Targeting Parasympathetic Activity to Improve Autonomic Tone and Clinical Outcomes.


Journal

Physiology (Bethesda, Md.)
ISSN: 1548-9221
Titre abrégé: Physiology (Bethesda)
Pays: United States
ID NLM: 101208185

Informations de publication

Date de publication:
01 01 2022
Historique:
pubmed: 7 9 2021
medline: 1 2 2022
entrez: 6 9 2021
Statut: ppublish

Résumé

In this review we will briefly summarize the evidence that autonomic imbalance, more specifically reduced parasympathetic activity to the heart, generates and/or maintains many cardiorespiratory diseases and will discuss mechanisms and sites, from myocytes to the brain, that are potential translational targets for restoring parasympathetic activity and improving cardiorespiratory health.

Identifiants

pubmed: 34486396
doi: 10.1152/physiol.00023.2021
pmc: PMC8742722
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

39-45

Subventions

Organisme : NHLBI NIH HHS
ID : R01 HL133862
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL147279
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL146169
Pays : United States

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Auteurs

Matthew W Kay (MW)

Department of Biomedical Engineering, George Washington University, Washington, District of Columbia.

Vivek Jain (V)

Division of Pulmonary Medicine, Department of Medicine, George Washington University, Washington, District of Columbia.

Gurusher Panjrath (G)

Division of Cardiology, Department of Medicine, George Washington University, Washington, District of Columbia.

David Mendelowitz (D)

Department of Pharmacology and Physiology, George Washington University, Washington, District of Columbia.

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Classifications MeSH