Dopamine transporter binding in symptomatic controls and healthy volunteers: Considerations for neuroimaging trials.


Journal

NeuroImage. Clinical
ISSN: 2213-1582
Titre abrégé: Neuroimage Clin
Pays: Netherlands
ID NLM: 101597070

Informations de publication

Date de publication:
2021
Historique:
received: 26 03 2021
revised: 20 08 2021
accepted: 22 08 2021
pubmed: 6 9 2021
medline: 20 1 2022
entrez: 5 9 2021
Statut: ppublish

Résumé

To evaluate possible differences between brain dopamine transporter (DAT) binding in a group of symptomatic parkinsonism patients without dopaminergic degeneration and healthy individuals. Dopaminergic neuroimaging studies of Parkinson's disease (PD) have often used control groups formed from symptomatic patients with apparently normal striatal dopamine function. We sought to investigate whether symptomatic patients can be used to represent dopaminergically normal healthy controls. Forty healthy elderly individuals were scanned with DAT [ DAT binding in symptomatic patients was 8.6% higher in the posterior putamen than in healthy controls (p = 0.03). Binding correlated negatively with age in both groups but not with motor symptom severity, cognitive function or depression ratings. Putaminal DAT binding, as measured with [

Sections du résumé

OBJECTIVE
To evaluate possible differences between brain dopamine transporter (DAT) binding in a group of symptomatic parkinsonism patients without dopaminergic degeneration and healthy individuals.
BACKGROUND
Dopaminergic neuroimaging studies of Parkinson's disease (PD) have often used control groups formed from symptomatic patients with apparently normal striatal dopamine function. We sought to investigate whether symptomatic patients can be used to represent dopaminergically normal healthy controls.
METHODS
Forty healthy elderly individuals were scanned with DAT [
RESULTS
DAT binding in symptomatic patients was 8.6% higher in the posterior putamen than in healthy controls (p = 0.03). Binding correlated negatively with age in both groups but not with motor symptom severity, cognitive function or depression ratings.
CONCLUSIONS
Putaminal DAT binding, as measured with [

Identifiants

pubmed: 34482280
pii: S2213-1582(21)00251-5
doi: 10.1016/j.nicl.2021.102807
pmc: PMC8416950
pii:
doi:

Substances chimiques

Dopamine Plasma Membrane Transport Proteins 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

102807

Informations de copyright

Copyright © 2021 The Author(s). Published by Elsevier Inc. All rights reserved.

Auteurs

Emma A Honkanen (EA)

Clinical Neurosciences, University of Turku and Turku University Hospital, Turku, Finland; Department of Neurology, Satasairaala Central Hospital, Pori, Finland; Turku PET Centre, Turku University Hospital, Turku, Finland. Electronic address: ealhonk@utu.fi.

Mikael Eklund (M)

Clinical Neurosciences, University of Turku and Turku University Hospital, Turku, Finland; Turku PET Centre, Turku University Hospital, Turku, Finland.

Simo Nuuttila (S)

Clinical Neurosciences, University of Turku and Turku University Hospital, Turku, Finland.

Tommi Noponen (T)

Department of Clinical Physiology and Nuclear Medicine, University of Turku and Turku University Hospital, Turku, Finland; Department of Medical Physics, Turku University Hospital, Turku, Finland.

Elina Jaakkola (E)

Clinical Neurosciences, University of Turku and Turku University Hospital, Turku, Finland.

Elina Mäkinen (E)

Clinical Neurosciences, University of Turku and Turku University Hospital, Turku, Finland.

Risto Hirvilammi (R)

Department of Clinical Physiology and Nuclear Medicine, University of Turku and Turku University Hospital, Turku, Finland; Department of Medical Physics, Turku University Hospital, Turku, Finland.

Marko Seppänen (M)

Department of Clinical Physiology and Nuclear Medicine, University of Turku and Turku University Hospital, Turku, Finland.

Kari Lindholm (K)

Clinical Neurosciences, University of Turku and Turku University Hospital, Turku, Finland.

Filip Scheperjans (F)

Department of Neurology, Helsinki University Hospital and Department of Clinical Neurosciences (Neurology), University of Helsinki, Helsinki, Finland.

Riitta Parkkola (R)

Department of Radiology, University of Turku and Turku University Hospital, Turku, Finland.

Juho Joutsa (J)

Clinical Neurosciences, University of Turku and Turku University Hospital, Turku, Finland; Turku PET Centre, Turku University Hospital, Turku, Finland; Turku Brain and Mind Center, University of Turku, Turku, Finland.

Andrea Varrone (A)

Department of Clinical Neuroscience, Centre for Psychiatry Research, Karolinska Institutet and Stockholm Health Care Services, Stockholm, Sweden.

Valtteri Kaasinen (V)

Clinical Neurosciences, University of Turku and Turku University Hospital, Turku, Finland.

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