Influences on the trajectory and subsequent outcomes in CDKL5 deficiency disorder.

CDKL5 deficiency disorder developmental and epileptic encephalopathy developmental trajectory quality of life seizure burden

Journal

Epilepsia
ISSN: 1528-1167
Titre abrégé: Epilepsia
Pays: United States
ID NLM: 2983306R

Informations de publication

Date de publication:
02 2022
Historique:
revised: 28 10 2021
received: 06 04 2021
accepted: 02 11 2021
pubmed: 28 11 2021
medline: 21 4 2022
entrez: 27 11 2021
Statut: ppublish

Résumé

The study investigated the effect of seizure and medication burden at initial contact with the International CDKL5 Disorder Database on subsequent development and clinical severity and compared quality of life among those whose development progressed, remained stable, or regressed between baseline and follow-up. The effects of seizure and medication burden at baseline (high or low) on the CDKL5 Disorder Severity Scores and CDKL5 Developmental Score (CDS) at follow-up were assessed using linear and negative binomial regressions, respectively, with adjustment for age at baseline, gender, and follow-up duration with and without genotype. Seizure and medication burden were defined by average daily seizure count (high, ≥5/day; low, <5/day) and number of antiseizure medications (high, ≥3/day; low, <3/day), respectively. The effects of change in CDS over time (improved, stable, or deteriorated) on Quality of Life Inventory-Disability (QI-Disability) total and domain scores at follow-up were assessed in those aged at least 3 years at follow-up using linear regression models with adjustment for baseline CDS, gender, and follow-up duration. The expected follow-up CDS was lower for individuals with high compared to low seizure burden at baseline (β = -.49, 95% confidence interval [CI] = -.84 to -.13). The average total QI-Disability score was 5.6 (95% CI = -.2 to 11.5) points higher among those with improved compared with stable or deteriorating CDS and 8.5 (95% CI = 3.1-13.8) points lower for those with deteriorating compared to stable or improved CDS. Our finding that later development showed slight improvement in those with better earlier seizure control even after adjustment for genotype suggests that the trajectory for an individual child is not necessarily predetermined and could possibly be influenced by optimal seizure management. This has implications for children's quality of life.

Identifiants

pubmed: 34837650
doi: 10.1111/epi.17125
doi:

Substances chimiques

Protein Serine-Threonine Kinases EC 2.7.11.1
CDKL5 protein, human EC 2.7.11.22

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

352-363

Subventions

Organisme : NINDS NIH HHS
ID : U01 NS114312
Pays : United States

Informations de copyright

© 2021 International League Against Epilepsy.

Références

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Auteurs

Helen Leonard (H)

Telethon Kids Institute, University of Western Australia, Nedlands, Western Australia, Australia.
Faculty of Health and Medical Sciences, Centre of Child Health Research, University of Western Australia, Nedlands, Western Australia, Australia.

Mohammed Junaid (M)

Telethon Kids Institute, University of Western Australia, Nedlands, Western Australia, Australia.

Kingsley Wong (K)

Telethon Kids Institute, University of Western Australia, Nedlands, Western Australia, Australia.

Alex A Aimetti (AA)

Marinus Pharmaceuticals, Radnor, Pennsylvania, USA.

Elia Pestana Knight (E)

Pediatric Epilepsy Section, Epilepsy Center, Cleveland Clinic, Neurological Institute, Cleveland, Ohio, USA.

Jenny Downs (J)

Telethon Kids Institute, University of Western Australia, Nedlands, Western Australia, Australia.

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