Contact-dependent inhibition of HIV-1 replication in


Journal

Cell reports. Medicine
ISSN: 2666-3791
Titre abrégé: Cell Rep Med
Pays: United States
ID NLM: 101766894

Informations de publication

Date de publication:
15 06 2021
Historique:
received: 24 07 2020
revised: 02 03 2021
accepted: 20 05 2021
entrez: 1 7 2021
pubmed: 2 7 2021
medline: 2 7 2021
Statut: epublish

Résumé

Polymorphonuclear neutrophils (PMNs), the most abundant white blood cells, are recruited rapidly to sites of infection to exert potent anti-microbial activity. Information regarding their role in infection with human immunodeficiency virus (HIV) is limited. Here we report that addition of PMNs to HIV-infected cultures of human tonsil tissue or peripheral blood mononuclear cells causes immediate and long-lasting suppression of HIV-1 spread and virus-induced depletion of CD4 T cells. This inhibition of HIV-1 spread strictly requires PMN contact with infected cells and is not mediated by soluble factors. 2-Photon (2PM) imaging visualized contacts of PMNs with HIV-1-infected CD4 T cells in tonsil tissue that do not result in lysis or uptake of infected cells. The anti-HIV activity of PMNs also does not involve degranulation, formation of neutrophil extracellular traps, or integrin-dependent cell communication. These results reveal that PMNs efficiently blunt HIV-1 replication in primary target cells and tissue by an unconventional mechanism.

Identifiants

pubmed: 34195682
doi: 10.1016/j.xcrm.2021.100317
pii: S2666-3791(21)00160-9
pmc: PMC8233696
doi:

Substances chimiques

Integrins 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Pagination

100317

Informations de copyright

© 2021 The Author(s).

Déclaration de conflit d'intérêts

The authors declare no competing interests.

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Auteurs

Tatjana Reif (T)

Department of Infectious Diseases, Integrative Virology, University Hospital Heidelberg, 69120 Heidelberg, Germany.

Gerhard Dyckhoff (G)

Department of Otorhinolaryngology, Head and Neck Surgery, University Hospital Heidelberg, 69120 Heidelberg, Germany.

Ralph Hohenberger (R)

Department of Otorhinolaryngology, Head and Neck Surgery, University Hospital Heidelberg, 69120 Heidelberg, Germany.

Carl-Christian Kolbe (CC)

Institute of Innate Immunity, Department of Innate Immunity and Metaflammation, University Hospital Bonn, 53127 Bonn, Germany.

Henning Gruell (H)

Laboratory of Experimental Immunology, Institute of Virology, Faculty of Medicine and University Hospital Cologne, University of Cologne, 50931 Cologne, Germany.
German Center for Infection Research (DZIF), Partner Site Bonn-Cologne, 50931 Cologne, Germany.

Florian Klein (F)

Laboratory of Experimental Immunology, Institute of Virology, Faculty of Medicine and University Hospital Cologne, University of Cologne, 50931 Cologne, Germany.
German Center for Infection Research (DZIF), Partner Site Bonn-Cologne, 50931 Cologne, Germany.

Eicke Latz (E)

Institute of Innate Immunity, Department of Innate Immunity and Metaflammation, University Hospital Bonn, 53127 Bonn, Germany.

Bettina Stolp (B)

Department of Infectious Diseases, Integrative Virology, University Hospital Heidelberg, 69120 Heidelberg, Germany.

Oliver T Fackler (OT)

Department of Infectious Diseases, Integrative Virology, University Hospital Heidelberg, 69120 Heidelberg, Germany.
German Center for Infection Research (DZIF), Partner Site Heidelberg, 69120 Heidelberg, Germany.

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