survival (PFS) rate at 6 months. Secondary endpoints included PFS, overall survival (OS), overall response rate (ORR), disease control rate (DCR), time to treatment failure (TTF), and safety.
RESULTS RESULTS
Fifty-six patients were enrolled (phase 1, n = 7; phase 2, n = 49) at 25 Japanese centres. No dose-limiting toxicities were observed in patients receiving panitumumab (6 mg/kg every 2 weeks) plus FTD/TPI (35 mg/m
CONCLUSION CONCLUSIONS
Panitumumab plus FTD/TPI exhibited favourable anti-tumour activity with a manageable safety profile and may be a therapeutic option for pre-treated mCRC patients.

Identifiants

pubmed: 33928486
doi: 10.1007/s10147-021-01902-2
pii: 10.1007/s10147-021-01902-2
pmc: PMC8213662
doi:

Substances chimiques

Pyrrolidines 0
Panitumumab 6A901E312A
tipiracil NGO10K751P
Thymine QR26YLT7LT
Trifluridine RMW9V5RW38

Types de publication

Clinical Trial, Phase I Clinical Trial, Phase II Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

1238-1247

Références

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Auteurs

Takeshi Kato (T)

National Hospital Organization Osaka National Hospital, 2 Chome-1-14 Hoenzaka, Chuo Ward, Osaka, 540-0006, Japan.

Yoshinori Kagawa (Y)

Kansai Rosai Hospital, 3 Chome-1-69 Inabaso, Amagasaki, Hyogo, 660-8511, Japan.

Yasutoshi Kuboki (Y)

National Cancer Center Hospital East, 6 Chome-5-1 Kashiwanoha, Kashiwa, Chiba, 277-8577, Japan.

Makio Gamoh (M)

Osaki Citizen Hospital, Furukawa Honami, 3 Chome, Osaki, 989-6183, Japan.

Yoshito Komatsu (Y)

Hokkaido University Hospital, 5 Chome Kita 14 Jonishi, Kita Ward, Sapporo, Hokkaido, 060-8648, Japan.

Hirofumi Yasui (H)

Shizuoka Cancer Center, 1007 Shimonagakubo, Nagaizumi, Sunto District, Shizuoka, 411-0934, Japan.

Hironaga Satake (H)

Kobe City Medical Center General Hospital, 2 Chome-1-1 Minatojima Minamimachi, Chuo Ward, Kobe, Hyogo, 650-0047, Japan.
Cancer Treatment Center, Kansai Medical University Hospital, 2 Chome-3-1 Shinmachi, Hirakata, Osaka, 573-1191, Japan.

Eiji Oki (E)

Kyushu University, Maidashi 3 Chome-1-3, Higashi Ward, Fukuoka, 812-0053, Japan.

Hiroaki Tanioka (H)

Okayama Rosai Hospital, 1 Chome-10-25 Chikkomidorimachi, Minami Ward, Okayama, 702-8055, Japan.
Medical Oncology Kawasaki Medical School, 577 Matsushima, Kurashiki, Okayama, 701-0192, Japan.

Masahito Kotaka (M)

Sano Hospital, 2 Chome-5-1 Shimizugaoka, Tarumi Ward, Kobe, Hyogo, 655-0031, Japan.

Akitaka Makiyama (A)

Japan Community Healthcare Organization Kyushu Hospital, 1 Chome-8-1 Kishinoura, Yahatanishi Ward, Kitakyushu, Fukuoka, 806-8501, Japan.
Gifu University Hospital, 1-1 Yanagido, Gifu, 501-1194, Japan.

Tadamichi Denda (T)

Chiba Cancer Center, 666-2 Nitona-cho, Chuo Ward, Chiba, 260-8717, Japan.

Masahiro Goto (M)

Osaka Medical College Hospital, 2-7 Daigakumachi, Takatsuki, Osaka, 569-0096, Japan.

Takayuki Yoshino (T)

National Cancer Center Hospital East, 6 Chome-5-1 Kashiwanoha, Kashiwa, Chiba, 277-8577, Japan.

Kentaro Yamazaki (K)

Shizuoka Cancer Center, 1007 Shimonagakubo, Nagaizumi, Sunto District, Shizuoka, 411-0934, Japan.

Junpei Soeda (J)

Takeda Pharmaceutical Company, Ltd, Nihonbashi-Honcho 2 Chome-1-1, Chuo Ward, Tokyo, 103-8668, Japan.

Kazunori Shibuya (K)

Takeda Pharmaceutical Company, Ltd, Nihonbashi-Honcho 2 Chome-1-1, Chuo Ward, Tokyo, 103-8668, Japan.

Masaru Iwata (M)

Takeda Pharmaceutical Company, Ltd, Nihonbashi-Honcho 2 Chome-1-1, Chuo Ward, Tokyo, 103-8668, Japan.

Koji Oba (K)

University of Tokyo, 7 Chome-3-1 Hongo, Bunkyo, Tokyo, 113-8654, Japan.

Kensei Yamaguchi (K)

Gastroenterological Chemotherapy Department, Cancer Institute Hospital of Japanese Foundation for Cancer Research, 3 Chome-8-31, Ariake, Koto, Tokyo, 135-8550, Japan. kensei.yamaguchi@jfcr.or.jp.

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