Chemotherapy Shifts the Balance in Favor of CD8+ TNFR2+ TILs in Triple-Negative Breast Tumors.


Journal

Cells
ISSN: 2073-4409
Titre abrégé: Cells
Pays: Switzerland
ID NLM: 101600052

Informations de publication

Date de publication:
08 06 2021
Historique:
received: 28 04 2021
revised: 31 05 2021
accepted: 02 06 2021
entrez: 2 7 2021
pubmed: 3 7 2021
medline: 30 10 2021
Statut: epublish

Résumé

Triple-negative breast cancer (TNBC) is primarily treated via chemotherapy; in parallel, efforts are made to introduce immunotherapies into TNBC treatment. CD4+ TNFR2+ lymphocytes were reported as Tregs that contribute to tumor progression. However, our published study indicated that TNFR2+ tumor-infiltrating lymphocytes (TNFR2+ TILs) were associated with improved survival in TNBC patient tumors. Based on our analyses of the contents of CD4+ and CD8+ TILs in TNBC patient tumors, in the current study, we determined the impact of chemotherapy on CD4+ and CD8+ TIL subsets in TNBC mouse tumors. We found that chemotherapy led to (1) a reduction in CD4+ TNFR2+ FOXP3+ TILs, indicating that chemotherapy decreased the content of CD4+ TNFR2+ Tregs, and (2) an elevation in CD8+ TNFR2+ and CD8+ TNFR2+ PD-1+ TILs; high levels of these two subsets were significantly associated with reduced tumor growth. In spleens of tumor-bearing mice, chemotherapy down-regulated CD4+ TNFR2+ FOXP3+ cells but the subset of CD8+ TNFR2+ PD-1+ was not present prior to chemotherapy and was not increased by the treatment. Thus, our data suggest that chemotherapy promotes the proportion of protective CD8+ TNFR2+ TILs and that, unlike other cancer types, therapeutic strategies directed against TNFR2 may be detrimental in TNBC.

Identifiants

pubmed: 34201054
pii: cells10061429
doi: 10.3390/cells10061429
pmc: PMC8229590
pii:
doi:

Substances chimiques

Biomarkers, Tumor 0
Receptors, Tumor Necrosis Factor, Type II 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Israel Cancer Research Fund
ID : N/A
Organisme : Federico Foundation
ID : N/A

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Auteurs

Tamir Baram (T)

George S. Wise Faculty of Life Sciences, The Shmunis School of Biomedicine and Cancer Research, Tel Aviv University, Tel Aviv 69978-01, Israel.

Nofar Erlichman (N)

George S. Wise Faculty of Life Sciences, The Shmunis School of Biomedicine and Cancer Research, Tel Aviv University, Tel Aviv 69978-01, Israel.

Maya Dadiani (M)

Sheba Medical Center, Breast Oncology Institute, Ramat Gan 5211401, Israel.

Nora Balint-Lahat (N)

Sheba Medical Center, Pathology Institute, Ramat Gan 5211401, Israel.

Anya Pavlovski (A)

Sheba Medical Center, Pathology Institute, Ramat Gan 5211401, Israel.

Tsipi Meshel (T)

George S. Wise Faculty of Life Sciences, The Shmunis School of Biomedicine and Cancer Research, Tel Aviv University, Tel Aviv 69978-01, Israel.

Dana Morzaev-Sulzbach (D)

Sheba Medical Center, Breast Oncology Institute, Ramat Gan 5211401, Israel.

Einav Nili Gal-Yam (EN)

Sheba Medical Center, Breast Oncology Institute, Ramat Gan 5211401, Israel.

Iris Barshack (I)

Sheba Medical Center, Pathology Institute, Ramat Gan 5211401, Israel.
Sackler School of Medicine, Tel Aviv University, Tel Aviv 69978-01, Israel.

Adit Ben-Baruch (A)

George S. Wise Faculty of Life Sciences, The Shmunis School of Biomedicine and Cancer Research, Tel Aviv University, Tel Aviv 69978-01, Israel.

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