A Reassessment of Copy Number Variations in Congenital Heart Defects: Picturing the Whole Genome.


Journal

Genes
ISSN: 2073-4425
Titre abrégé: Genes (Basel)
Pays: Switzerland
ID NLM: 101551097

Informations de publication

Date de publication:
08 07 2021
Historique:
received: 08 06 2021
revised: 03 07 2021
accepted: 05 07 2021
entrez: 6 8 2021
pubmed: 7 8 2021
medline: 2 2 2022
Statut: epublish

Résumé

Copy number variations (CNVs) can modulate phenotypes by affecting protein-coding sequences directly or through interference of gene expression. Recent studies in cancer and limb defects pinpointed the relevance of non-coding gene regulatory elements such as long non-coding RNAs (lncRNAs) and topologically associated domain (TAD)-related gene-enhancer interactions. The contribution of such non-coding elements is largely unexplored in congenital heart defects (CHD). We performed a retrospective analysis of CNVs reported in a cohort of 270 CHD patients. We reviewed the diagnostic yield of pathogenic CNVs, and performed a comprehensive reassessment of 138 CNVs of unknown significance (CNV-US), evaluating protein-coding genes, lncRNA genes, and potential interferences with TAD-related gene-enhancer interactions. Fifty-two of the 138 CNV-US may relate to CHD, revealing three candidate CHD regions, 19 candidate CHD genes, 80 lncRNA genes of interest, and six potentially CHD-related TAD interferences. Our study thus indicates a potential relevance of non-coding gene regulatory elements in CNV-related CHD pathogenesis. Shortcomings in our current knowledge on genomic variation call for continuous reporting of CNV-US in international databases, careful patient counseling, and additional functional studies to confirm these preliminary findings.

Identifiants

pubmed: 34356064
pii: genes12071048
doi: 10.3390/genes12071048
pmc: PMC8304049
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Wellcome Trust
Pays : United Kingdom

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Auteurs

Ilse Meerschaut (I)

Center for Medical Genetics, Ghent University Hospital, Belgium and Department of Biomolecular Medicine, Ghent University, 9000 Ghent, Belgium.
Department of Pediatric Cardiology, Ghent University Hospital, 9000 Ghent, Belgium.

Sarah Vergult (S)

Center for Medical Genetics, Ghent University Hospital, Belgium and Department of Biomolecular Medicine, Ghent University, 9000 Ghent, Belgium.

Annelies Dheedene (A)

Center for Medical Genetics, Ghent University Hospital, Belgium and Department of Biomolecular Medicine, Ghent University, 9000 Ghent, Belgium.

Björn Menten (B)

Center for Medical Genetics, Ghent University Hospital, Belgium and Department of Biomolecular Medicine, Ghent University, 9000 Ghent, Belgium.

Katya De Groote (K)

Department of Pediatric Cardiology, Ghent University Hospital, 9000 Ghent, Belgium.

Hans De Wilde (H)

Department of Pediatric Cardiology, Ghent University Hospital, 9000 Ghent, Belgium.

Laura Muiño Mosquera (L)

Center for Medical Genetics, Ghent University Hospital, Belgium and Department of Biomolecular Medicine, Ghent University, 9000 Ghent, Belgium.
Department of Pediatric Cardiology, Ghent University Hospital, 9000 Ghent, Belgium.

Joseph Panzer (J)

Department of Pediatric Cardiology, Ghent University Hospital, 9000 Ghent, Belgium.

Kristof Vandekerckhove (K)

Department of Pediatric Cardiology, Ghent University Hospital, 9000 Ghent, Belgium.

Paul J Coucke (PJ)

Center for Medical Genetics, Ghent University Hospital, Belgium and Department of Biomolecular Medicine, Ghent University, 9000 Ghent, Belgium.

Daniël De Wolf (D)

Department of Pediatric Cardiology, Ghent University Hospital, 9000 Ghent, Belgium.
Department of Pediatric Cardiology, Brussels University Hospital, 1090 Brussels, Belgium.

Bert Callewaert (B)

Center for Medical Genetics, Ghent University Hospital, Belgium and Department of Biomolecular Medicine, Ghent University, 9000 Ghent, Belgium.

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Classifications MeSH