Translational investigation of electrophysiology in hypertrophic cardiomyopathy.
Action Potentials
/ drug effects
Animals
Biomarkers
Calcium
/ metabolism
Cardiomyopathy, Hypertrophic
/ diagnosis
Carrier Proteins
/ genetics
Disease Models, Animal
Disease Susceptibility
Electrophysiology
Gene Expression
Humans
Induced Pluripotent Stem Cells
/ metabolism
Mice
Mice, Knockout
Myocardial Contraction
/ drug effects
Myocardium
/ metabolism
Myocytes, Cardiac
/ cytology
Potassium
/ metabolism
Potassium Channels
/ genetics
Translational Research, Biomedical
Action potential
Cardiac arrhythmia
Engineered heart tissue
Human tissue
Hypertrophic cardiomyopathy
Mouse model
Sudden cardiac death
Journal
Journal of molecular and cellular cardiology
ISSN: 1095-8584
Titre abrégé: J Mol Cell Cardiol
Pays: England
ID NLM: 0262322
Informations de publication
Date de publication:
08 2021
08 2021
Historique:
received:
10
03
2021
revised:
14
04
2021
accepted:
29
04
2021
pubmed:
7
5
2021
medline:
11
1
2022
entrez:
6
5
2021
Statut:
ppublish
Résumé
Hypertrophic cardiomyopathy (HCM) patients are at increased risk of ventricular arrhythmias and sudden cardiac death, which can occur even in the absence of structural changes of the heart. HCM mouse models suggest mutations in myofilament components to affect Ca
Identifiants
pubmed: 33957110
pii: S0022-2828(21)00088-2
doi: 10.1016/j.yjmcc.2021.04.009
pmc: PMC8320769
pii:
doi:
Substances chimiques
Biomarkers
0
Carrier Proteins
0
Potassium Channels
0
myosin-binding protein C
0
Potassium
RWP5GA015D
Calcium
SY7Q814VUP
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
77-89Informations de copyright
Copyright © 2021 The Authors. Published by Elsevier Ltd.. All rights reserved.
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