Efficacy of umeclidinium/vilanterol according to the degree of reversibility of airflow limitation at screening: a post hoc analysis of the EMAX trial.


Journal

Respiratory research
ISSN: 1465-993X
Titre abrégé: Respir Res
Pays: England
ID NLM: 101090633

Informations de publication

Date de publication:
28 Oct 2021
Historique:
received: 13 05 2021
accepted: 08 10 2021
entrez: 29 10 2021
pubmed: 30 10 2021
medline: 8 2 2022
Statut: epublish

Résumé

In patients with chronic obstructive pulmonary disease (COPD), the relationship between short-term bronchodilator reversibility and longer-term response to bronchodilators is unclear. Here, we investigated whether the efficacy of long-acting bronchodilators is associated with reversibility of airflow limitation in patients with COPD with a low exacerbation risk not receiving inhaled corticosteroids. The double-blind, double-dummy EMAX trial randomised patients to umeclidinium/vilanterol 62.5/25 µg once daily, umeclidinium 62.5 µg once daily, or salmeterol 50 µg twice daily. Bronchodilator reversibility to salbutamol was measured once at screening and defined as an increase in forced expiratory volume in 1 s (FEV The mean (standard deviation) reversibility was 130 mL (156) and the median was 113 mL; 625/2425 (26%) patients were reversible. There was a trend towards greater improvements in trough FEV FP analyses suggest that patients with higher levels of reversibility have greater improvements in lung function and symptoms in response to bronchodilators. Improvements in lung function and rescue medication use were greater with umeclidinium/vilanterol versus monotherapy across the full range of reversibility, suggesting that the dual bronchodilator umeclidinium/vilanterol may be an appropriate treatment for patients with symptomatic COPD, regardless of their level of reversibility.

Sections du résumé

BACKGROUND BACKGROUND
In patients with chronic obstructive pulmonary disease (COPD), the relationship between short-term bronchodilator reversibility and longer-term response to bronchodilators is unclear. Here, we investigated whether the efficacy of long-acting bronchodilators is associated with reversibility of airflow limitation in patients with COPD with a low exacerbation risk not receiving inhaled corticosteroids.
METHODS METHODS
The double-blind, double-dummy EMAX trial randomised patients to umeclidinium/vilanterol 62.5/25 µg once daily, umeclidinium 62.5 µg once daily, or salmeterol 50 µg twice daily. Bronchodilator reversibility to salbutamol was measured once at screening and defined as an increase in forced expiratory volume in 1 s (FEV
RESULTS RESULTS
The mean (standard deviation) reversibility was 130 mL (156) and the median was 113 mL; 625/2425 (26%) patients were reversible. There was a trend towards greater improvements in trough FEV
CONCLUSIONS CONCLUSIONS
FP analyses suggest that patients with higher levels of reversibility have greater improvements in lung function and symptoms in response to bronchodilators. Improvements in lung function and rescue medication use were greater with umeclidinium/vilanterol versus monotherapy across the full range of reversibility, suggesting that the dual bronchodilator umeclidinium/vilanterol may be an appropriate treatment for patients with symptomatic COPD, regardless of their level of reversibility.

Identifiants

pubmed: 34711232
doi: 10.1186/s12931-021-01859-w
pii: 10.1186/s12931-021-01859-w
pmc: PMC8555352
doi:

Substances chimiques

Adrenergic beta-2 Receptor Agonists 0
Benzyl Alcohols 0
Bronchodilator Agents 0
Chlorobenzenes 0
Drug Combinations 0
GSK573719 0
Muscarinic Antagonists 0
Quinuclidines 0
vilanterol 028LZY775B

Types de publication

Journal Article Multicenter Study Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

279

Subventions

Organisme : GSK
ID : 201749 [NCT03034915]

Informations de copyright

© 2021. The Author(s).

Références

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Auteurs

Claus F Vogelmeier (CF)

Department of Medicine, Pulmonary and Critical Care Medicine, University Medical Centre Giessen and Marburg, Philipps-Universität Marburg, German Centre for Lung Research (DZL), Baldingerstraße, 35043, Marburg, Germany. claus.vogelmeier@med.uni-marburg.de.

Paul W Jones (PW)

GSK, Brentford, Middlesex, UK.

Edward M Kerwin (EM)

Altitude Clinical Consulting and Clinical Research Institute of Southern Oregon, Medford, OR, USA.

Isabelle H Boucot (IH)

GSK, Brentford, Middlesex, UK.

François Maltais (F)

Centre de Pneumologie, Institut Universitaire de Cardiologie et de Pneumologie de Québec, Université Laval, Québec, Québec, Canada.

Lee Tombs (L)

Precise Approach Ltd, GSK, Brentford, Middlesex, UK.

Chris Compton (C)

GSK, Brentford, Middlesex, UK.

David A Lipson (DA)

Respiratory Clinical Sciences, GSK, Collegeville, PA, USA.
Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.

Leif H Bjermer (LH)

Respiratory Medicine and Allergology, Lund University, Lund, Sweden.

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