Clinical Significance of Global Wasted Work in Patients with Heart Failure Receiving Cardiac Resynchronization Therapy.


Journal

Journal of the American Society of Echocardiography : official publication of the American Society of Echocardiography
ISSN: 1097-6795
Titre abrégé: J Am Soc Echocardiogr
Pays: United States
ID NLM: 8801388

Informations de publication

Date de publication:
09 2021
Historique:
received: 12 05 2021
revised: 14 06 2021
accepted: 14 06 2021
pubmed: 23 6 2021
medline: 25 2 2023
entrez: 22 6 2021
Statut: ppublish

Résumé

The relationship between myocardial work assessment using pressure-strain loops by echocardiography before cardiac resynchronization therapy (CRT) and response to CRT has been recently revealed. Among myocardial work parameters, the impact of left ventricular myocardial global wasted work (GWW) on response to CRT and outcome following CRT has been seldom studied. Hence, the authors evaluated the relationship between preprocedural GWW and outcome in a large prospective cohort of patients with heart failure (HF) and reduced ejection fraction receiving CRT. The study included 249 patients with HF. Myocardial work indices including GWW were calculated using speckle-tracking strain two-dimensional echocardiography using pressure-strain loops. End points of the study were (1) response to CRT, defined as left ventricular reverse remodeling and/or absence of hospitalization for HF, and (2) all-cause death during follow-up. Median follow-up duration was 48 months (interquartile range, 43-54 months). Median preoperative GWW was 281 mm Hg% (interquartile range, 184-388 mm Hg%). Preoperative GWW was associated with CRT response (area under the curve, 0.74; P < .0001), and a 200 mm Hg% threshold discriminated CRT nonresponders from responders with 85% specificity and 50% sensitivity, even after adjustment for known predictors of CRT response (adjusted odds ratio, 4.03; 95% CI, 1.91-8.68; P < .001). After adjustment for established predictors of outcome in patients with HF with reduced ejection fraction receiving CRT, GWW < 200 mm Hg% remained associated with a relative increased risk for all-cause death compared with GWW ≥ 200 mm Hg% (adjusted hazard ratio, 2.0; 95% CI, 1.1-3.9; P = .0245). Adding GWW to a baseline model including known predictors of outcome in CRT resulted in an improvement of this model (χ Low preoperative GWW (<200 mm Hg%) is associated with absence of CRT response in CRT candidates and with a relative increased risk for all-cause death. GWW appears to be a promising parameter to improve selection for CRT of patients with HF with reduced ejection fraction.

Sections du résumé

BACKGROUND
The relationship between myocardial work assessment using pressure-strain loops by echocardiography before cardiac resynchronization therapy (CRT) and response to CRT has been recently revealed. Among myocardial work parameters, the impact of left ventricular myocardial global wasted work (GWW) on response to CRT and outcome following CRT has been seldom studied. Hence, the authors evaluated the relationship between preprocedural GWW and outcome in a large prospective cohort of patients with heart failure (HF) and reduced ejection fraction receiving CRT.
METHODS
The study included 249 patients with HF. Myocardial work indices including GWW were calculated using speckle-tracking strain two-dimensional echocardiography using pressure-strain loops. End points of the study were (1) response to CRT, defined as left ventricular reverse remodeling and/or absence of hospitalization for HF, and (2) all-cause death during follow-up.
RESULTS
Median follow-up duration was 48 months (interquartile range, 43-54 months). Median preoperative GWW was 281 mm Hg% (interquartile range, 184-388 mm Hg%). Preoperative GWW was associated with CRT response (area under the curve, 0.74; P < .0001), and a 200 mm Hg% threshold discriminated CRT nonresponders from responders with 85% specificity and 50% sensitivity, even after adjustment for known predictors of CRT response (adjusted odds ratio, 4.03; 95% CI, 1.91-8.68; P < .001). After adjustment for established predictors of outcome in patients with HF with reduced ejection fraction receiving CRT, GWW < 200 mm Hg% remained associated with a relative increased risk for all-cause death compared with GWW ≥ 200 mm Hg% (adjusted hazard ratio, 2.0; 95% CI, 1.1-3.9; P = .0245). Adding GWW to a baseline model including known predictors of outcome in CRT resulted in an improvement of this model (χ
CONCLUSIONS
Low preoperative GWW (<200 mm Hg%) is associated with absence of CRT response in CRT candidates and with a relative increased risk for all-cause death. GWW appears to be a promising parameter to improve selection for CRT of patients with HF with reduced ejection fraction.

Identifiants

pubmed: 34157400
pii: S0894-7317(21)00539-3
doi: 10.1016/j.echo.2021.06.008
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

976-986

Informations de copyright

Copyright © 2021 American Society of Echocardiography. Published by Elsevier Inc. All rights reserved.

Auteurs

Clemence Riolet (C)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France.

Aymeric Menet (A)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France.

Amandine Mailliet (A)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France.

Camille Binda (C)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France.

Alexandre Altes (A)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France.

Ludovic Appert (L)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France.

Anne Laure Castel (AL)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France.

François Delelis (F)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France.

Guillaume Viart (G)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France.

Yves Guyomar (Y)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France.

Caroline Le Goffic (C)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France.

Marie Decroocq (M)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France.

Pierre-Vladimir Ennezat (PV)

Centre Hospitalier Universitaire de Grenoble, Service de Cardiologie, Grenoble, France.

Pierre Graux (P)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France.

Christophe Tribouilloy (C)

EA 7517 MP3CV Jules Verne University of Picardie, Amiens, France; Centre Hospitalier Universitaire d'Amiens, Amiens, France.

Sylvestre Marechaux (S)

GCS-Groupement des Hôpitaux de l'Institut Catholique de Lille, Laboratoire d'Échocardiographie, Service de Cardiologie Nord, Centre des Valvulopathies, Faculté de Médecine et de Maïeutique, Université Catholique de Lille, Lille, France; EA 7517 MP3CV Jules Verne University of Picardie, Amiens, France. Electronic address: marechaux.sylvestre@ghicl.net.

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