Short- and Long-Lived Autoantibody-Secreting Cells in Autoimmune Neurological Disorders.
IgG4
autoantibody-mediated disorders
long-lived plasma cells
neurological autoimmunity
rituximab
short-lived
Journal
Frontiers in immunology
ISSN: 1664-3224
Titre abrégé: Front Immunol
Pays: Switzerland
ID NLM: 101560960
Informations de publication
Date de publication:
2021
2021
Historique:
received:
26
03
2021
accepted:
28
05
2021
entrez:
5
7
2021
pubmed:
6
7
2021
medline:
29
10
2021
Statut:
epublish
Résumé
As B cells differentiate into antibody-secreting cells (ASCs), short-lived plasmablasts (SLPBs) are produced by a primary extrafollicular response, followed by the generation of memory B cells and long-lived plasma cells (LLPCs) in germinal centers (GCs). Generation of IgG4 antibodies is T helper type 2 (Th2) and IL-4, -13, and -10-driven and can occur parallel to IgE, in response to chronic stimulation by allergens and helminths. Although IgG4 antibodies are non-crosslinking and have limited ability to mobilize complement and cellular cytotoxicity, when self-tolerance is lost, they can disrupt ligand-receptor binding and cause a wide range of autoimmune disorders including neurological autoimmunity. In myasthenia gravis with predominantly IgG4 autoantibodies against muscle-specific kinase (MuSK), it has been observed that one-time CD20
Identifiants
pubmed: 34220839
doi: 10.3389/fimmu.2021.686466
pmc: PMC8248361
doi:
Substances chimiques
Autoantibodies
0
Immunoglobulin G
0
Receptors, Cholinergic
0
Rituximab
4F4X42SYQ6
MUSK protein, human
EC 2.7.10.1
Receptor Protein-Tyrosine Kinases
EC 2.7.10.1
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
686466Informations de copyright
Copyright © 2021 Zografou, Vakrakou and Stathopoulos.
Déclaration de conflit d'intérêts
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
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