Integrative transcriptomic profiling of a mouse model of hypertension-accelerated diabetic kidney disease.


Journal

Disease models & mechanisms
ISSN: 1754-8411
Titre abrégé: Dis Model Mech
Pays: England
ID NLM: 101483332

Informations de publication

Date de publication:
01 10 2021
Historique:
received: 05 05 2021
accepted: 01 09 2021
pubmed: 9 9 2021
medline: 22 3 2022
entrez: 8 9 2021
Statut: ppublish

Résumé

The current understanding of molecular mechanisms driving diabetic kidney disease (DKD) is limited, partly due to the complex structure of the kidney. To identify genes and signalling pathways involved in the progression of DKD, we compared kidney cortical versus glomerular transcriptome profiles in uninephrectomized (UNx) db/db mouse models of early-stage (UNx only) and advanced [UNxplus adeno-associated virus-mediated renin-1 overexpression (UNx-Renin)] DKD using RNAseq. Compared to normoglycemic db/m mice, db/db UNx and db/db UNx-Renin mice showed marked changes in their kidney cortical and glomerular gene expression profiles. UNx-Renin mice displayed more marked perturbations in gene components associated with the activation of the immune system and enhanced extracellular matrix remodelling, supporting histological hallmarks of progressive DKD in this model. Single-nucleus RNAseq enabled the linking of transcriptome profiles to specific kidney cell types. In conclusion, integration of RNAseq at the cortical, glomerular and single-nucleus level provides an enhanced resolution of molecular signalling pathways associated with disease progression in preclinical models of DKD, and may thus be advantageous for identifying novel therapeutic targets in DKD.

Identifiants

pubmed: 34494644
pii: 272567
doi: 10.1242/dmm.049086
pmc: PMC8560499
pii:
doi:

Substances chimiques

Renin EC 3.4.23.15

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© 2021. Published by The Company of Biologists Ltd.

Déclaration de conflit d'intérêts

Competing interests F.E.S., H.M.Æ., J.C.N., T.S., M.V.Ø., K.T.G.R. and L.N.F. are employees of Gubra ApS. N.V. and J.J. are owners of Gubra ApS.

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Auteurs

Frederikke E Sembach (FE)

Gubra ApS, Hørsholm Kongevej 11B, 2970 Hørsholm, Denmark.
Department of Clinical Medicine, University of Copenhagen, Blegdamsvej 3B, 2200 Copenhagen, Denmark.

Helene M Ægidius (HM)

Gubra ApS, Hørsholm Kongevej 11B, 2970 Hørsholm, Denmark.

Lisbeth N Fink (LN)

Gubra ApS, Hørsholm Kongevej 11B, 2970 Hørsholm, Denmark.

Thomas Secher (T)

Gubra ApS, Hørsholm Kongevej 11B, 2970 Hørsholm, Denmark.

Annemarie Aarup (A)

Gubra ApS, Hørsholm Kongevej 11B, 2970 Hørsholm, Denmark.

Jacob Jelsing (J)

Gubra ApS, Hørsholm Kongevej 11B, 2970 Hørsholm, Denmark.

Niels Vrang (N)

Gubra ApS, Hørsholm Kongevej 11B, 2970 Hørsholm, Denmark.

Bo Feldt-Rasmussen (B)

Department of Clinical Medicine, University of Copenhagen, Blegdamsvej 3B, 2200 Copenhagen, Denmark.
Department of Nephrology, Rigshospitalet, University of Copenhagen, Blegdamsvej 9, 2100 Copenhagen, Denmark.

Kristoffer T G Rigbolt (KTG)

Gubra ApS, Hørsholm Kongevej 11B, 2970 Hørsholm, Denmark.

Jens C Nielsen (JC)

Gubra ApS, Hørsholm Kongevej 11B, 2970 Hørsholm, Denmark.

Mette V Østergaard (MV)

Gubra ApS, Hørsholm Kongevej 11B, 2970 Hørsholm, Denmark.

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Classifications MeSH