Inhibition of sphingosine 1-phosphate protects mice against chondrocyte catabolism and osteoarthritis.


Journal

Osteoarthritis and cartilage
ISSN: 1522-9653
Titre abrégé: Osteoarthritis Cartilage
Pays: England
ID NLM: 9305697

Informations de publication

Date de publication:
09 2021
Historique:
received: 11 02 2021
revised: 18 05 2021
accepted: 07 06 2021
pubmed: 19 6 2021
medline: 23 2 2022
entrez: 18 6 2021
Statut: ppublish

Résumé

Cartilage loss observed in osteoarthritis (OA) is prevented when osteoclasts in the subchondral bone are inhibited in mice. Here, we investigated the role of the osteoclast secretome and of the lipid mediator sphingosine 1-phosphate (S1P) in chondrocyte metabolism and OA. We used SphK1 The osteoclast secretome increased the expression of Mmp3 and Mmp13 in murine chondrocytes and cartilage explants and activated the JNK signaling pathway, which led to matrix degradation. JTE013 reversed the osteoclast-mediated chondrocyte catabolism and protected mice against OA, suggesting that osteoclastic S1P contributes to cartilage damage in OA via S1P/S1P Lack of S1P in myeloid cells and local S1P neutralization alleviates from osteoarthritis in mice. These data identify S1P as a therapeutic target in OA.

Identifiants

pubmed: 34144150
pii: S1063-4584(21)00807-4
doi: 10.1016/j.joca.2021.06.001
pii:
doi:

Substances chimiques

Lysophospholipids 0
sphingosine 1-phosphate 26993-30-6
Sphingosine NGZ37HRE42

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1335-1345

Informations de copyright

Copyright © 2021 The Author(s). Published by Elsevier Ltd.. All rights reserved.

Auteurs

C Cherifi (C)

Université de Paris, BIOSCAR Inserm U1132, Paris, F-75010, France.

A Latourte (A)

Université de Paris, BIOSCAR Inserm U1132, Paris, F-75010, France; Service de rhumatologie, AP-HP, Hôpital Lariboisière, Paris, F-75010, France.

S Vettorazzi (S)

Institute of Comparative Molecular Endocrinology, University of Ulm, ZC 89081, Germany.

J Tuckermann (J)

Institute of Comparative Molecular Endocrinology, University of Ulm, ZC 89081, Germany.

S Provot (S)

Université de Paris, BIOSCAR Inserm U1132, Paris, F-75010, France.

H-K Ea (HK)

Université de Paris, BIOSCAR Inserm U1132, Paris, F-75010, France; Service de rhumatologie, AP-HP, Hôpital Lariboisière, Paris, F-75010, France.

A Ledoux (A)

Institut de Pharmacologie et de Biologie Structurale, Université de Toulouse, CNRS, UPS, Toulouse, F-31000, France.

J Casas (J)

RUBAM, Department Biological Chemistry, IQAC-CSIC, Jordi Girona 18-26, Barcelona, S-08034, Spain; Networking Biomedical Research Centre (CIBEREHD), Madrid, S-28706, Spain.

O Cuvillier (O)

Institut de Pharmacologie et de Biologie Structurale, Université de Toulouse, CNRS, UPS, Toulouse, F-31000, France.

P Richette (P)

Université de Paris, BIOSCAR Inserm U1132, Paris, F-75010, France; Service de rhumatologie, AP-HP, Hôpital Lariboisière, Paris, F-75010, France.

A Ostertag (A)

Université de Paris, BIOSCAR Inserm U1132, Paris, F-75010, France.

E Hay (E)

Université de Paris, BIOSCAR Inserm U1132, Paris, F-75010, France.

M Cohen-Solal (M)

Université de Paris, BIOSCAR Inserm U1132, Paris, F-75010, France; Service de rhumatologie, AP-HP, Hôpital Lariboisière, Paris, F-75010, France. Electronic address: martine.cohen-solal@inserm.fr.

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Classifications MeSH