Compensatory mechanisms in resistant Anopheles gambiae AcerKis and KdrKis neurons modulate insecticide-based mosquito control.
Acetylcholine
/ pharmacology
Acetylcholinesterase
/ genetics
Animals
Anopheles
/ drug effects
Calcium
/ metabolism
Calcium Channels
/ metabolism
Female
Genes, Insect
Humans
In Vitro Techniques
Insecticide Resistance
/ genetics
Insecticides
/ pharmacology
Malaria
/ transmission
Mosquito Control
/ methods
Mosquito Vectors
/ drug effects
Neurons
/ drug effects
Patch-Clamp Techniques
Point Mutation
Receptors, Nicotinic
/ metabolism
Sodium Channels
/ genetics
Journal
Communications biology
ISSN: 2399-3642
Titre abrégé: Commun Biol
Pays: England
ID NLM: 101719179
Informations de publication
Date de publication:
02 06 2021
02 06 2021
Historique:
received:
07
01
2021
accepted:
06
05
2021
entrez:
3
6
2021
pubmed:
4
6
2021
medline:
11
8
2021
Statut:
epublish
Résumé
In the malaria vector Anopheles gambiae, two point mutations in the acetylcholinesterase (ace-1
Identifiants
pubmed: 34079061
doi: 10.1038/s42003-021-02192-0
pii: 10.1038/s42003-021-02192-0
pmc: PMC8172894
doi:
Substances chimiques
Calcium Channels
0
Insecticides
0
Receptors, Nicotinic
0
Sodium Channels
0
Acetylcholinesterase
EC 3.1.1.7
Acetylcholine
N9YNS0M02X
Calcium
SY7Q814VUP
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
665Références
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