Estrogen suppresses HOXB2 expression via ERα in breast cancer cells.


Journal

Gene
ISSN: 1879-0038
Titre abrégé: Gene
Pays: Netherlands
ID NLM: 7706761

Informations de publication

Date de publication:
20 Aug 2021
Historique:
received: 28 08 2020
revised: 27 04 2021
accepted: 27 05 2021
pubmed: 2 6 2021
medline: 24 6 2021
entrez: 1 6 2021
Statut: ppublish

Résumé

The expression of HOXB2, a homeobox transcription factor, is altered in a variety of solid tumors. Using an in vivo screen to identify regulators of breast tumor growth in murine mammary fat pads, Boimel and co-workers recently identified HOXB2 as a tumor suppressor. However, the mechanistic underpinnings of its role in breast cancer is not understood. Given the emerging interaction of estrogen-regulated gene expression and altered HOX gene expression network in the pathophysiology of breast cancer, this study addressed the relationship between estrogen signaling and HOXB2 expression. Using a mouse model and human breast cancer cell lines, we show that estrogen suppresses HOXB2 expression. Suppression of HOXB2 by PPT, a known ERα agonist, in MCF-7 and T47D cells indicated the involvement of ERα, which was confirmed by siRNA-mediated ERα knockdown experiments. In-silico analysis of the upstream promoter region revealed the presence of three putative EREs. Chromatin immunoprecipitation experiments showed that upon estrogen binding, ERα engaged with EREs in the 5' upstream region of HOXB2 in MCF-7 and T47D cells. Future investigations should address the implications of estrogen-mediated suppression on the proposed tumor suppressor function of HOXB2.

Identifiants

pubmed: 34062258
pii: S0378-1119(21)00340-1
doi: 10.1016/j.gene.2021.145746
pii:
doi:

Substances chimiques

ESR1 protein, human 0
Estrogen Receptor alpha 0
HOXB2 protein, human 0
Homeodomain Proteins 0
Phenols 0
Pyrazoles 0
Transcription Factors 0
4,4',4''-(4-propyl-((1)H)-pyrazole-1,3,5-triyl) tris-phenol 0T83Y6JZPF

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

145746

Informations de copyright

Copyright © 2021 Elsevier B.V. All rights reserved.

Auteurs

Ajay Kumar (A)

Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati 781039, Assam, India.

Arun Dhillon (A)

Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati 781039, Assam, India.

Mohan Chowdenahalli Manjegowda (MC)

Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati 781039, Assam, India.

Neha Singh (N)

Molecular and Cellular Biology Laboratory, ICMR-National Institute of Research in Reproductive Health, Indian Council of Medical Research (ICMR), Parel 400012, Mumbai, India.

Dixcy Jaba Sheeba John Mary (DJSJ)

Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati 781039, Assam, India.

Sachin Kumar (S)

Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati 781039, Assam, India.

Deepak Modi (D)

Molecular and Cellular Biology Laboratory, ICMR-National Institute of Research in Reproductive Health, Indian Council of Medical Research (ICMR), Parel 400012, Mumbai, India.

Anil Mukund Limaye (AM)

Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati 781039, Assam, India. Electronic address: amul@iitg.ac.in.

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Classifications MeSH