Bacterial chromosomal mobility via lateral transduction exceeds that of classical mobile genetic elements.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
08 11 2021
Historique:
received: 01 04 2021
accepted: 03 09 2021
entrez: 9 11 2021
pubmed: 10 11 2021
medline: 28 12 2021
Statut: epublish

Résumé

It is commonly assumed that the horizontal transfer of most bacterial chromosomal genes is limited, in contrast to the frequent transfer observed for typical mobile genetic elements. However, this view has been recently challenged by the discovery of lateral transduction in Staphylococcus aureus, where temperate phages can drive the transfer of large chromosomal regions at extremely high frequencies. Here, we analyse previously published as well as new datasets to compare horizontal gene transfer rates mediated by different mechanisms in S. aureus and Salmonella enterica. We find that the horizontal transfer of core chromosomal genes via lateral transduction can be more efficient than the transfer of classical mobile genetic elements via conjugation or generalized transduction. These results raise questions about our definition of mobile genetic elements, and the potential roles played by lateral transduction in bacterial evolution.

Identifiants

pubmed: 34750368
doi: 10.1038/s41467-021-26004-5
pii: 10.1038/s41467-021-26004-5
pmc: PMC8575950
doi:

Substances chimiques

Bacterial Proteins 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

6509

Subventions

Organisme : Medical Research Council
ID : MR/M003876/1
Pays : United Kingdom
Organisme : Biotechnology and Biological Sciences Research Council
ID : BB/V002376/1
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 201531/Z/16/Z
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/S00940X/2
Pays : United Kingdom
Organisme : Biotechnology and Biological Sciences Research Council
ID : BB/N002873/1
Pays : United Kingdom
Organisme : Wellcome Trust
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/S00940X/1
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/V000772/1
Pays : United Kingdom

Commentaires et corrections

Type : CommentIn
Type : CommentIn

Informations de copyright

© 2021. The Author(s).

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Auteurs

Suzanne Humphrey (S)

Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, G12 8TA, UK.

Alfred Fillol-Salom (A)

Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, G12 8TA, UK.
MRC Centre for Molecular Bacteriology and Infection, Imperial College London, London, SW7 2AZ, UK.

Nuria Quiles-Puchalt (N)

Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, G12 8TA, UK.
MRC Centre for Molecular Bacteriology and Infection, Imperial College London, London, SW7 2AZ, UK.

Rodrigo Ibarra-Chávez (R)

Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, G12 8TA, UK.
Department of Biology, Section of Microbiology, University of Copenhagen, Universitetsparken 15, Bldg. 1, DK2100, Copenhagen, Denmark.

Andreas F Haag (AF)

Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, G12 8TA, UK.

John Chen (J)

Department of Microbiology and Immunology, Yong Loo Lin School of Medicine, National University of Singapore, 5 Science Drive 2, Singapore, Singapore.

José R Penadés (JR)

Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, G12 8TA, UK. j.penades@imperial.ac.uk.
MRC Centre for Molecular Bacteriology and Infection, Imperial College London, London, SW7 2AZ, UK. j.penades@imperial.ac.uk.
Departamento de Ciencias Biomédicas, Facultad de Ciencias de la Salud, Universidad CEU Cardenal Herrera, Valencia, 46113, Spain. j.penades@imperial.ac.uk.

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