Oral Treatment with RD2RD2 Impedes Development of Motoric Phenotype and Delays Symptom Onset in SOD1
SOD1G93A
amyotrophic lateral sclerosis
behaviour
d-enantiomeric peptide
motor coordination
neuroinflammation
plasma cytokines
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
30 Jun 2021
30 Jun 2021
Historique:
received:
05
06
2021
revised:
25
06
2021
accepted:
25
06
2021
entrez:
2
7
2021
pubmed:
3
7
2021
medline:
22
7
2021
Statut:
epublish
Résumé
Neuroinflammation is a pathological hallmark of several neurodegenerative disorders and plays a key role in the pathogenesis of amyotrophic lateral sclerosis (ALS). It has been implicated as driver of disease progression and is observed in ALS patients, as well as in the transgenic SOD1
Identifiants
pubmed: 34209129
pii: ijms22137066
doi: 10.3390/ijms22137066
pmc: PMC8269060
pii:
doi:
Substances chimiques
Peptides
0
SOD1 G93A protein
EC 1.15.1.1
Superoxide Dismutase
EC 1.15.1.1
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Russian Science Foundation
ID : 20-64-46027
Organisme : Forschungszentrum Jülich
ID : Technology Transfer Fund
Organisme : Impuls und Vernetzungs-Fonds der Helmholtzgemeinschaft
ID : Portfolio Drug Research
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