Matrix metalloproteinase MMP-8, TIMP-1 and MMP-8/TIMP-1 ratio in plasma in methicillin-sensitive Staphylococcus aureus bacteremia.


Journal

PloS one
ISSN: 1932-6203
Titre abrégé: PLoS One
Pays: United States
ID NLM: 101285081

Informations de publication

Date de publication:
2021
Historique:
received: 01 11 2020
accepted: 10 05 2021
entrez: 27 5 2021
pubmed: 28 5 2021
medline: 25 2 2023
Statut: epublish

Résumé

Matrix metalloproteinase-8 (MMP-8) and tissue inhibitor of metalloproteinases-1 (TIMP-1) have been shown to predict prognosis in sepsis. However, MMP-8 and TIMP-1 in Staphylococcus aureus bacteremia (SAB) lacks evaluation and their role in the pathogenesis of SAB is unclear. MMP-8 and TIMP-1 and MMP-8/TIMP-1 molar ratio were determined at days 3, 5 and 28 from positive blood cultures in patients with methicillin-sensitive SAB and the connection to disease severity and early mortality was determined. Altogether 395 SAB patients were included. Patients with severe sepsis or infection focus presented higher MMP-8 levels at day 3 and 5 (p<0.01). Higher day 3 and 5 MMP-8 levels were associated to mortality at day 14 and 28 (p<0.01) and day 90 (p<0.05). Day 3 MMP-8 cut-off value of 203 ng/ml predicted death within 14 days with an area under the curve (AUC) of 0.70 (95% CI 0.57-0.82) (p<0.01). Day 5 MMP-8 cut-off value of 239 ng/ml predicted death within 14 days with an AUC of 0.76 (95% CI 0.65-0.87) (p<0.001). The results for MMP-8/TIMP-1 resembled that of MMP-8. TIMP-1 had no prognostic impact. In Cox regression analysis day 3 or 5 MMP-8 or day 3 MMP-8/TIMP-1 had no prognostic impact whereas day 5 MMP-8/TIMP-1 predicted mortality within 14 days (HR, 4.71; CI, 95% 1.67-13.3; p<0.01). MMP-8 and MMP-8/TIMP-1 ratio were high 3-5 days after MS-SAB diagnosis in patients with an infection focus, severe sepsis or mortality within 14 days suggesting that matrix metalloproteinase activation might play a role in severe SAB.

Sections du résumé

BACKGROUND
Matrix metalloproteinase-8 (MMP-8) and tissue inhibitor of metalloproteinases-1 (TIMP-1) have been shown to predict prognosis in sepsis. However, MMP-8 and TIMP-1 in Staphylococcus aureus bacteremia (SAB) lacks evaluation and their role in the pathogenesis of SAB is unclear.
METHODS
MMP-8 and TIMP-1 and MMP-8/TIMP-1 molar ratio were determined at days 3, 5 and 28 from positive blood cultures in patients with methicillin-sensitive SAB and the connection to disease severity and early mortality was determined.
RESULTS
Altogether 395 SAB patients were included. Patients with severe sepsis or infection focus presented higher MMP-8 levels at day 3 and 5 (p<0.01). Higher day 3 and 5 MMP-8 levels were associated to mortality at day 14 and 28 (p<0.01) and day 90 (p<0.05). Day 3 MMP-8 cut-off value of 203 ng/ml predicted death within 14 days with an area under the curve (AUC) of 0.70 (95% CI 0.57-0.82) (p<0.01). Day 5 MMP-8 cut-off value of 239 ng/ml predicted death within 14 days with an AUC of 0.76 (95% CI 0.65-0.87) (p<0.001). The results for MMP-8/TIMP-1 resembled that of MMP-8. TIMP-1 had no prognostic impact. In Cox regression analysis day 3 or 5 MMP-8 or day 3 MMP-8/TIMP-1 had no prognostic impact whereas day 5 MMP-8/TIMP-1 predicted mortality within 14 days (HR, 4.71; CI, 95% 1.67-13.3; p<0.01).
CONCLUSION
MMP-8 and MMP-8/TIMP-1 ratio were high 3-5 days after MS-SAB diagnosis in patients with an infection focus, severe sepsis or mortality within 14 days suggesting that matrix metalloproteinase activation might play a role in severe SAB.

