Effects of the PCSK9 antibody alirocumab on coronary atherosclerosis in patients with acute myocardial infarction: a serial, multivessel, intravascular ultrasound, near-infrared spectroscopy and optical coherence tomography imaging study-Rationale and design of the PACMAN-AMI trial.


Journal

American heart journal
ISSN: 1097-6744
Titre abrégé: Am Heart J
Pays: United States
ID NLM: 0370465

Informations de publication

Date de publication:
08 2021
Historique:
received: 16 11 2020
accepted: 26 04 2021
pubmed: 6 5 2021
medline: 27 8 2021
entrez: 5 5 2021
Statut: ppublish

Résumé

The risk for cardiovascular adverse events after acute myocardial infarction (AMI) remains high despite potent medical treatment including low-density lipoprotein cholesterol (LDL-C) lowering with statins. Proprotein convertase subtilisin/kexin type 9 (PCSK9) antibodies substantially reduce LDL-C when added to statin. Alirocumab, a monoclonal antibody to PCSK9, reduces major adverse cardiovascular events after AMI. The effects of alirocumab on coronary atherosclerosis including plaque burden, plaque composition and fibrous cap thickness in patients presenting with AMI remains unknown. To determine the effect of LDL-C lowering with alirocumab on top of high-intensity statin therapy on intravascular ultrasound (IVUS)-derived percent atheroma volume (PAV), near-infrared spectroscopy (NIRS)-derived maximum lipid core burden index within 4 mm (maxLCBI In this multicenter, double-blind, placebo-controlled trial, 300 patients with AMI (ST-elevation or non-ST-elevation myocardial infarction) were randomly assigned to receive either biweekly subcutaneous alirocumab (150 mg) or placebo beginning <24 hours after the acute event as add-on therapy to rosuvastatin 20 mg. Patients undergo serial IVUS, NIRS and OCT in the two non-infarct related arteries at baseline (at the time of treatment of the culprit lesion) and at 52 weeks. The primary endpoint, change in IVUS-derived PAV, and the powered secondary endpoints, change in NIRS-derived maxLCBI The PACMAN-AMI trial will determine the effect of alirocumab on top of high-intensity statin therapy on high-risk coronary plaque characteristics as assessed by serial, multimodality intracoronary imaging in patients presenting with AMI. NCT03067844.

Sections du résumé

BACKGROUND
The risk for cardiovascular adverse events after acute myocardial infarction (AMI) remains high despite potent medical treatment including low-density lipoprotein cholesterol (LDL-C) lowering with statins. Proprotein convertase subtilisin/kexin type 9 (PCSK9) antibodies substantially reduce LDL-C when added to statin. Alirocumab, a monoclonal antibody to PCSK9, reduces major adverse cardiovascular events after AMI. The effects of alirocumab on coronary atherosclerosis including plaque burden, plaque composition and fibrous cap thickness in patients presenting with AMI remains unknown.
AIMS
To determine the effect of LDL-C lowering with alirocumab on top of high-intensity statin therapy on intravascular ultrasound (IVUS)-derived percent atheroma volume (PAV), near-infrared spectroscopy (NIRS)-derived maximum lipid core burden index within 4 mm (maxLCBI
METHODS
In this multicenter, double-blind, placebo-controlled trial, 300 patients with AMI (ST-elevation or non-ST-elevation myocardial infarction) were randomly assigned to receive either biweekly subcutaneous alirocumab (150 mg) or placebo beginning <24 hours after the acute event as add-on therapy to rosuvastatin 20 mg. Patients undergo serial IVUS, NIRS and OCT in the two non-infarct related arteries at baseline (at the time of treatment of the culprit lesion) and at 52 weeks. The primary endpoint, change in IVUS-derived PAV, and the powered secondary endpoints, change in NIRS-derived maxLCBI
SUMMARY
The PACMAN-AMI trial will determine the effect of alirocumab on top of high-intensity statin therapy on high-risk coronary plaque characteristics as assessed by serial, multimodality intracoronary imaging in patients presenting with AMI.
CLINICAL TRIAL REGISTRATION
NCT03067844.

Identifiants

pubmed: 33951415
pii: S0002-8703(21)00108-3
doi: 10.1016/j.ahj.2021.04.006
pii:
doi:

Substances chimiques

Antibodies, Monoclonal, Humanized 0
Cholesterol, LDL 0
Hydroxymethylglutaryl-CoA Reductase Inhibitors 0
Placebos 0
Rosuvastatin Calcium 83MVU38M7Q
PCSK9 protein, human EC 3.4.21.-
Proprotein Convertase 9 EC 3.4.21.-
alirocumab PP0SHH6V16

Banques de données

ClinicalTrials.gov
['NCT03067844']

Types de publication

Journal Article Multicenter Study Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

33-44

Informations de copyright

Copyright © 2021. Published by Elsevier Inc.

Auteurs

Christian Zanchin (C)

Department of Cardiology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.

Konstantinos C Koskinas (KC)

Department of Cardiology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.

Yasushi Ueki (Y)

Department of Cardiology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.

Sylvain Losdat (S)

Institute of Social and Preventive Medicine and Clinical Trials Unit, Bern University Hospital, Bern, Switzerland.

Jonas D Häner (JD)

Department of Cardiology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.

Sarah Bär (S)

Department of Cardiology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.

Tatsuhiko Otsuka (T)

Department of Cardiology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.

Andrea Inderkum (A)

Department of Cardiology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.

Maria Radu Juul Jensen (MRJ)

Department of Cardiology, Rigshospitalet, Copenhagen University Hospital, Copenhagen, Denmark.

Jacob Lonborg (J)

Department of Cardiology, Rigshospitalet, Copenhagen University Hospital, Copenhagen, Denmark.

Gregor Fahrni (G)

Department of Cardiology, University Hospital Basel, Basel, Switzerland.

Anna S Ondracek (AS)

Department of Cardiology, Medical University of Vienna, Vienna, Austria.

Joost Daemen (J)

Department of Cardiology, Erasmus Medical Center, Rotterdam, The Netherlands.

Robert-Jan van Geuns (RJ)

Department of Cardiology, Radboud UMC, Nijmegen, The Netherlands.

Juan F Iglesias (JF)

Department of Cardiology, Geneva University Hospital, Geneva, Switzerland.

Christian M Matter (CM)

Department of Cardiology, University Heart Center Zurich, University Hospital Zurich, Zurich, Switzerland.

David Spirk (D)

Department of Pharmacology, Bern University Hospital, Bern, Switzerland and Sanofi, Switzerland.

Peter Juni (P)

Department of Medicine and Institute of Health Policy, Management and Evaluation, University of Toronto, Toronto, Ontario, Canada.

Francois Mach (F)

Department of Cardiology, Geneva University Hospital, Geneva, Switzerland.

Dik Heg (D)

Institute of Social and Preventive Medicine and Clinical Trials Unit, Bern University Hospital, Bern, Switzerland.

Thomas Engstrom (T)

Department of Cardiology, Rigshospitalet, Copenhagen University Hospital, Copenhagen, Denmark.

Irene Lang (I)

Department of Cardiology, Medical University of Vienna, Vienna, Austria.

Stephan Windecker (S)

Department of Cardiology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.

Lorenz Räber (L)

Department of Cardiology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland. Electronic address: lorenz.raeber@insel.ch.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH