Features of post-endoscopic submucosal dissection electrocoagulation syndrome for early gastric neoplasm.

Early gastric neoplasm Endoscopic submucosal dissection Post-endoscopic submucosal dissection electrocoagulation syndrome

Journal

Journal of gastroenterology and hepatology
ISSN: 1440-1746
Titre abrégé: J Gastroenterol Hepatol
Pays: Australia
ID NLM: 8607909

Informations de publication

Date de publication:
Nov 2021
Historique:
revised: 01 06 2021
received: 07 03 2021
accepted: 13 06 2021
pubmed: 16 6 2021
medline: 3 3 2022
entrez: 15 6 2021
Statut: ppublish

Résumé

Post-endoscopic submucosal dissection electrocoagulation syndrome (PECS) has become a common adverse event after colorectal endoscopic submucosal dissection (ESD) and esophageal ESD. However, little is known about PECS after gastric ESD. Therefore, this study aimed to investigate the clinical features of PECS after gastric ESD. Patients who underwent ESD for gastric cancer or adenoma between January 2016 and December 2017 were retrospectively investigated. PECS was clinically diagnosed based on the presence of upper abdominal pain and localized abdominal tenderness with a temperature of >37.5°C, without perforation. We analyzed the clinical features of PECS. A total of 637 ESD cases were enrolled; PECS occurred in 32 patients (5.0%), all of whom were diagnosed on postoperative Day 1. Among PECS cases, unplanned prolongation of hospitalization or fasting period was observed in 15 patients (47%). As a result, the median durations of hospitalization and fasting period were significantly longer in PECS cases (P = 0.008 and P < 0.001, respectively); however, the mean differences were less than a day. Additionally, all PECS cases recovered with conservative treatment. PECS is considered a common adverse event after gastric ESD. More than half of patients with PECS could start diets and be discharged as well as those without PECS.

Sections du résumé

BACKGROUND AND AIM OBJECTIVE
Post-endoscopic submucosal dissection electrocoagulation syndrome (PECS) has become a common adverse event after colorectal endoscopic submucosal dissection (ESD) and esophageal ESD. However, little is known about PECS after gastric ESD. Therefore, this study aimed to investigate the clinical features of PECS after gastric ESD.
METHODS METHODS
Patients who underwent ESD for gastric cancer or adenoma between January 2016 and December 2017 were retrospectively investigated. PECS was clinically diagnosed based on the presence of upper abdominal pain and localized abdominal tenderness with a temperature of >37.5°C, without perforation. We analyzed the clinical features of PECS.
RESULTS RESULTS
A total of 637 ESD cases were enrolled; PECS occurred in 32 patients (5.0%), all of whom were diagnosed on postoperative Day 1. Among PECS cases, unplanned prolongation of hospitalization or fasting period was observed in 15 patients (47%). As a result, the median durations of hospitalization and fasting period were significantly longer in PECS cases (P = 0.008 and P < 0.001, respectively); however, the mean differences were less than a day. Additionally, all PECS cases recovered with conservative treatment.
CONCLUSIONS CONCLUSIONS
PECS is considered a common adverse event after gastric ESD. More than half of patients with PECS could start diets and be discharged as well as those without PECS.

Identifiants

pubmed: 34129729
doi: 10.1111/jgh.15583
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

3164-3169

Informations de copyright

© 2021 Journal of Gastroenterology and Hepatology Foundation and John Wiley & Sons Australia, Ltd.

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Auteurs

Hidenori Kimura (H)

Division of Endoscopy, Shizuoka Cancer Center, Nagaizumi, Japan.
Division of Gastroenterology, Department of Medicine, Shiga University of Medical Science, Otsu, Japan.

Yohei Yabuuchi (Y)

Division of Endoscopy, Shizuoka Cancer Center, Nagaizumi, Japan.

Akifumi Notsu (A)

Clinical Research Center, Shizuoka Cancer Center, Nagaizumi, Japan.

Yoichi Yamamoto (Y)

Division of Endoscopy, Shizuoka Cancer Center, Nagaizumi, Japan.

Masao Yoshida (M)

Division of Endoscopy, Shizuoka Cancer Center, Nagaizumi, Japan.

Noboru Kawata (N)

Division of Endoscopy, Shizuoka Cancer Center, Nagaizumi, Japan.

Kohei Takizawa (K)

Division of Endoscopy, Shizuoka Cancer Center, Nagaizumi, Japan.

Yoshihiro Kishida (Y)

Division of Endoscopy, Shizuoka Cancer Center, Nagaizumi, Japan.

Kenichiro Imai (K)

Division of Endoscopy, Shizuoka Cancer Center, Nagaizumi, Japan.

Sayo Ito (S)

Division of Endoscopy, Shizuoka Cancer Center, Nagaizumi, Japan.

Kinichi Hotta (K)

Division of Endoscopy, Shizuoka Cancer Center, Nagaizumi, Japan.

Hirotoshi Ishiwatari (H)

Division of Endoscopy, Shizuoka Cancer Center, Nagaizumi, Japan.

Hiroyuki Matsubayashi (H)

Division of Endoscopy, Shizuoka Cancer Center, Nagaizumi, Japan.

Hiroyuki Ono (H)

Division of Endoscopy, Shizuoka Cancer Center, Nagaizumi, Japan.

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