Deletion of heterogeneous nuclear ribonucleoprotein F in renal tubules downregulates SGLT2 expression and attenuates hyperfiltration and kidney injury in a mouse model of diabetes.


Journal

Diabetologia
ISSN: 1432-0428
Titre abrégé: Diabetologia
Pays: Germany
ID NLM: 0006777

Informations de publication

Date de publication:
11 2021
Historique:
received: 11 02 2021
accepted: 26 05 2021
pubmed: 10 8 2021
medline: 26 3 2022
entrez: 9 8 2021
Statut: ppublish

Résumé

We previously reported that renal tubule-specific deletion of heterogeneous nuclear ribonucleoprotein F (Hnrnpf) results in upregulation of renal angiotensinogen (Agt) and downregulation of sodium-glucose co-transporter 2 (Sglt2) in Hnrnpf Akita Hnrnpf Akita mice exhibited elevated systolic blood pressure as compared with Ctrl mice, which was significantly higher in Akita Hnrnpf The renoprotective effect of Sglt2 downregulation overcomes the renal injurious effect of Agt when these opposing factors coexist under diabetic conditions, at least partly via the activation of tubuloglomerular feedback.

Identifiants

pubmed: 34370045
doi: 10.1007/s00125-021-05538-9
pii: 10.1007/s00125-021-05538-9
pmc: PMC8992778
mid: NIHMS1792369
doi:

Substances chimiques

Agt protein, mouse 0
Blood Glucose 0
Heterogeneous-Nuclear Ribonucleoprotein Group F-H 0
Purinergic P1 Receptor Antagonists 0
Slc5a2 protein, mouse 0
Sodium-Glucose Transporter 2 0
Angiotensinogen 11002-13-4
8-cyclopentyl-1,3-dimethylxanthine 35873-49-5
Theophylline C137DTR5RG

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2589-2601

Subventions

Organisme : NCATS NIH HHS
ID : KL2 TR001882
Pays : United States
Organisme : CIHR
ID : PJT 173512
Pays : Canada
Organisme : CIHR
ID : MOP-84363
Pays : Canada
Organisme : CIHR
ID : MOP-142378
Pays : Canada
Organisme : CIHR
ID : MOP-97742
Pays : Canada

Informations de copyright

© 2021. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany, part of Springer Nature.

Références

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Auteurs

Kana N Miyata (KN)

Département de Médecine, Université de Montréal, Centre de recherche du Centre hospitalier de l'Université de Montréal (CRCHUM), Montréal, QC, Canada.
Division of Nephrology, Department of Internal Medicine, Saint Louis University, St. Louis, MO, USA.

Chao-Sheng Lo (CS)

Département de Médecine, Université de Montréal, Centre de recherche du Centre hospitalier de l'Université de Montréal (CRCHUM), Montréal, QC, Canada.

Shuiling Zhao (S)

Département de Médecine, Université de Montréal, Centre de recherche du Centre hospitalier de l'Université de Montréal (CRCHUM), Montréal, QC, Canada.

Xin-Ping Zhao (XP)

Département de Médecine, Université de Montréal, Centre de recherche du Centre hospitalier de l'Université de Montréal (CRCHUM), Montréal, QC, Canada.

Isabelle Chenier (I)

Département de Médecine, Université de Montréal, Centre de recherche du Centre hospitalier de l'Université de Montréal (CRCHUM), Montréal, QC, Canada.

Michifumi Yamashita (M)

Department of Pathology and Laboratory Medicine, Cedars-Sinai Medical Center, Los Angeles, CA, USA.

Janos G Filep (JG)

Université de Montréal, Centre de recherche de l'Hopital Maisonneuve-Rosemont, Montréal, QC, Canada.

Julie R Ingelfinger (JR)

Harvard Medical School, Pediatric Nephrology Unit, Massachusetts General Hospital, Boston, MA, USA.

Shao-Ling Zhang (SL)

Département de Médecine, Université de Montréal, Centre de recherche du Centre hospitalier de l'Université de Montréal (CRCHUM), Montréal, QC, Canada. shao.ling.zhang@umontreal.ca.

John S D Chan (JSD)

Département de Médecine, Université de Montréal, Centre de recherche du Centre hospitalier de l'Université de Montréal (CRCHUM), Montréal, QC, Canada. john.chan@umontreal.ca.

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Classifications MeSH