Does chronic dietary exposure to the mycotoxin deoxynivalenol affect the porcine hepatic transcriptome when an acute-phase response is initiated through first or second-pass LPS challenge of the liver?
Deoxynivalenol
Sus scrofa
acute-phase response
first-pass LPS exposure
hepatic transcriptome
second-pass LPS exposure
systemic inflammation
Journal
Innate immunity
ISSN: 1753-4267
Titre abrégé: Innate Immun
Pays: United States
ID NLM: 101469670
Informations de publication
Date de publication:
07 2021
07 2021
Historique:
pubmed:
3
8
2021
medline:
9
3
2022
entrez:
2
8
2021
Statut:
ppublish
Résumé
The sensitivity of pigs to deoxynivalenol (DON) might be increased by systemic inflammation (SI), which also has consequences for hepatic integrity. Liver lesions and a dys-regulated gene network might hamper hepatic handling and elimination of DON whereby the way of initiation of hepatic inflammation might play an additional role. First and second-pass exposure of the liver with LPS for triggering a SI was achieved by LPS infusion
Identifiants
pubmed: 34338001
doi: 10.1177/17534259211030563
pmc: PMC8419296
doi:
Substances chimiques
Lipopolysaccharides
0
Mycotoxins
0
Trichothecenes
0
deoxynivalenol
JT37HYP23V
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
388-408Références
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