Targeting Parasympathetic Activity to Improve Autonomic Tone and Clinical Outcomes.
OSA
autonomic
cardiac
heart failure
parasympathetic
Journal
Physiology (Bethesda, Md.)
ISSN: 1548-9221
Titre abrégé: Physiology (Bethesda)
Pays: United States
ID NLM: 101208185
Informations de publication
Date de publication:
01 01 2022
01 01 2022
Historique:
pubmed:
7
9
2021
medline:
1
2
2022
entrez:
6
9
2021
Statut:
ppublish
Résumé
In this review we will briefly summarize the evidence that autonomic imbalance, more specifically reduced parasympathetic activity to the heart, generates and/or maintains many cardiorespiratory diseases and will discuss mechanisms and sites, from myocytes to the brain, that are potential translational targets for restoring parasympathetic activity and improving cardiorespiratory health.
Identifiants
pubmed: 34486396
doi: 10.1152/physiol.00023.2021
pmc: PMC8742722
doi:
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
39-45Subventions
Organisme : NHLBI NIH HHS
ID : R01 HL133862
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL147279
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL146169
Pays : United States
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