Loss of mTORC2-induced metabolic reprogramming in monocytes uncouples migration and maturation from production of proinflammatory mediators.


Journal

Journal of leukocyte biology
ISSN: 1938-3673
Titre abrégé: J Leukoc Biol
Pays: England
ID NLM: 8405628

Informations de publication

Date de publication:
05 2022
Historique:
pubmed: 30 9 2021
medline: 26 4 2022
entrez: 29 9 2021
Statut: ppublish

Résumé

Monocyte migration to the sites of inflammation and maturation into macrophages are key steps for their immune effector function. Here, we show that mechanistic target of rapamycin complex 2 (mTORC2)-dependent Akt activation is instrumental for metabolic reprogramming at the early stages of macrophage-mediated immunity. Despite an increased production of proinflammatory mediators, monocytes lacking expression of the mTORC2 component Rictor fail to efficiently migrate to inflammatory sites and fully mature into macrophages, resulting in reduced inflammatory responses in vivo. The mTORC2-dependent phosphorylation of Akt is instrumental for the enhancement of glycolysis and mitochondrial respiration, required to sustain monocyte maturation and motility. These observations are discussed in the context of therapeutic strategies aimed at selective inhibition of mTORC2 activity.

Identifiants

pubmed: 34585416
doi: 10.1002/JLB.1A0920-588R
doi:

Substances chimiques

Rapamycin-Insensitive Companion of mTOR Protein 0
Mechanistic Target of Rapamycin Complex 2 EC 2.7.11.1
Proto-Oncogene Proteins c-akt EC 2.7.11.1
Sirolimus W36ZG6FT64

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

967-980

Subventions

Organisme : British Heart Foundation
ID : RG/09/002/26425
Pays : United Kingdom
Organisme : British Heart Foundation
ID : FS/16/18/31973
Pays : United Kingdom
Organisme : British Heart Foundation
ID : PG/14/84/31136
Pays : United Kingdom
Organisme : Cancer Research UK
ID : 14355
Pays : United Kingdom
Organisme : Cancer Research UK
ID : 12888
Pays : United Kingdom
Organisme : British Heart Foundation
ID : RG/14/2/30616
Pays : United Kingdom
Organisme : British Heart Foundation
ID : CH/15/2/32064
Pays : United Kingdom
Organisme : Medical Research Council
ID : G0901084
Pays : United Kingdom
Organisme : British Heart Foundation
ID : FS/11/64/28945
Pays : United Kingdom
Organisme : British Heart Foundation
ID : RG/20/8/34995
Pays : United Kingdom

Informations de copyright

© 2021 The Authors. Journal of Leukocyte Biology published by Wiley Periodicals LLC on behalf of Society for Leukocyte Biology.

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Auteurs

Maryam Jangani (M)

William Harvey Research Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, Charterhouse Square, London, UK.

Juho Vuononvirta (J)

William Harvey Research Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, Charterhouse Square, London, UK.

Lamya Yamani (L)

Barts Cancer Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, Charterhouse Square, London, UK.

Eleanor Ward (E)

William Harvey Research Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, Charterhouse Square, London, UK.

Melania Capasso (M)

Barts Cancer Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, Charterhouse Square, London, UK.
German Center for Neurodegenerative Diseases (DZNE) within the Helmholtz Association, Bonn, Germany.

Suchita Nadkarni (S)

William Harvey Research Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, Charterhouse Square, London, UK.

Frances Balkwill (F)

Barts Cancer Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, Charterhouse Square, London, UK.

Federica Marelli-Berg (F)

William Harvey Research Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, Charterhouse Square, London, UK.

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Classifications MeSH