Prenatal phthalate exposure and early childhood wheeze in the SELMA study.


Journal

Journal of exposure science & environmental epidemiology
ISSN: 1559-064X
Titre abrégé: J Expo Sci Environ Epidemiol
Pays: United States
ID NLM: 101262796

Informations de publication

Date de publication:
03 2022
Historique:
received: 22 02 2021
accepted: 20 08 2021
revised: 20 08 2021
pubmed: 4 9 2021
medline: 19 3 2022
entrez: 3 9 2021
Statut: ppublish

Résumé

Prenatal maternal phthalate exposure has been associated with wheeze and asthma in children, but results are inconclusive. Previous studies typically assessed exposure in late pregnancy, included only a small number of old phthalates, and assessed outcomes in children aged 5 years or older. We explored associations between 1st trimester prenatal maternal exposure to a wider range of phthalates and wheeze in early childhood. First trimester concentrations of 14 metabolites from 8 phthalates and one alternative plasticizer were quantified in first-morning void urine from 1148 mothers in the Swedish SELMA study. Associations between log-transformed metabolite concentrations and parental reported ever wheeze among 24-month-old children were investigated with logistic regression models adjusted for parental asthma/rhinitis, sex of child, maternal education, smoking, and creatinine. Metabolites of replacement phthalates di-iso-decyl phthalate (DiDP) and di-2-propylheptyl phthalate (DPHP) were associated with increased risk for wheeze (aOR 1.47, 95% CI 1.08-2.01 and aOR 1.49, 95% CI 1.04-2.15, respectively). The associations with DiDP and DPHP were stronger among children whose parents did not have asthma or rhinitis. In this group, wheeze was also associated with metabolites of butyl-benzyl phthalate (BBzP) and di-iso-nonyl phthalate (DiNP). Maternal phthalate exposure during early pregnancy may be a risk factor for wheeze in early childhood, especially among children whose parents do not have asthma or rhinitis symptoms.

Sections du résumé

BACKGROUND
Prenatal maternal phthalate exposure has been associated with wheeze and asthma in children, but results are inconclusive. Previous studies typically assessed exposure in late pregnancy, included only a small number of old phthalates, and assessed outcomes in children aged 5 years or older.
OBJECTIVE
We explored associations between 1st trimester prenatal maternal exposure to a wider range of phthalates and wheeze in early childhood.
METHODS
First trimester concentrations of 14 metabolites from 8 phthalates and one alternative plasticizer were quantified in first-morning void urine from 1148 mothers in the Swedish SELMA study. Associations between log-transformed metabolite concentrations and parental reported ever wheeze among 24-month-old children were investigated with logistic regression models adjusted for parental asthma/rhinitis, sex of child, maternal education, smoking, and creatinine.
RESULTS
Metabolites of replacement phthalates di-iso-decyl phthalate (DiDP) and di-2-propylheptyl phthalate (DPHP) were associated with increased risk for wheeze (aOR 1.47, 95% CI 1.08-2.01 and aOR 1.49, 95% CI 1.04-2.15, respectively). The associations with DiDP and DPHP were stronger among children whose parents did not have asthma or rhinitis. In this group, wheeze was also associated with metabolites of butyl-benzyl phthalate (BBzP) and di-iso-nonyl phthalate (DiNP).
SIGNIFICANCE
Maternal phthalate exposure during early pregnancy may be a risk factor for wheeze in early childhood, especially among children whose parents do not have asthma or rhinitis symptoms.

Identifiants

pubmed: 34475495
doi: 10.1038/s41370-021-00382-w
pii: 10.1038/s41370-021-00382-w
doi:

Substances chimiques

Environmental Pollutants 0
Phthalic Acids 0
Plasticizers 0
phthalic acid 6O7F7IX66E

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

303-311

Informations de copyright

© 2021. The Author(s), under exclusive licence to Springer Nature America, Inc.

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Auteurs

Anna-Sofia Preece (AS)

Department of Health Sciences, Karlstad University, Karlstad, Sweden.

Malin Knutz (M)

Department of Health Sciences, Karlstad University, Karlstad, Sweden.

Christian H Lindh (CH)

Division of Occupational and Environmental Medicine, Lund University, Lund, Sweden.

Carl-Gustaf Bornehag (CG)

Department of Health Sciences, Karlstad University, Karlstad, Sweden.
Department of Preventive Medicine, Icahn School of Medicine at Mount Sinai, New York, NY, USA.

Huan Shu (H)

Department of Health Sciences, Karlstad University, Karlstad, Sweden. huan.shu@kau.se.

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