HSD17B7 gene in self-renewal and oncogenicity of keratinocytes from Black versus White populations.

HSD enzymes OXPHOS genetic cancer susceptibility squamous cell carcinoma stem cell potential

Journal

EMBO molecular medicine
ISSN: 1757-4684
Titre abrégé: EMBO Mol Med
Pays: England
ID NLM: 101487380

Informations de publication

Date de publication:
07 07 2021
Historique:
revised: 19 05 2021
received: 15 02 2021
accepted: 20 05 2021
pubmed: 30 6 2021
medline: 26 10 2021
entrez: 29 6 2021
Statut: ppublish

Résumé

Human populations of Black African ancestry have a relatively high risk of aggressive cancer types, including keratinocyte-derived squamous cell carcinomas (SCCs). We show that primary keratinocytes (HKCs) from Black African (Black) versus White Caucasian (White) individuals have on average higher oncogenic and self-renewal potential, which are inversely related to mitochondrial electron transfer chain activity and ATP and ROS production. HSD17B7 is the top-ranked differentially expressed gene in HKCs and Head/Neck SCCs from individuals of Black African versus Caucasian ancestries, with several ancestry-specific eQTLs linked to its expression. Mirroring the differences between Black and White HKCs, modulation of the gene, coding for an enzyme involved in sex steroid and cholesterol biosynthesis, determines HKC and SCC cell proliferation and oncogenicity as well as mitochondrial OXPHOS activity. Overall, the findings point to a targetable determinant of cancer susceptibility among different human populations, amenable to prevention and management of the disease.

Identifiants

pubmed: 34185380
doi: 10.15252/emmm.202114133
pmc: PMC8261506
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e14133

Subventions

Organisme : NIAMS NIH HHS
ID : R01 AR039190
Pays : United States

Informations de copyright

© 2021 The Authors. Published under the terms of the CC BY 4.0 license.

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Auteurs

Xiaoying Xu (X)

Department of Biochemistry, University of Lausanne, Epalinges, Switzerland.

Beatrice Tassone (B)

Department of Biochemistry, University of Lausanne, Epalinges, Switzerland.

Paola Ostano (P)

Cancer Genomics Laboratory, Fondazione Edo ed Elvo Tempia, Biella, Italy.

Atul Katarkar (A)

Department of Biochemistry, University of Lausanne, Epalinges, Switzerland.

Tatiana Proust (T)

Department of Biochemistry, University of Lausanne, Epalinges, Switzerland.

Jean-Marc Joseph (JM)

Division of Pediatric Surgery, Women-Mother-Child Department, Lausanne University Hospital (CHUV), Lausanne, Switzerland.

Chiara Riganti (C)

Department of Oncology, University of Turin, Turin, Italy.

Giovanna Chiorino (G)

Cancer Genomics Laboratory, Fondazione Edo ed Elvo Tempia, Biella, Italy.

Zoltan Kutalik (Z)

University Center for Primary Care and Public Health, University of Lausanne, Lausanne, Switzerland.

Karine Lefort (K)

Department of Biochemistry, University of Lausanne, Epalinges, Switzerland.

Gian Paolo Dotto (GP)

Department of Biochemistry, University of Lausanne, Epalinges, Switzerland.
Cutaneous Biology Research Center, Massachusetts General Hospital, Charlestown, MA, USA.
International Cancer Prevention Institute, Epalinges, Switzerland.

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