Asymmetric dimethylarginine: Is it a risk facgtor in the repair of aortic coarctation?

asymmetric dimethylarginine coarctation of aorta endotelial dysfunction morbidity mortality

Journal

Journal of cardiac surgery
ISSN: 1540-8191
Titre abrégé: J Card Surg
Pays: United States
ID NLM: 8908809

Informations de publication

Date de publication:
Aug 2021
Historique:
revised: 08 03 2021
received: 12 11 2020
accepted: 14 03 2021
pubmed: 17 5 2021
medline: 14 7 2021
entrez: 16 5 2021
Statut: ppublish

Résumé

Asymmetric dimethylarginine (ADMA), an endogenous inhibitor of nitric oxide synthase, causes endothelial dysfunction which is an important risk factor for mortality in adult cardiovascular diseases. We aimed to investigate whether there was a relationship between the aortic cross-clamping (ACC) time and serum ADMA level in aortic coarctation surgery and importance of the serum ADMA level as a marker of mortality and morbidity. Twenty-two patients undergoing surgery for aortic coarctation in the neonatal and early infant period were included in the study, and the patients were divided into two groups according to the aortic cross-clamping time (Group I: <20-min, Group II: >20-min). In Group I, preoperative and postoperative mean ADMA values of the patients were 0.57 ± 0.78 and 0.54 ± 0.83 µmol/L, respectively. In Group II, preoperative and postoperative mean ADMA values of the patients were 0.69 ± 0.93 and 1.10 ± 0.30 µmol/L, respectively. Preoperative-postoperative change of ADMA correlates with ACC time (r = .802, p < .005) and duration of postoperative inotropic support (r = .719, p < .05). Also a high correlation has been found between the ACC time and duration of inotropic support in both groups (r = .689, p < .05). Perioperative serum ADMA levels could be used as a prognostic bio-marker in the patients undergoing aortic coarctation repair. Treatments to reduce serum ADMA levels can be valuable for preventing mortality and morbidity which develop after surgeries in a transient ischemia setting by clamping the aorta.

Sections du résumé

BACKGROUND BACKGROUND
Asymmetric dimethylarginine (ADMA), an endogenous inhibitor of nitric oxide synthase, causes endothelial dysfunction which is an important risk factor for mortality in adult cardiovascular diseases. We aimed to investigate whether there was a relationship between the aortic cross-clamping (ACC) time and serum ADMA level in aortic coarctation surgery and importance of the serum ADMA level as a marker of mortality and morbidity.
METHOD METHODS
Twenty-two patients undergoing surgery for aortic coarctation in the neonatal and early infant period were included in the study, and the patients were divided into two groups according to the aortic cross-clamping time (Group I: <20-min, Group II: >20-min).
RESULTS RESULTS
In Group I, preoperative and postoperative mean ADMA values of the patients were 0.57 ± 0.78 and 0.54 ± 0.83 µmol/L, respectively. In Group II, preoperative and postoperative mean ADMA values of the patients were 0.69 ± 0.93 and 1.10 ± 0.30 µmol/L, respectively. Preoperative-postoperative change of ADMA correlates with ACC time (r = .802, p < .005) and duration of postoperative inotropic support (r = .719, p < .05). Also a high correlation has been found between the ACC time and duration of inotropic support in both groups (r = .689, p < .05).
CONCLUSION CONCLUSIONS
Perioperative serum ADMA levels could be used as a prognostic bio-marker in the patients undergoing aortic coarctation repair. Treatments to reduce serum ADMA levels can be valuable for preventing mortality and morbidity which develop after surgeries in a transient ischemia setting by clamping the aorta.

Identifiants

pubmed: 33993556
doi: 10.1111/jocs.15601
doi:

Substances chimiques

N,N-dimethylarginine 63CV1GEK3Y
Arginine 94ZLA3W45F
Nitric Oxide Synthase EC 1.14.13.39

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2735-2740

Informations de copyright

© 2021 Wiley Periodicals LLC.

