Circulating tumor cells, circulating tumor DNA, and disease characteristics in young women with metastatic breast cancer.


Journal

Breast cancer research and treatment
ISSN: 1573-7217
Titre abrégé: Breast Cancer Res Treat
Pays: Netherlands
ID NLM: 8111104

Informations de publication

Date de publication:
Jun 2021
Historique:
received: 05 01 2021
accepted: 17 04 2021
pubmed: 3 6 2021
medline: 24 6 2021
entrez: 2 6 2021
Statut: ppublish

Résumé

Clinical and genomic data from patients with early-stage breast cancer suggest more aggressive disease in premenopausal women. However, the association between age, disease course, and molecular profile from liquid biopsy in metastatic breast cancer (MBC) is not well characterized. Patients were classified as premenopausal (< 45 years), perimenopausal (45-55 years), or postmenopausal (> 55 years). Cohort 1 consisted of patients with MBC who consented for prospective serial evaluation of circulating tumor cells (CTCs) using CellSearch™. Cohort 2 included patients who, as part of routine care, had circulating tumor DNA (ctDNA) sequenced by the Guardant360™ assay. Clinicopathologic data were collected from retrospective review to compare disease features between premenopausal and postmenopausal women. Premenopausal women represented 26% of 138 patients in Cohort 1 and 21% of 253 patients in Cohort 2. In Cohort 1, younger patients had a shorter time to metastases and a higher prevalence of lung and brain metastases. Overall, there were similar rates of ≥ 5 CTCs/7.5 mL, HER2 + CTC expression, and CTC clusters between pre- and postmenopausal women. However, for those with triple negative breast cancer, premenopausal women had a higher proportion of ≥ 5 CTCs/7.5 mL. In Cohort 2, premenopausal women had a higher incidence of FGFR1 (OR 2.75, p = 0.022) and CCND2 (OR 6.91, p = 0.024) alterations. There was no difference in the ctDNA mutant allele frequency or the number of detected alterations between these age groups. Our data reveal that premenopausal women diagnosed with MBC have unique clinical, pathologic, and molecular features when compared to their postmenopausal counterparts. Our results highlight FGFR1 inhibitors as potential therapeutics of particular interest among premenopausal women.

Sections du résumé

BACKGROUND BACKGROUND
Clinical and genomic data from patients with early-stage breast cancer suggest more aggressive disease in premenopausal women. However, the association between age, disease course, and molecular profile from liquid biopsy in metastatic breast cancer (MBC) is not well characterized.
METHODS METHODS
Patients were classified as premenopausal (< 45 years), perimenopausal (45-55 years), or postmenopausal (> 55 years). Cohort 1 consisted of patients with MBC who consented for prospective serial evaluation of circulating tumor cells (CTCs) using CellSearch™. Cohort 2 included patients who, as part of routine care, had circulating tumor DNA (ctDNA) sequenced by the Guardant360™ assay. Clinicopathologic data were collected from retrospective review to compare disease features between premenopausal and postmenopausal women.
RESULTS RESULTS
Premenopausal women represented 26% of 138 patients in Cohort 1 and 21% of 253 patients in Cohort 2. In Cohort 1, younger patients had a shorter time to metastases and a higher prevalence of lung and brain metastases. Overall, there were similar rates of ≥ 5 CTCs/7.5 mL, HER2 + CTC expression, and CTC clusters between pre- and postmenopausal women. However, for those with triple negative breast cancer, premenopausal women had a higher proportion of ≥ 5 CTCs/7.5 mL. In Cohort 2, premenopausal women had a higher incidence of FGFR1 (OR 2.75, p = 0.022) and CCND2 (OR 6.91, p = 0.024) alterations. There was no difference in the ctDNA mutant allele frequency or the number of detected alterations between these age groups.
CONCLUSIONS CONCLUSIONS
Our data reveal that premenopausal women diagnosed with MBC have unique clinical, pathologic, and molecular features when compared to their postmenopausal counterparts. Our results highlight FGFR1 inhibitors as potential therapeutics of particular interest among premenopausal women.

Identifiants

pubmed: 34076801
doi: 10.1007/s10549-021-06236-1
pii: 10.1007/s10549-021-06236-1
doi:

Substances chimiques

Biomarkers, Tumor 0
Circulating Tumor DNA 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

397-405

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Auteurs

Ami N Shah (AN)

Robert H. Lurie Comprehensive Cancer Center of Northwestern University, 676 N St. Clair St Suite 850, Chicago, IL, 60611, USA. amishah@northwestern.edu.

Kristen J Carroll (KJ)

Robert H. Lurie Comprehensive Cancer Center of Northwestern University, 676 N St. Clair St Suite 850, Chicago, IL, 60611, USA.

Lorenzo Gerratana (L)

Department of Medicine (DAME), University of Udine, 33100, Udine, Italy.
Department of Medical Oncology, IRCCS Centro Di Riferimento Oncologico Di Aviano (CRO), Aviano, Italy.

Chenyu Lin (C)

Duke Cancer Institute, Duke University Medical Center, Durham, NC, USA.

Andrew A Davis (AA)

Division of Oncology, Department of Medicine, Washington University School of Medicine, St. Louis, MO, USA.

Qiang Zhang (Q)

Robert H. Lurie Comprehensive Cancer Center of Northwestern University, 676 N St. Clair St Suite 850, Chicago, IL, 60611, USA.

Saya Jacob (S)

Robert H. Lurie Comprehensive Cancer Center of Northwestern University, 676 N St. Clair St Suite 850, Chicago, IL, 60611, USA.

Youbin Zhang (Y)

Robert H. Lurie Comprehensive Cancer Center of Northwestern University, 676 N St. Clair St Suite 850, Chicago, IL, 60611, USA.

Wenen Qiang (W)

Robert H. Lurie Comprehensive Cancer Center of Northwestern University, 676 N St. Clair St Suite 850, Chicago, IL, 60611, USA.

Paolo D'Amico (P)

Robert H. Lurie Comprehensive Cancer Center of Northwestern University, 676 N St. Clair St Suite 850, Chicago, IL, 60611, USA.
Division of Early Drug Development for Innovative Therapies, IEO, European Institute of Oncology IRCCS, 20132, Milan, Italy.

Carolina Reduzzi (C)

Robert H. Lurie Comprehensive Cancer Center of Northwestern University, 676 N St. Clair St Suite 850, Chicago, IL, 60611, USA.

William J Gradishar (WJ)

Robert H. Lurie Comprehensive Cancer Center of Northwestern University, 676 N St. Clair St Suite 850, Chicago, IL, 60611, USA.

Amir Behdad (A)

Robert H. Lurie Comprehensive Cancer Center of Northwestern University, 676 N St. Clair St Suite 850, Chicago, IL, 60611, USA.

Massimo Cristofanilli (M)

Robert H. Lurie Comprehensive Cancer Center of Northwestern University, 676 N St. Clair St Suite 850, Chicago, IL, 60611, USA.

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