Association between carotid plaque vulnerability and high mobility group box-1 serum levels in a diabetic population.


Journal

Cardiovascular diabetology
ISSN: 1475-2840
Titre abrégé: Cardiovasc Diabetol
Pays: England
ID NLM: 101147637

Informations de publication

Date de publication:
27 05 2021
Historique:
received: 25 03 2021
accepted: 19 05 2021
entrez: 28 5 2021
pubmed: 29 5 2021
medline: 3 11 2021
Statut: epublish

Résumé

Carotid atherosclerosis represents one of the complications of diabetes mellitus. In particular, plaque instability contributes to disease progression and stroke incidence. High mobility group box-1 (HMGB1) is a nuclear protein involved in promotion and progression of atherosclerosis and cardiovascular diseases. The aim of this study was to analyze the relationship between HMGB1 serum levels, main inflammatory cytokines, the presence of internal carotid stenosis and unstable plaque in a diabetic population. We studied 873 diabetic patients, including 347 patients with internal carotid artery stenosis (ICAS) who underwent carotid endarterectomy and 526 diabetic patients without internal carotid artery stenosis (WICAS). At baseline, HMGB1 and the main inflammatory cytokines serum levels were evaluated. For ICAS patients, the histological features of carotid plaque were also collected to differentiate them in patients with stable or unstable atherosclerotic lesions. We found that HMGB1 serum levels, osteoprotegerin, high-sensitivity C-reactive protein, tumor necrosis factor-alpha and interleukin-6, were significantly higher in diabetic ICAS patients compared to diabetic WICAS patients. Among ICAS patients, individuals with unstable plaque had higher levels of these cytokines, compared to patients with stable plaque. A multivariable stepwise logistic regression analysis showed that HMGB1 and osteoprotegerin remained independently associated with unstable plaque in ICAS patients. The present study demonstrated that HMGB1 is an independent risk factor for carotid plaque vulnerability in an Italian population with diabetes mellitus, representing a promising biomarker of carotid plaque instability and a possible molecular target to treat unstable carotid plaques and to prevent stroke.

Sections du résumé

BACKGROUND
Carotid atherosclerosis represents one of the complications of diabetes mellitus. In particular, plaque instability contributes to disease progression and stroke incidence. High mobility group box-1 (HMGB1) is a nuclear protein involved in promotion and progression of atherosclerosis and cardiovascular diseases. The aim of this study was to analyze the relationship between HMGB1 serum levels, main inflammatory cytokines, the presence of internal carotid stenosis and unstable plaque in a diabetic population.
RESEARCH DESIGN AND METHODS
We studied 873 diabetic patients, including 347 patients with internal carotid artery stenosis (ICAS) who underwent carotid endarterectomy and 526 diabetic patients without internal carotid artery stenosis (WICAS). At baseline, HMGB1 and the main inflammatory cytokines serum levels were evaluated. For ICAS patients, the histological features of carotid plaque were also collected to differentiate them in patients with stable or unstable atherosclerotic lesions.
RESULTS
We found that HMGB1 serum levels, osteoprotegerin, high-sensitivity C-reactive protein, tumor necrosis factor-alpha and interleukin-6, were significantly higher in diabetic ICAS patients compared to diabetic WICAS patients. Among ICAS patients, individuals with unstable plaque had higher levels of these cytokines, compared to patients with stable plaque. A multivariable stepwise logistic regression analysis showed that HMGB1 and osteoprotegerin remained independently associated with unstable plaque in ICAS patients.
CONCLUSIONS
The present study demonstrated that HMGB1 is an independent risk factor for carotid plaque vulnerability in an Italian population with diabetes mellitus, representing a promising biomarker of carotid plaque instability and a possible molecular target to treat unstable carotid plaques and to prevent stroke.

