Levosimendan increases brain tissue oxygen levels after cardiopulmonary resuscitation independent of cardiac function and cerebral perfusion.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
09 07 2021
Historique:
received: 18 05 2021
accepted: 28 06 2021
entrez: 10 7 2021
pubmed: 11 7 2021
medline: 9 11 2021
Statut: epublish

Résumé

Prompt reperfusion is important to rescue ischemic tissue; however, the process itself presents a key pathomechanism that contributes to a poor outcome following cardiac arrest. Experimental data have suggested the use of levosimendan to limit ischemia-reperfusion injury by improving cerebral microcirculation. However, recent studies have questioned this effect. The present study aimed to investigate the influence on hemodynamic parameters, cerebral perfusion and oxygenation following cardiac arrest by ventricular fibrillation in juvenile male pigs. Following the return of spontaneous circulation (ROSC), animals were randomly assigned to levosimendan (12 µg/kg, followed by 0.3 µg/kg/min) or vehicle treatment for 6 h. Levosimendan-treated animals showed significantly higher brain PbtO

Identifiants

pubmed: 34244561
doi: 10.1038/s41598-021-93621-x
pii: 10.1038/s41598-021-93621-x
pmc: PMC8270955
doi:

Substances chimiques

Simendan 349552KRHK
Oxygen S88TT14065

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

14220

Informations de copyright

© 2021. The Author(s).

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Auteurs

Andreas García-Bardon (A)

Department of Anesthesiology, Medical Center, Johannes Gutenberg-University, Langenbeckstraße 1, 55131, Mainz, Germany. a.garciabardon@uni-mainz.de.

Jens Kamuf (J)

Department of Anesthesiology, Medical Center, Johannes Gutenberg-University, Langenbeckstraße 1, 55131, Mainz, Germany.

Alexander Ziebart (A)

Department of Anesthesiology, Medical Center, Johannes Gutenberg-University, Langenbeckstraße 1, 55131, Mainz, Germany.

Tanghua Liu (T)

Department of Anesthesiology, Medical Center, Johannes Gutenberg-University, Langenbeckstraße 1, 55131, Mainz, Germany.

Nadia Krebs (N)

Department of Anesthesiology, Medical Center, Johannes Gutenberg-University, Langenbeckstraße 1, 55131, Mainz, Germany.

Bastian Dünges (B)

Department of Anesthesiology, Medical Center, Johannes Gutenberg-University, Langenbeckstraße 1, 55131, Mainz, Germany.

Robert F Kelm (RF)

Department of Anesthesiology, Medical Center, Johannes Gutenberg-University, Langenbeckstraße 1, 55131, Mainz, Germany.

Svenja Morsbach (S)

Max Planck Institute for Polymer Research, Ackermannweg 10, 55128, Mainz, Germany.

Kristin Mohr (K)

Max Planck Institute for Polymer Research, Ackermannweg 10, 55128, Mainz, Germany.

Axel Heimann (A)

Institute for Neurosurgical Pathophysiology, Medical Center, Johannes Gutenberg-University, Mainz, Germany.

Erik K Hartmann (EK)

Department of Anesthesiology, Medical Center, Johannes Gutenberg-University, Langenbeckstraße 1, 55131, Mainz, Germany.

Serge C Thal (SC)

Department of Anesthesiology, Medical Center, Johannes Gutenberg-University, Langenbeckstraße 1, 55131, Mainz, Germany. serge@thal.de.
Department of Anesthesiology, HELIOS University Hospital Wuppertal, Witten/Herdecke University, Heusnerstraße 40, 42283, Wuppertal, Germany. serge@thal.de.

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Classifications MeSH