Références

Gokce N , Keaney JF Jr , Hunter LM , et al. Predictive value of noninvasively determined endothelial dysfunction for long-term cardiovascular events in patients with peripheral vascular disease. J Am Coll Cardiol. 2003;41(10):1769-1775. http://doi.org/10.1016/s0735-1097(03)00333-4
Cooke JP . Asymmetrical dimethylarginine Circulation. 2004;109(15):1813-1818. http://doi.org/10.1161/01.cir.0000126823.07732.d5
Vallance P , Leone A , Calver A , Collier J , Moncada S . Endogenous dimethylarginine as an inhibitor of nitric oxide synthesis. J Cardiovasc Pharmacol. 1992;20:S60-S62. http://doi.org/10.1097/00005344-199204002-00018
Blackwell S . The biochemistry, measurement and current clinical significance of asymmetric dimethylarginine. Ann Clin Biochem. 2010;47:17-28.
García-Villalón AL , Amezquita YM , Monge L , Fernández N , Salcedo A , Diegues G . Endothelin −1 potention of coronary artery contraction after ischemia-reperfusion. Vascul Pharmacol. 2008;48:109-114.
Koch A , Weiskirchen R , Kunze J , et al. Elevated asymmetric dimethylarginine levels predict short-and long-term mortality risk in critically ill patients. J Crit Care. 2013;28(6):947-953. http://doi.org/10.1016/j.jcrc.2013.05.016
Stühlinger MC , Tsao PS , Her JH , Kimoto M , Balint RF , Cooke JP . Homocysteine impairs the nitric oxide synthase pathway. Circulation. 2001;104(21):2569-2575. http://doi.org/10.1161/hc4601.098514
Jang JJ , Ho HKV , Kwan HH , Fajardo LF , Cooke JP . Angiogenesis is impaired by hypercholesterolemia. Circulation. 2000;102(12):1414-1419. http://doi.org/10.1161/01.cir.102.12.1414
Lenzen H , Tsikas D , Bögerv RH . Asymmetric dimethylarginine (ADMA) and the risk for coronary heart disease: the multicenter cardiac study. Eur J Clin Pharmacol. 2006;62:45-49.
Winkler MS , Kluge S , Holzmann M , et al. Markers of nitric oxide are associated with sepsis severity: an observational study. Crit Care. 2017;21(1). http://doi.org/10.1186/s13054-017-1782-2
St. Louis JD , Harvey BA , Menk JS , O′Brien Jr. JE , Kochilas LK . Mortality and operative management for patients undergoing repair of coarctation of the aorta. World J Pediatr Congenital Heart Surg. 2015;6(3):431-437. http://doi.org/10.1177/2150135115590458
Cziráki A , Ajtay Z , Németh Á , et al. Effects of coronary revascularization with or without cardiopulmonary bypass on plasma levels of asymmetric dimethylarginine. Coron Artery Dis. 2011;22(4):245-252. http://doi.org/10.1097/mca.0b013e3283441d5c
Selçuk M. T. , Selçuk H. , Temizhan A. , et al. The effect of plasma asymmetric dimethylarginine (ADMA) level and L-arginine/ADMA ratio on the development of coronary collaterals. Türk Kardiyoloji Derneği Arşivi. 2008;36(3):150-155.
Kocaman SA , Sahinarslan A , Biberoglu G , et al. Asymmetric dimethylarginine and coronary collateral vessel development. Coron Artery Dis. 2008;19(7):469-474. http://doi.org/10.1097/mca.0b013e328311d32b
Mizia-Stec K , Trojnarska O , Szczepaniak-Chicheł L , et al. Asymmetric dimethylarginine and vascular indices of atherosclerosis in patients after coarctation of aorta repair. Int J Cardiol. 2012;158(3):364-369.
Nijveldt RJ , Siroen MPC , Teerlink T , Leeuwen PAM . Elimination of asymmetric dimethylarginine by the kidney and the liver: a link to the development of multiple organ failure. Am Soc Nutr Sci. 2004;42:2848-2852.
Böger RH , Ron ES . L-arginine improves vascular function by overcoming deleterious effects of ADMA, a novel cardiovascular risk factor. Altern Med Rev. 2005;10:14-23.
Lu TM , Ding YA , Lin SJ , Lee WS , Tai HC . Plasma levels of asymmetrical dimethylarginine and adverse cardiovascular events after percutaneous coronary intervention. Eur Heart J. 2003;24(21):1912-1919. http://doi.org/10.1016/j.ehj.2003.08.013
Böger RH , Bode-Böger SM , Sydow K , Heistad DD , Lentz SR . Plasma concentration of asymmetric dimethylarginine, an endogenous inhibitor of nitric oxide synthase, is elevated in monkeys with hyperhomocyst (e) inemia or hypercholesterolemia. Arterioscler Thromb Vasc Biol. 2000;20(6):1557-1564.
Önem G , Akbulut M , Baltalarlı A , et al. Oral L-arginine reduces lung injury following reperfusion after ischemia in the lower torso of rats. Turk Gogus Kalp Dama. 2006;14:133-137.

Auteurs

Tolga Baş (T)

Department of Cardiovascular Surgery, Istanbul Kartal Koşuyolu High Specialization Research and Training Hospital, University of Health Sciences, Istanbul, Turkey.

Murat Koç (M)

Department of Cardiovascular Surgery, Ankara Dr. Sami Ulus Obstetrics, Gynecology and Pediatrics Research and Training Hospital, University of Health Sciences, Ankara, Turkey.

Onur Işık (O)

Department of Congenital Heart Surgery, Tepecik Research and Training Hospital, University of Health Sciences, Izmir, Turkey.

Hakan Hançer (H)

Department of Cardiovascular Surgery, Istanbul Kartal Koşuyolu High Specialization Research and Training Hospital, University of Health Sciences, Istanbul, Turkey.

Ali Kutsal (A)

Department of Cardiovascular Surgery, Ankara Dr. Sami Ulus Obstetrics, Gynecology and Pediatrics Research and Training Hospital, University of Health Sciences, Ankara, Turkey.

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Classifications MeSH