The 30-year Natural History of Non-classic Fabry Disease with an R112H Mutation.


Journal

Internal medicine (Tokyo, Japan)
ISSN: 1349-7235
Titre abrégé: Intern Med
Pays: Japan
ID NLM: 9204241

Informations de publication

Date de publication:
01 Jun 2022
Historique:
pubmed: 23 11 2021
medline: 7 6 2022
entrez: 22 11 2021
Statut: ppublish

Résumé

Fabry disease is a rare X-linked lysosomal storage disorder caused by mutations in the alpha-galactosidase A (GLA) gene that results in deficiency of the enzyme GLA and leads to the accumulation of globotriaosylceramide (GL-3) in cells. The accumulation of GL-3 may lead to life-threatening complications. Significant advances in genetic sequencing technology have led to a better understanding of genotype-phenotype interactions in Fabry disease. Fabry disease with an R112H mutation is known as the non-classic type. However, the long-term clinical course of the disease remains unknown. We herein report a patient with a 30-year natural history of non-classic Fabry disease with an R112H mutation.

Identifiants

pubmed: 34803097
doi: 10.2169/internalmedicine.8213-21
pmc: PMC9259301
doi:

Substances chimiques

alpha-Galactosidase EC 3.2.1.22

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1727-1730

Références

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Auteurs

Reiko Muto (R)

Department of Nephrology, Nagoya University Graduate School of Medicine, Japan.

Koji Inagaki (K)

Department of Nephrology, Chutoen General Medical Center, Japan.

Noritoshi Kato (N)

Department of Nephrology, Nagoya University Graduate School of Medicine, Japan.

Shoichi Maruyama (S)

Department of Nephrology, Nagoya University Graduate School of Medicine, Japan.

Toshiyuki Akahori (T)

Department of Nephrology, Chutoen General Medical Center, Japan.

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Classifications MeSH