Growth hormone replacement improved oocyte quality in a patient with hypopituitarism: A study of follicular fluid.

Déficit en hormone de croissance (GH) Fécondation in vitro (FIV) Growth hormone (GH) replacement Growth hormone deficiency (GHD) Hypopituitarism Hypopituitarisme In-vitro fertilization (IVF) Oocyte quality Qualité ovocytaire Substitution par GH

Journal

Annales d'endocrinologie
ISSN: 2213-3941
Titre abrégé: Ann Endocrinol (Paris)
Pays: France
ID NLM: 0116744

Informations de publication

Date de publication:
Dec 2021
Historique:
received: 16 03 2021
revised: 10 04 2021
accepted: 21 05 2021
pubmed: 30 6 2021
medline: 23 3 2022
entrez: 29 6 2021
Statut: ppublish

Résumé

Growth hormone (GH) is known to be involved in ovarian folliculogenesis and oocyte maturation. In patients with poor ovarian response without growth hormone deficiency (GHD), adjuvant GH treatment improves in-vitro fertilization (IVF) results. Improvement of oocyte quality in IVF by GH replacement was reported in only a few patients with GHD. We report on a new case with study of follicular fluid. A 29-year-old patient with hypopituitarism was referred to our infertility center. She was undergoing hormonal replacement for hypogonadotropic hypogonadism and diabetes insipidus, and did not consider at first GH replacement. Four IVF procedures were performed between 2011 and 2014. Growth hormone replacement (somatotropin 1.1mg/day) was initiated before the fourth IVF procedure and unmasked central hypothyroidism; levothyroxine (75mg/day) was introduced. It took 10 months to reach the treatment objectives for insulin-like growth factor 1 (IGF1), free triiodothyronine (fT3) and free thyroxine (fT4). GH, IGF1 and thyroid hormones were measured in the blood and follicular fluid before and after GH and thyroid hormone replacement. Oocyte and embryo quality were also compared. The first 3 IVF procedures were performed without GH replacement. 62% to 100% of mature oocytes presented one or more morphologic abnormalities: diffuse cytoplasmic granularity, large perivitelline space with fragments, fragmentation of the first polar body, ovoid shape, or difficult denudation. Embryo quality was moderate to poor (grade B to D), and no pregnancy was obtained after embryo transfer. After GH replacement, hormones levels increased in follicular fluid: GH [7.68 vs. 1.39 mIU/L], IGF1 [109 vs. <25ng/mL], fT3 [3.7 vs. 2.5pmol/L] and fT4 [1.45 vs. 0.84ng/mL]. Concomitantly, there was dramatic improvement in oocyte quality (no abnormal morphologies) and embryo quality (grade A), allowing an embryo transfer with successful pregnancy. This is the first report illustrating changes in hormonal levels in follicular fluid and the beneficial effect of GH replacement on oocyte and embryo quality during an IVF procedure in a patient with hypopituitarism. These results suggest that GH replacement is beneficial for oocyte quality in patients with GHD.

Sections du résumé

BACKGROUND BACKGROUND
Growth hormone (GH) is known to be involved in ovarian folliculogenesis and oocyte maturation. In patients with poor ovarian response without growth hormone deficiency (GHD), adjuvant GH treatment improves in-vitro fertilization (IVF) results. Improvement of oocyte quality in IVF by GH replacement was reported in only a few patients with GHD. We report on a new case with study of follicular fluid.
METHODS METHODS
A 29-year-old patient with hypopituitarism was referred to our infertility center. She was undergoing hormonal replacement for hypogonadotropic hypogonadism and diabetes insipidus, and did not consider at first GH replacement. Four IVF procedures were performed between 2011 and 2014. Growth hormone replacement (somatotropin 1.1mg/day) was initiated before the fourth IVF procedure and unmasked central hypothyroidism; levothyroxine (75mg/day) was introduced. It took 10 months to reach the treatment objectives for insulin-like growth factor 1 (IGF1), free triiodothyronine (fT3) and free thyroxine (fT4). GH, IGF1 and thyroid hormones were measured in the blood and follicular fluid before and after GH and thyroid hormone replacement. Oocyte and embryo quality were also compared.
RESULTS RESULTS
The first 3 IVF procedures were performed without GH replacement. 62% to 100% of mature oocytes presented one or more morphologic abnormalities: diffuse cytoplasmic granularity, large perivitelline space with fragments, fragmentation of the first polar body, ovoid shape, or difficult denudation. Embryo quality was moderate to poor (grade B to D), and no pregnancy was obtained after embryo transfer. After GH replacement, hormones levels increased in follicular fluid: GH [7.68 vs. 1.39 mIU/L], IGF1 [109 vs. <25ng/mL], fT3 [3.7 vs. 2.5pmol/L] and fT4 [1.45 vs. 0.84ng/mL]. Concomitantly, there was dramatic improvement in oocyte quality (no abnormal morphologies) and embryo quality (grade A), allowing an embryo transfer with successful pregnancy.
CONCLUSIONS CONCLUSIONS
This is the first report illustrating changes in hormonal levels in follicular fluid and the beneficial effect of GH replacement on oocyte and embryo quality during an IVF procedure in a patient with hypopituitarism. These results suggest that GH replacement is beneficial for oocyte quality in patients with GHD.

Identifiants

pubmed: 34186075
pii: S0003-4266(21)00077-9
doi: 10.1016/j.ando.2021.05.003
pii:
doi:

Substances chimiques

Human Growth Hormone 12629-01-5

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

590-596

Informations de copyright

Copyright © 2021 Elsevier Masson SAS. All rights reserved.

Auteurs

Florence Scheffler (F)

Reproductive Medicine and Biology Department, Cytogenetics and CECOS of Picardy, Amiens University Hospital, Picardy, France; Peritox UMR_I 01, CURS, Jules Verne University of Picardy, Amiens, France. Electronic address: scheffler.florence@chu-amiens.fr.

Rosalie Cabry (R)

Reproductive Medicine and Biology Department, Cytogenetics and CECOS of Picardy, Amiens University Hospital, Picardy, France; Peritox UMR_I 01, CURS, Jules Verne University of Picardy, Amiens, France. Electronic address: cabry.rosalie@chu-amiens.fr.

Marion Soyez (M)

Endocrine and Bone Biology Department, Amiens University Hospital, Picardy, France. Electronic address: soyez.marion@chu-amiens.fr.

Henri Copin (H)

Reproductive Medicine and Biology Department, Cytogenetics and CECOS of Picardy, Amiens University Hospital, Picardy, France. Electronic address: copin.henri@chu-amiens.fr.

Moncef Ben Khalifa (M)

Reproductive Medicine and Biology Department, Cytogenetics and CECOS of Picardy, Amiens University Hospital, Picardy, France; Peritox UMR_I 01, CURS, Jules Verne University of Picardy, Amiens, France. Electronic address: benkhalifa.moncef@chu-amiens.fr.

Aviva Devaux (A)

Reproductive Medicine and Biology Department, Cytogenetics and CECOS of Picardy, Amiens University Hospital, Picardy, France; Peritox UMR_I 01, CURS, Jules Verne University of Picardy, Amiens, France. Electronic address: devaux.aviva@chu-amiens.fr.

Rachel Desailloud (R)

Endocrinology, Diabetes, and Nutrition Department, Amiens University Hospital, Picardy, France; Peritox UMR_I 01, CURS, Jules Verne University of Picardy, Amiens, France. Electronic address: desailloud.rachel@chu-amiens.fr.

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