Early Initiation of Antiretroviral Therapy Following In Utero HIV Infection Is Associated With Low Viral Reservoirs but Other Factors Determine Viral Rebound.


Journal

The Journal of infectious diseases
ISSN: 1537-6613
Titre abrégé: J Infect Dis
Pays: United States
ID NLM: 0413675

Informations de publication

Date de publication:
01 12 2021
Historique:
received: 24 11 2020
accepted: 29 04 2021
pubmed: 9 5 2021
medline: 3 2 2022
entrez: 8 5 2021
Statut: ppublish

Résumé

Early HIV diagnosis allows combination antiretroviral therapy (cART) initiation in the first days of life following in utero (IU) infection. The impact of early cART initiation on infant viral reservoir size in the setting of high-frequency cART nonadherence is unknown. Peripheral blood total HIV DNA from 164 early treated (day 0-21 of life) IU HIV-infected South African infants was measured using droplet digital PCR at birth and following suppressive cART. We evaluated the impact of cART initiation timing on HIV reservoir size and decay, and on the risk of subsequent plasma viremia in cART-suppressed infants. Baseline HIV DNA (median 2.8 log10 copies/million peripheral blood mononuclear cells, range 0.7-4.8) did not correlate with age at cART initiation (0-21 days) but instead with maternal antenatal cART use. In 98 infants with plasma viral suppression on cART, HIV DNA half-life was 28 days. However, the probability of maintenance of plasma aviremia was low (0.46 at 12 months) and not influenced by HIV DNA load. Unexpectedly, longer time to viral suppression was associated with protection against subsequent viral rebound. With effective prophylaxis against mother-to-child transmission, cART initiation timing in the first 3 weeks of life is not critical to reservoir size.

Sections du résumé

BACKGROUND
Early HIV diagnosis allows combination antiretroviral therapy (cART) initiation in the first days of life following in utero (IU) infection. The impact of early cART initiation on infant viral reservoir size in the setting of high-frequency cART nonadherence is unknown.
METHODS
Peripheral blood total HIV DNA from 164 early treated (day 0-21 of life) IU HIV-infected South African infants was measured using droplet digital PCR at birth and following suppressive cART. We evaluated the impact of cART initiation timing on HIV reservoir size and decay, and on the risk of subsequent plasma viremia in cART-suppressed infants.
RESULTS
Baseline HIV DNA (median 2.8 log10 copies/million peripheral blood mononuclear cells, range 0.7-4.8) did not correlate with age at cART initiation (0-21 days) but instead with maternal antenatal cART use. In 98 infants with plasma viral suppression on cART, HIV DNA half-life was 28 days. However, the probability of maintenance of plasma aviremia was low (0.46 at 12 months) and not influenced by HIV DNA load. Unexpectedly, longer time to viral suppression was associated with protection against subsequent viral rebound.
CONCLUSIONS
With effective prophylaxis against mother-to-child transmission, cART initiation timing in the first 3 weeks of life is not critical to reservoir size.

Identifiants

pubmed: 33963757
pii: 6272368
doi: 10.1093/infdis/jiab223
pmc: PMC8643423
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1925-1934

Subventions

Organisme : NIAID NIH HHS
ID : R01 AI133673
Pays : United States
Organisme : Wellcome Trust
Pays : United Kingdom
Organisme : Wellcome Trust
ID : WT104748MA
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 110110/Z/15/Z
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 107752/Z/15/Z
Pays : United Kingdom

Informations de copyright

© The Author(s) 2021. Published by Oxford University Press for the Infectious Diseases Society of America.

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Auteurs

Jane R Millar (JR)

HIV Pathogenesis Programme, The Doris Duke Medical Research Institute, University of KwaZulu-Natal, Durban, South Africa.
Department of Paediatrics, University of Oxford, Oxford, United Kingdom.

Nomonde Bengu (N)

Umkhuseli Innovation and Research Management, Pietermaritzburg, South Africa.

Vinicius A Vieira (VA)

Department of Paediatrics, University of Oxford, Oxford, United Kingdom.

Emily Adland (E)

Department of Paediatrics, University of Oxford, Oxford, United Kingdom.

Julia Roider (J)

HIV Pathogenesis Programme, The Doris Duke Medical Research Institute, University of KwaZulu-Natal, Durban, South Africa.
Department of Paediatrics, University of Oxford, Oxford, United Kingdom.
Africa Health Research Institute, Durban, South Africa.
German Center for Infection Research, Munich,Germany.
Department of Infectious Diseases, Ludwig Maximilian University, Munich, Germany.

Maximilian Muenchhoff (M)

German Center for Infection Research, Munich,Germany.
Max von Pettenkofer Institute, Virology, National Reference Center for Retroviruses, Faculty of Medicine, Ludwig Maximilian University, Munich, Germany.

Rowena Fillis (R)

Umkhuseli Innovation and Research Management, Pietermaritzburg, South Africa.

Kenneth Sprenger (K)

Umkhuseli Innovation and Research Management, Pietermaritzburg, South Africa.

Vuyokazi Ntlantsana (V)

School of Clinical Medicine, University of KwaZulu-Natal, Durban, South Africa.

Isabella Fatti (I)

Umkhuseli Innovation and Research Management, Pietermaritzburg, South Africa.

Moherndran Archary (M)

Department of Paediatrics, King Edward VIII Hospital/University of KwaZulu-Natal, Durban, South Africa.

Andreas Groll (A)

Technical University Dortmund, Department of Statistics, Dortmund, Germany.

Nasreen Ismail (N)

HIV Pathogenesis Programme, The Doris Duke Medical Research Institute, University of KwaZulu-Natal, Durban, South Africa.

Maria C García-Guerrero (MC)

IrsiCaixa AIDS Research Institute, Badalona, Spain.

Philippa C Matthews (PC)

Nuffield Department of Medicine, University of Oxford, Oxford, United Kingdom.
Department of Microbiology and Infectious Diseases, Oxford University Hospitals NHS Foundation Trust, Oxford, United Kingdom.
Oxford Biomedical Research Centre, John Radcliffe Hospital, Oxford, United Kingdom.

Thumbi Ndung'u (T)

HIV Pathogenesis Programme, The Doris Duke Medical Research Institute, University of KwaZulu-Natal, Durban, South Africa.
Africa Health Research Institute, Durban, South Africa.
Ragon Institute of Massachusetts General Hospital, Massachusetts Institute of Technology, and Harvard, Cambridge, Massachusetts, USA.
Max Planck Institute for Infection Biology, Berlin, Germany.
Division of Infection and Immunity, University College London, London, United Kingdom.

Maria C Puertas (MC)

IrsiCaixa AIDS Research Institute, Badalona, Spain.

Javier Martinez-Picado (J)

IrsiCaixa AIDS Research Institute, Badalona, Spain.
University of Vic-Central University of Catalonia, Vic, Spain.
Catalan Institution for Research and Advanced Studies, Barcelona, Spain.
Germans Trias i Pujol Research Institute, Badalona, Spain.

Philip Goulder (P)

HIV Pathogenesis Programme, The Doris Duke Medical Research Institute, University of KwaZulu-Natal, Durban, South Africa.
Department of Paediatrics, University of Oxford, Oxford, United Kingdom.
Ragon Institute of Massachusetts General Hospital, Massachusetts Institute of Technology, and Harvard, Cambridge, Massachusetts, USA.

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