Autophagy gene


Journal

American journal of physiology. Renal physiology
ISSN: 1522-1466
Titre abrégé: Am J Physiol Renal Physiol
Pays: United States
ID NLM: 100901990

Informations de publication

Date de publication:
01 11 2021
Historique:
pubmed: 21 9 2021
medline: 9 11 2021
entrez: 20 9 2021
Statut: ppublish

Résumé

Receptor-mediated albumin transport in proximal tubule epithelial cells (PTECs) is important to control proteinuria. Autophagy is an evolutionarily conserved degradation pathway, and its role in intracellular trafficking through interactions with the endocytic pathway has recently been highlighted. Here, we determined whether autophagy regulates albumin transcytosis in PTECs and suppresses albumin-induced cytotoxicity using human proximal tubule (HK-2) cells. The neonatal Fc receptor (FcRn), a receptor for albumin transcytosis, is partially colocalized with autophagosomes. Recycling of FcRn was attenuated, and FcRn accumulated in autophagy-related 7 (

Identifiants

pubmed: 34541900
doi: 10.1152/ajprenal.00172.2021
doi:

Substances chimiques

Atg5 protein, mouse 0
Atg7 protein, mouse 0
Autophagy-Related Protein 5 0
Havcr1 protein, mouse 0
Hepatitis A Virus Cellular Receptor 1 0
Histocompatibility Antigens Class I 0
Receptors, Fc 0
fluorescein isothiocyanate bovine serum albumin 0
rab7 GTP-Binding Proteins 0
rab7 GTP-binding proteins, human 0
rab7 GTP-binding proteins, mouse 0
Serum Albumin, Bovine 27432CM55Q
rab11 protein EC 3.6.1.-
rab GTP-Binding Proteins EC 3.6.5.2
ATG7 protein, human EC 6.2.1.45
Autophagy-Related Protein 7 EC 6.2.1.45
Fluorescein-5-isothiocyanate I223NX31W9
Fc receptor, neonatal TW3XAW0RCY

Banques de données

figshare
['10.6084/m9.figshare.14528991', '10.6084/m9.figshare.16404117', '10.6084/m9.figshare.16160871']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

F572-F586

Auteurs

Yushi Uchida (Y)

Department of Medicine and Clinical Science, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.

Kumiko Torisu (K)

Department of Medicine and Clinical Science, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.
Department of Integrated Therapy for Chronic Kidney Disease, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.

Kenji Ueki (K)

Department of Medicine and Clinical Science, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.

Kazuhiko Tsuruya (K)

Department of Nephrology, Nara Medical University, Nara, Japan.

Toshiaki Nakano (T)

Department of Medicine and Clinical Science, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.

Takanari Kitazono (T)

Department of Medicine and Clinical Science, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.

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Classifications MeSH