Role of Mitochondrial Dynamics in Microglial Activation and Metabolic Switch.


Journal

ImmunoHorizons
ISSN: 2573-7732
Titre abrégé: Immunohorizons
Pays: United States
ID NLM: 101708159

Informations de publication

Date de publication:
10 08 2021
Historique:
received: 19 07 2021
accepted: 20 07 2021
entrez: 11 8 2021
pubmed: 12 8 2021
medline: 29 1 2022
Statut: epublish

Résumé

Microglia act as sensors of injury in the brain, favoring its homeostasis. Their activation and polarization toward a proinflammatory phenotype are associated with injury and disease. These processes are linked to a metabolic reprogramming of the cells, characterized by high rates of glycolysis and suppressed oxidative phosphorylation. This metabolic switch can be reproduced in vitro by microglial stimulation with LPS plus IFN-γ. To understand the mechanisms regulating mitochondrial respiration abolishment, we examined potential alterations in mitochondrial features during this switch using rat primary microglia. Cells did not show any change in mitochondrial membrane potential, suggesting a limited impact in the mitochondrial viability. We provide evidence that reverse operation of F

Identifiants

pubmed: 34376483
pii: immunohorizons.2100068
doi: 10.4049/immunohorizons.2100068
doi:

Substances chimiques

3-(2,4-dichloro-5-methoxyphenyl)-2-sulfanyl-4(3H)-quinazolinone 0
Lipopolysaccharides 0
Quinazolinones 0
Reactive Oxygen Species 0
Interferon-gamma 82115-62-6
Proton-Translocating ATPases EC 3.6.3.14
Dnm1l protein, rat EC 3.6.5.5
Dynamins EC 3.6.5.5
Calcium SY7Q814VUP

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

615-626

Informations de copyright

Copyright © 2021 The Authors.

Auteurs

Alejandro Montilla (A)

Achucarro Basque Center for Neuroscience and Department of Neuroscience, University of the Basque Country, Leioa, Spain.
Centro de Investigación Biomédica en Red de Enfermedades Neurodegenerativas, Leioa, Spain; and.

Asier Ruiz (A)

Achucarro Basque Center for Neuroscience and Department of Neuroscience, University of the Basque Country, Leioa, Spain.
Centro de Investigación Biomédica en Red de Enfermedades Neurodegenerativas, Leioa, Spain; and.

Mar Marquez (M)

Achucarro Basque Center for Neuroscience and Department of Neuroscience, University of the Basque Country, Leioa, Spain.

Amanda Sierra (A)

Achucarro Basque Center for Neuroscience and Department of Neuroscience, University of the Basque Country, Leioa, Spain.
Ikerbasque Foundation, Bilbao, Spain.

Carlos Matute (C)

Achucarro Basque Center for Neuroscience and Department of Neuroscience, University of the Basque Country, Leioa, Spain.
Centro de Investigación Biomédica en Red de Enfermedades Neurodegenerativas, Leioa, Spain; and.

Maria Domercq (M)

Achucarro Basque Center for Neuroscience and Department of Neuroscience, University of the Basque Country, Leioa, Spain; maria.domercq@ehu.es.
Centro de Investigación Biomédica en Red de Enfermedades Neurodegenerativas, Leioa, Spain; and.

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Classifications MeSH