Cell-specific and divergent roles of the CD40L-CD40 axis in atherosclerotic vascular disease.
Animals
Atherosclerosis
/ pathology
Blood Platelets
/ metabolism
CD4-Positive T-Lymphocytes
/ cytology
CD40 Antigens
/ metabolism
CD40 Ligand
/ metabolism
Cardiovascular Diseases
/ pathology
Dendritic Cells
/ immunology
Interferon-gamma
/ metabolism
Mice
Mice, Knockout
Myocytes, Smooth Muscle
/ cytology
Plaque, Atherosclerotic
/ pathology
Signal Transduction
/ physiology
Thrombosis
/ pathology
Journal
Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555
Informations de publication
Date de publication:
18 06 2021
18 06 2021
Historique:
received:
02
02
2020
accepted:
19
05
2021
entrez:
19
6
2021
pubmed:
20
6
2021
medline:
7
7
2021
Statut:
epublish
Résumé
Atherosclerosis is a major underlying cause of cardiovascular disease. Previous studies showed that inhibition of the co-stimulatory CD40 ligand (CD40L)-CD40 signaling axis profoundly attenuates atherosclerosis. As CD40L exerts multiple functions depending on the cell-cell interactions involved, we sought to investigate the function of the most relevant CD40L-expressing cell types in atherosclerosis: T cells and platelets. Atherosclerosis-prone mice with a CD40L-deficiency in CD4
Identifiants
pubmed: 34145241
doi: 10.1038/s41467-021-23909-z
pii: 10.1038/s41467-021-23909-z
pmc: PMC8213756
doi:
Substances chimiques
CD40 Antigens
0
IFNG protein, mouse
0
CD40 Ligand
147205-72-9
Interferon-gamma
82115-62-6
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
3754Commentaires et corrections
Type : CommentIn
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