Deferoxamine accelerates endothelial progenitor cell senescence and compromises angiogenesis.


Journal

Aging
ISSN: 1945-4589
Titre abrégé: Aging (Albany NY)
Pays: United States
ID NLM: 101508617

Informations de publication

Date de publication:
11 09 2021
Historique:
received: 19 02 2021
accepted: 02 08 2021
pubmed: 12 9 2021
medline: 15 1 2022
entrez: 11 9 2021
Statut: ppublish

Résumé

Senescence reduces the circulating number and angiogenic activity of endothelial progenitor cells (EPCs), and is associated with aging-related vascular diseases. However, it is very time-consuming to obtain aged cells (~1 month of repeated replication) or animals (~2 years) for senescence studies. Here, we established an accelerated senescence model by treating EPCs with deferoxamine (DFO), an FDA-approved iron chelator. Four days of low-dose (3 μM) DFO induced senescent phenotypes in EPCs, including a senescent pattern of protein expression, impaired mitochondrial bioenergetics, altered mitochondrial protein levels and compromised angiogenic activity. DFO-treated early EPCs from young and old donors (< 35 vs. > 70 years old) displayed similar senescent phenotypes, including elevated senescence-associated β-galactosidase activity and reduced relative telomere lengths, colony-forming units and adenosine triphosphate levels. To validate this accelerated senescence model

Identifiants

pubmed: 34508614
pii: 203469
doi: 10.18632/aging.203469
pmc: PMC8457614
doi:

Substances chimiques

Telomerase EC 2.7.7.49
Deferoxamine J06Y7MXW4D

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

21364-21384

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Auteurs

Yi-Nan Lee (YN)

Cardiovascular Center, Department of Internal Medicine, MacKay Memorial Hospital, Taipei 10449, Taiwan.
Department of Medical Research, MacKay Memorial Hospital, Taipei 10449, Taiwan.

Hsueh-Hsiao Wang (HH)

Department of Medicine, MacKay Medical College, New Taipei 25245, Taiwan.

Cheng-Huang Su (CH)

Cardiovascular Center, Department of Internal Medicine, MacKay Memorial Hospital, Taipei 10449, Taiwan.
Department of Medical Research, MacKay Memorial Hospital, Taipei 10449, Taiwan.
Department of Medicine, MacKay Medical College, New Taipei 25245, Taiwan.

Hsin-I Lee (HI)

Department of Medicine, MacKay Medical College, New Taipei 25245, Taiwan.

Yen-Hung Chou (YH)

Department of Medicine, MacKay Medical College, New Taipei 25245, Taiwan.
Institute of Biomedical Sciences, MacKay Medical College, New Taipei 25245, Taiwan.

Chin-Ling Hsieh (CL)

Cardiovascular Center, Department of Internal Medicine, MacKay Memorial Hospital, Taipei 10449, Taiwan.
Department of Medical Research, MacKay Memorial Hospital, Taipei 10449, Taiwan.

Wen-Ting Liu (WT)

Cardiovascular Center, Department of Internal Medicine, MacKay Memorial Hospital, Taipei 10449, Taiwan.
Department of Medical Research, MacKay Memorial Hospital, Taipei 10449, Taiwan.

Kuo-Tung Shu (KT)

Cardiovascular Center, Department of Internal Medicine, MacKay Memorial Hospital, Taipei 10449, Taiwan.
Department of Medical Research, MacKay Memorial Hospital, Taipei 10449, Taiwan.

Kai-Ting Chang (KT)

Cardiovascular Center, Department of Internal Medicine, MacKay Memorial Hospital, Taipei 10449, Taiwan.
Department of Medical Research, MacKay Memorial Hospital, Taipei 10449, Taiwan.

Hung-I Yeh (HI)

Cardiovascular Center, Department of Internal Medicine, MacKay Memorial Hospital, Taipei 10449, Taiwan.
Department of Medical Research, MacKay Memorial Hospital, Taipei 10449, Taiwan.
Department of Medicine, MacKay Medical College, New Taipei 25245, Taiwan.

Yih-Jer Wu (YJ)

Cardiovascular Center, Department of Internal Medicine, MacKay Memorial Hospital, Taipei 10449, Taiwan.
Department of Medical Research, MacKay Memorial Hospital, Taipei 10449, Taiwan.
Department of Medicine, MacKay Medical College, New Taipei 25245, Taiwan.
Institute of Biomedical Sciences, MacKay Medical College, New Taipei 25245, Taiwan.

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