Sample size calculation for phylogenetic case linkage.


Journal

PLoS computational biology
ISSN: 1553-7358
Titre abrégé: PLoS Comput Biol
Pays: United States
ID NLM: 101238922

Informations de publication

Date de publication:
07 2021
Historique:
received: 02 12 2020
accepted: 14 06 2021
revised: 16 07 2021
pubmed: 7 7 2021
medline: 26 10 2021
entrez: 6 7 2021
Statut: epublish

Résumé

Sample size calculations are an essential component of the design and evaluation of scientific studies. However, there is a lack of clear guidance for determining the sample size needed for phylogenetic studies, which are becoming an essential part of studying pathogen transmission. We introduce a statistical framework for determining the number of true infector-infectee transmission pairs identified by a phylogenetic study, given the size and population coverage of that study. We then show how characteristics of the criteria used to determine linkage and aspects of the study design can influence our ability to correctly identify transmission links, in sometimes counterintuitive ways. We test the overall approach using outbreak simulations and provide guidance for calculating the sensitivity and specificity of the linkage criteria, the key inputs to our approach. The framework is freely available as the R package phylosamp, and is broadly applicable to designing and evaluating a wide array of pathogen phylogenetic studies.

Identifiants

pubmed: 34228722
doi: 10.1371/journal.pcbi.1009182
pii: PCOMPBIOL-D-20-02147
pmc: PMC8284614
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e1009182

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

Références

Nature. 2015 Aug 6;524(7563):97-101
pubmed: 26083749
PLoS Pathog. 2007 Sep 14;3(9):1220-8
pubmed: 17941707
Lancet HIV. 2017 Jan;4(1):e8-e9
pubmed: 27914876
PLoS Biol. 2020 Feb 11;18(2):e3000611
pubmed: 32045407
Epidemics. 2016 Dec;17:10-18
pubmed: 27744095
Bioinformatics. 2018 Dec 1;34(23):4121-4123
pubmed: 29790939
Virus Evol. 2016 Mar 02;2(1):vew003
pubmed: 27774296
Nature. 2019 Jan;565(7738):230-233
pubmed: 30602788
Acad Radiol. 2013 Jul;20(7):807-15
pubmed: 23582776
Proc Natl Acad Sci U S A. 2012 Jun 12;109(24):9535-8
pubmed: 22645364
PLoS One. 2020 Nov 11;15(11):e0242128
pubmed: 33175914
Stat Med. 2012 Oct 15;31(23):2676-86
pubmed: 22307964
Genome Med. 2014 Nov 15;6(11):101
pubmed: 25484920
Cancer. 1950 Jan;3(1):32-5
pubmed: 15405679
Am J Epidemiol. 2014 Nov 1;180(9):865-75
pubmed: 25294601
Cell Host Microbe. 2018 Jun 13;23(6):855-864.e7
pubmed: 29805095
Virus Evol. 2018 Jan 08;4(1):vex042
pubmed: 29340210
PLoS Pathog. 2018 Feb 8;14(2):e1006885
pubmed: 29420641
PLoS Comput Biol. 2014 Jan;10(1):e1003457
pubmed: 24465202
Am J Epidemiol. 2006 Apr 1;163(7):670-5
pubmed: 16410346
N Engl J Med. 2011 Feb 24;364(8):730-9
pubmed: 21345102
Bioinformatics. 2015 Aug 1;31(15):2461-8
pubmed: 25819674
J R Soc Interface. 2012 Aug 7;9(73):1797-808
pubmed: 22337627
Science. 2020 May 8;368(6491):
pubmed: 32234805
PLoS Comput Biol. 2013;9(12):e1003397
pubmed: 24367249
Genetics. 2014 Dec;198(4):1395-404
pubmed: 25313129
Cell. 2015 Jun 18;161(7):1516-26
pubmed: 26091036
MMWR Morb Mortal Wkly Rep. 2016 Apr 08;65(13):346-51
pubmed: 27054558
Science. 2020 Jul 17;369(6501):297-301
pubmed: 32471856
Proc Biol Sci. 2012 Feb 7;279(1728):444-50
pubmed: 21733899
J Mol Evol. 2002 Feb;54(2):156-65
pubmed: 11821909
Epidemics. 2015 Mar;10:88-92
pubmed: 25843391
Nature. 2016 Feb 11;530(7589):228-232
pubmed: 26840485
Clin Infect Dis. 2016 Aug 1;63(3):380-6
pubmed: 27090985
Microb Genom. 2016 Nov 30;2(11):e000094
pubmed: 28348834
J R Soc Interface. 2010 Jul 6;7(48):1119-27
pubmed: 20147314
PLoS Comput Biol. 2017 May 18;13(5):e1005495
pubmed: 28545083
Lancet HIV. 2020 Mar;7(3):e173-e183
pubmed: 31953184
PLoS Comput Biol. 2012;8(11):e1002768
pubmed: 23166481
PLoS One. 2020 Sep 24;15(9):e0239800
pubmed: 32970786
J Clin Microbiol. 2018 Oct 25;56(11):
pubmed: 30135232
Curr Genomics. 2011 Nov;12(7):466-74
pubmed: 22547954
Infect Dis Model. 2017 Jun 29;2(3):288-303
pubmed: 29928743

Auteurs

Shirlee Wohl (S)

Johns Hopkins Bloomberg School of Public Health, Department of Epidemiology, Baltimore, Maryland, United States of America.

John R Giles (JR)

Johns Hopkins Bloomberg School of Public Health, Department of Epidemiology, Baltimore, Maryland, United States of America.

Justin Lessler (J)

Johns Hopkins Bloomberg School of Public Health, Department of Epidemiology, Baltimore, Maryland, United States of America.

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Classifications MeSH