Synaptotagmin-11 inhibits spontaneous neurotransmission through vti1a.
Animals
Electrophysiological Phenomena
Excitatory Postsynaptic Potentials
Gene Knock-In Techniques
Hippocampus
/ pathology
Immunoprecipitation
Mice
Mice, Inbred C57BL
Mice, Knockout
Neurons
/ pathology
Primary Cell Culture
Qb-SNARE Proteins
/ drug effects
Synaptic Transmission
/ drug effects
Synaptotagmins
/ pharmacology
SNARE
spontaneous release
synaptotagmin
vti1a
Journal
Journal of neurochemistry
ISSN: 1471-4159
Titre abrégé: J Neurochem
Pays: England
ID NLM: 2985190R
Informations de publication
Date de publication:
11 2021
11 2021
Historique:
revised:
25
11
2020
received:
06
06
2019
accepted:
26
09
2021
pubmed:
3
10
2021
medline:
7
1
2022
entrez:
2
10
2021
Statut:
ppublish
Résumé
Recent work has revealed that spontaneous release plays critical roles in the central nervous system, but how it is regulated remains elusive. Here, we report that synaptotagmin-11 (Syt11), a Ca
Substances chimiques
Qb-SNARE Proteins
0
Syt11 protein, mouse
0
Vti1a protein, mouse
0
Synaptotagmins
134193-27-4
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
729-741Informations de copyright
© 2021 International Society for Neurochemistry.
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