Effect of Rosuvastatin Therapy on Biomarkers of Inflammation and Immune Activation in People With Human Immunodeficiency Virus at Intermediate Cardiovascular Risk.


Journal

The Journal of infectious diseases
ISSN: 1537-6613
Titre abrégé: J Infect Dis
Pays: United States
ID NLM: 0413675

Informations de publication

Date de publication:
16 08 2021
Historique:
received: 10 10 2020
accepted: 16 12 2020
entrez: 16 8 2021
pubmed: 17 8 2021
medline: 2 2 2022
Statut: ppublish

Résumé

Statins may help prevent cardiovascular disease (CVD) in people with human immunodeficiency virus (PWH) with chronic inflammation owing to their pleotropic lipid-lowering and anti-inflammatory properties. The impact of 48 weeks of rosuvastatin therapy on inflammation and immune activation in a double-blind, placebo-controlled trial in PWH at moderate cardiovascular disease risk was assessed. Rosuvastatin did not alter plasma levels of interleukin 6, soluble tumor necrosis factor receptor type 2, CXCL10, soluble CD14, or soluble vascular cellular adhesion molecule 1 (P ≥ .1 for all). Proportions of CD16+ monocyte subsets were increased in PWH receiving rosuvastatin. The potential benefits of statin use in PWH with normal lipid levels requires further clinical outcome research.

Sections du résumé

BACKGROUND
Statins may help prevent cardiovascular disease (CVD) in people with human immunodeficiency virus (PWH) with chronic inflammation owing to their pleotropic lipid-lowering and anti-inflammatory properties.
METHODS
The impact of 48 weeks of rosuvastatin therapy on inflammation and immune activation in a double-blind, placebo-controlled trial in PWH at moderate cardiovascular disease risk was assessed.
RESULTS
Rosuvastatin did not alter plasma levels of interleukin 6, soluble tumor necrosis factor receptor type 2, CXCL10, soluble CD14, or soluble vascular cellular adhesion molecule 1 (P ≥ .1 for all). Proportions of CD16+ monocyte subsets were increased in PWH receiving rosuvastatin.
CONCLUSIONS
The potential benefits of statin use in PWH with normal lipid levels requires further clinical outcome research.

Identifiants

pubmed: 34398237
pii: 6044066
doi: 10.1093/infdis/jiaa775
doi:

Substances chimiques

Biomarkers 0
Hydroxymethylglutaryl-CoA Reductase Inhibitors 0
Rosuvastatin Calcium 83MVU38M7Q

Types de publication

Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

667-672

Informations de copyright

© The Author(s) 2020. Published by Oxford University Press for the Infectious Diseases Society of America. All rights reserved. For permissions, e-mail: journals.permissions@oup.com.

Auteurs

Anna C Hearps (AC)

Life Sciences Discipline, Burnet Institute, Melbourne, Victoria, Australia.
Department of Infectious Diseases, The Alfred Hospital and Monash University, Melbourne, Victoria, Australia.

Thomas A Angelovich (TA)

Life Sciences Discipline, Burnet Institute, Melbourne, Victoria, Australia.
School of Health and Biomedical Sciences, RMIT University, Bundoora, Victoria, Australia.

Janine M Trevillyan (JM)

Department of Infectious Diseases, The Alfred Hospital and Monash University, Melbourne, Victoria, Australia.

Michelle E Wong (ME)

Life Sciences Discipline, Burnet Institute, Melbourne, Victoria, Australia.
Central Clinical School, Monash University, Melbourne, Victoria, Australia.

Alexandra Calmy (A)

Division of Infectious Diseases, HIV/AIDS Unit, Geneva University Hospitals, Geneva, Switzerland.

Jennifer F Hoy (JF)

Department of Infectious Diseases, The Alfred Hospital and Monash University, Melbourne, Victoria, Australia.

Anthony Jaworowski (A)

Life Sciences Discipline, Burnet Institute, Melbourne, Victoria, Australia.
Department of Infectious Diseases, The Alfred Hospital and Monash University, Melbourne, Victoria, Australia.
School of Health and Biomedical Sciences, RMIT University, Bundoora, Victoria, Australia.

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Classifications MeSH