Identifiants

pubmed: 34043679
doi: 10.1371/journal.pone.0252046
pii: PONE-D-20-34377
pmc: PMC8158883
doi:

Substances chimiques

Anti-Bacterial Agents 0
Biomarkers 0
TIMP1 protein, human 0
Tissue Inhibitor of Metalloproteinase-1 0
MMP8 protein, human EC 3.4.24.34
Matrix Metalloproteinase 8 EC 3.4.24.34

Types de publication

Journal Article Multicenter Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0252046

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

Références

PLoS One. 2017 Feb 13;12(2):e0171191
pubmed: 28192449
Acta Anaesthesiol Scand. 2015 Feb;59(2):176-84
pubmed: 25329743
Matrix Biol. 2015 May-Jul;44-46:247-54
pubmed: 25805621
Wien Klin Wochenschr. 2021 Feb;133(3-4):86-95
pubmed: 31932967
Front Immunol. 2018 Apr 06;9:691
pubmed: 29681903
Crit Care. 2009;13(5):R158
pubmed: 19799791
PLoS One. 2014 Apr 11;9(4):e94318
pubmed: 24727739
Shock. 2004 Oct;22(4):376-9
pubmed: 15377895
Shock. 2009 Feb;31(2):146-50
pubmed: 18636041
Ann N Y Acad Sci. 1994 Sep 6;732:424-6
pubmed: 7978828
J Intern Med. 2006 Feb;259(2):179-90
pubmed: 16420547
Shock. 2005 Sep;24(3):214-8
pubmed: 16135959
Clin Infect Dis. 2003 Jan 1;36(1):53-9
pubmed: 12491202
Crit Care. 2010;14(2):R49
pubmed: 20356362
Shock. 2007 Feb;27(2):145-50
pubmed: 17224788
Scand J Infect Dis. 2005;37(8):572-578
pubmed: 16138425
J Intensive Care Med. 2020 Jan;35(1):95-103
pubmed: 28931365
Clin Infect Dis. 2003 Jun 1;36(11):1418-23
pubmed: 12766837
Int J Infect Dis. 2011 Jan;15(1):e58-62
pubmed: 21111647
Crit Care Med. 2003 Apr;31(4):1250-6
pubmed: 12682500
J Infect Dis. 1982 Aug;146(2):227-34
pubmed: 7108273
J Hosp Infect. 2011 Jun;78(2):102-7
pubmed: 21511366
Pharmacol Res. 2011 Dec;64(6):590-4
pubmed: 21742038
Ann Med. 2009;41(3):208-15
pubmed: 18979291
Clin Microbiol Infect. 2015 Aug;21(8):779-85
pubmed: 26033668
J Biol Chem. 1997 Dec 12;272(50):31504-9
pubmed: 9395486
Clin Infect Dis. 2000 Apr;30(4):633-8
pubmed: 10770721
Inflamm Res. 2014 May;63(5):329-34
pubmed: 24378958
Br J Surg. 1997 Feb;84(2):160-6
pubmed: 9052425
Biochem Soc Symp. 2003;(70):201-12
pubmed: 14587293
Anesth Analg. 2014 Apr;118(4):790-8
pubmed: 24651234
Am J Med. 2010 Jul;123(7):631-7
pubmed: 20493464
Infect Immun. 2000 Jul;68(7):3965-70
pubmed: 10858210
Am J Med Sci. 1998 Dec;316(6):355-60
pubmed: 9856688
Crit Care. 2013 May 25;17(3):R94
pubmed: 23706069
Scand J Infect Dis. 2006;38(10):867-72
pubmed: 17008230
Clin Microbiol Infect. 2016 Nov;22(11):948.e1-948.e7
pubmed: 27515395
Clin Infect Dis. 2009 Oct 1;49(7):997-1005
pubmed: 19719417
Eur J Clin Invest. 2016 Sep;46(9):757-65
pubmed: 27296149
Ann Med. 2006;38(5):306-21
pubmed: 16938801
Clin Exp Immunol. 2005 Oct;142(1):12-20
pubmed: 16178851
Diagn Microbiol Infect Dis. 1999 Apr;33(4):283-97
pubmed: 10212756
J Infect. 2009 Oct;59(4):232-9
pubmed: 19654021
PLoS One. 2015 Apr 13;10(4):e0122824
pubmed: 25874546
FEMS Immunol Med Microbiol. 1993 Oct;7(3):281-7
pubmed: 8275059
Ann Intern Med. 2004 Jan 6;140(1):26-32
pubmed: 14706969
Trends Cardiovasc Med. 2011 May;21(4):112-8
pubmed: 22681966

Auteurs

Erik Forsblom (E)

Division of Infectious Diseases, Inflammation Center, University of Helsinki and Helsinki University Hospital, Helsinki, Finland.
Division of Infectious Diseases, Inflammation Center, Helsinki University Central Hospital, Meilahti Triangle Hospital, Helsinki, Finland.

Taina Tervahartiala (T)

Department of Oral and Maxillofacial Diseases, University of Helsinki and Helsinki University Hospital, Helsinki, Finland.

Eeva Ruotsalainen (E)

Division of Infectious Diseases, Inflammation Center, University of Helsinki and Helsinki University Hospital, Helsinki, Finland.

Asko Järvinen (A)

Division of Infectious Diseases, Inflammation Center, University of Helsinki and Helsinki University Hospital, Helsinki, Finland.

Timo Sorsa (T)

Department of Oral and Maxillofacial Diseases, University of Helsinki and Helsinki University Hospital, Helsinki, Finland.
Division of Periodontology, Department of Dental Medicine, Karolinska Institutet, Huddinge, Sweden.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH