Optic chiasm involvement in AQP-4 antibody-positive NMO and MOG antibody-associated disorder.


Journal

Multiple sclerosis (Houndmills, Basingstoke, England)
ISSN: 1477-0970
Titre abrégé: Mult Scler
Pays: England
ID NLM: 9509185

Informations de publication

Date de publication:
01 2022
Historique:
pubmed: 13 5 2021
medline: 28 1 2022
entrez: 12 5 2021
Statut: ppublish

Résumé

Optic neuritis (ON) is often the presenting symptom in inflammatory central nervous system demyelinating disorders. To compare the frequency and pattern of optic chiasm involvement in patients with aquaporin-4-immunoglobulin G (AQP4-IgG)-associated ON to patients with myelin oligodendrocyte glycoprotein-immunoglobulin G (MOG-IgG)-associated ON. Retrospective review of all patients evaluated at Mayo Clinic, Stanford University and Ramathibodi Hospital who were found to have: (1) ON, (2) either MOG-IgG or AQP4-IgG by cell-based assay, and (3) magnetic resonance imaging (MRI) at the time of ON. MRI was reviewed for contrast enhancement of the optic chiasm and the pattern of involvement. One hundred and fifty-four patients (74 AQP4-IgG and 80 MOG-IgG) were included. Among patients with AQP4-IgG-ON, 20% had chiasmal involvement, compared with 16% of patients with MOG-IgG-ON ( Chiasmal involvement of MOG-IgG-ON and AQP4-IgG-ON occur at more similar frequencies than previously reported. Furthermore, MOG-IgG-ON chiasmal involvement is more likely to be part of a longitudinally extensive optic nerve lesion.

Sections du résumé

BACKGROUND
Optic neuritis (ON) is often the presenting symptom in inflammatory central nervous system demyelinating disorders.
OBJECTIVE
To compare the frequency and pattern of optic chiasm involvement in patients with aquaporin-4-immunoglobulin G (AQP4-IgG)-associated ON to patients with myelin oligodendrocyte glycoprotein-immunoglobulin G (MOG-IgG)-associated ON.
METHODS
Retrospective review of all patients evaluated at Mayo Clinic, Stanford University and Ramathibodi Hospital who were found to have: (1) ON, (2) either MOG-IgG or AQP4-IgG by cell-based assay, and (3) magnetic resonance imaging (MRI) at the time of ON. MRI was reviewed for contrast enhancement of the optic chiasm and the pattern of involvement.
RESULTS
One hundred and fifty-four patients (74 AQP4-IgG and 80 MOG-IgG) were included. Among patients with AQP4-IgG-ON, 20% had chiasmal involvement, compared with 16% of patients with MOG-IgG-ON (
CONCLUSION
Chiasmal involvement of MOG-IgG-ON and AQP4-IgG-ON occur at more similar frequencies than previously reported. Furthermore, MOG-IgG-ON chiasmal involvement is more likely to be part of a longitudinally extensive optic nerve lesion.

Identifiants

pubmed: 33975499
doi: 10.1177/13524585211011450
pmc: PMC8586045
mid: NIHMS1690068
doi:

Substances chimiques

Aquaporin 4 0
Autoantibodies 0
Myelin-Oligodendrocyte Glycoprotein 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

149-153

Subventions

Organisme : NEI NIH HHS
ID : P30 EY026877
Pays : United States
Organisme : NINDS NIH HHS
ID : R01 NS113828
Pays : United States

Références

Brain. 2017 Dec 1;140(12):3128-3138
pubmed: 29136091
Am J Ophthalmol. 2018 Nov;195:8-15
pubmed: 30055153
Br J Ophthalmol. 2018 Oct;102(10):1372-1377
pubmed: 29363529
Neurology. 2018 May 22;90(21):e1858-e1869
pubmed: 29695592
Curr Neurol Neurosci Rep. 2019 Nov 26;19(12):100
pubmed: 31773369
J Neuroinflammation. 2018 May 3;15(1):134
pubmed: 29724224
Mult Scler. 2016 Apr;22(4):470-82
pubmed: 26163068
J Neurol Neurosurg Psychiatry. 2016 Apr;87(4):446-8
pubmed: 25749692

Auteurs

Deena Tajfirouz (D)

Department of Neurology, Mayo Clinic, Rochester, MN, USA.

Tanyatuth Padungkiatsagul (T)

Department of Ophthalmology, Faculty of Medicine Ramathibodi Hospital, Mahidol University, Bangkok, Thailand/Department of Ophthalmology, Stanford University, Palo Alto, CA, USA.

Shannon Beres (S)

Department of Ophthalmology, Stanford University, Palo Alto, CA, USA/Department of Neurology and Neurological Sciences, Stanford University, Palo Alto, CA, USA.

Heather E Moss (HE)

Department of Ophthalmology, Stanford University, Palo Alto, CA, USA/Department of Neurology and Neurological Sciences, Stanford University, Palo Alto, CA, USA.

Sean Pittock (S)

Department of Neurology, Mayo Clinic, Rochester, MN, USA/Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA/Center for MS and Autoimmune Neurology, Mayo Clinic, Rochester, MN, USA.

Eoin Flanagan (E)

Department of Neurology, Mayo Clinic, Rochester, MN, USA/Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA/Center for MS and Autoimmune Neurology, Mayo Clinic, Rochester, MN, USA.

Amy Kunchok (A)

Mellen Center for Multiple Sclerosis Treatment and Research, Neurological Institute, Cleveland Clinic, Cleveland, Ohio.

Shailee Shah (S)

Department of Neurology, Mayo Clinic, Rochester, MN, USA/Center for MS and Autoimmune Neurology, Mayo Clinic, Rochester, MN, USA.

M Tariq Bhatti (MT)

Department of Neurology, Mayo Clinic, Rochester, MN, USA/Department of Ophthalmology, Mayo Clinic, Rochester, MN, USA.

John J Chen (JJ)

Department of Neurology, Mayo Clinic, Rochester, MN, USA/Department of Ophthalmology, Mayo Clinic, Rochester, MN, USA.

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Classifications MeSH