Identifiants

pubmed: 34044825
doi: 10.1186/s12933-021-01304-8
pii: 10.1186/s12933-021-01304-8
pmc: PMC8161555
doi:

Substances chimiques

Biomarkers 0
HMGB1 Protein 0
HMGB1 protein, human 0
IL6 protein, human 0
Inflammation Mediators 0
Interleukin-6 0
Osteoprotegerin 0
TNF protein, human 0
TNFRSF11B protein, human 0
Tumor Necrosis Factor-alpha 0
C-Reactive Protein 9007-41-4

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

114

Commentaires et corrections

Type : ErratumIn

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Auteurs

Federico Biscetti (F)

Fondazione Policlinico Universitario A. Gemelli IRCCS, Roma, Italy. f.biscetti@gmail.com.
Cardiovascular Internal Medicine, Fondazione Policlinico Universitario A. Gemelli IRCCS, Catholic University School of Medicine, Largo Francesco Vito, 1, 00168, Roma, Italy. f.biscetti@gmail.com.
Laboratory of Vascular Biology and Genetics, Università Cattolica del Sacro Cuore, Roma, Italy. f.biscetti@gmail.com.

Giovanni Tinelli (G)

Fondazione Policlinico Universitario A. Gemelli IRCCS, Roma, Italy.
Vascular Surgery, Fondazione Policlinico Universitario A. Gemelli IRCCS, Roma, Italy.
Università Cattolica del Sacro Cuore, Roma, Italy.

Maria Margherita Rando (MM)

Fondazione Policlinico Universitario A. Gemelli IRCCS, Roma, Italy.
Cardiovascular Internal Medicine, Fondazione Policlinico Universitario A. Gemelli IRCCS, Catholic University School of Medicine, Largo Francesco Vito, 1, 00168, Roma, Italy.

Elisabetta Nardella (E)

Laboratory of Vascular Biology and Genetics, Università Cattolica del Sacro Cuore, Roma, Italy.

Andrea Leonardo Cecchini (AL)

Laboratory of Vascular Biology and Genetics, Università Cattolica del Sacro Cuore, Roma, Italy.

Flavia Angelini (F)

Laboratory of Vascular Biology and Genetics, Università Cattolica del Sacro Cuore, Roma, Italy.

Giuseppe Straface (G)

Department of Internal Medicine, St. M. Goretti Hospital, Roma, Italy.

Marco Filipponi (M)

Ospedale San Giovanni Battista-ACISMOM, Roma, Italy.

Vincenzo Arena (V)

Department of Pathology, Fondazione Policlinico Universitario A. Gemelli IRCCS, Roma, Italy.

Dario Pitocco (D)

Fondazione Policlinico Universitario A. Gemelli IRCCS, Roma, Italy.
Università Cattolica del Sacro Cuore, Roma, Italy.
Diabetology Unit, Fondazione Policlinico Universitario A. Gemelli IRCCS, Roma, Italy.

Antonio Gasbarrini (A)

Fondazione Policlinico Universitario A. Gemelli IRCCS, Roma, Italy.
Università Cattolica del Sacro Cuore, Roma, Italy.
Department of Internal Medicine, Fondazione Policlinico Universitario A. Gemelli IRCCS, Roma, Italy.

Massimo Massetti (M)

Fondazione Policlinico Universitario A. Gemelli IRCCS, Roma, Italy.
Università Cattolica del Sacro Cuore, Roma, Italy.
Cardiovascular Surgery, Fondazione Policlinico Universitario A. Gemelli IRCCS, Roma, Italy.

Andrea Flex (A)

Fondazione Policlinico Universitario A. Gemelli IRCCS, Roma, Italy.
Cardiovascular Internal Medicine, Fondazione Policlinico Universitario A. Gemelli IRCCS, Catholic University School of Medicine, Largo Francesco Vito, 1, 00168, Roma, Italy.
Laboratory of Vascular Biology and Genetics, Università Cattolica del Sacro Cuore, Roma, Italy.
Università Cattolica del Sacro Cuore, Roma, Italy.

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