Oncogenic dependency on STAT3 serine phosphorylation in KRAS mutant lung cancer.


Journal

Oncogene
ISSN: 1476-5594
Titre abrégé: Oncogene
Pays: England
ID NLM: 8711562

Informations de publication

Date de publication:
02 2022
Historique:
received: 30 10 2020
accepted: 23 11 2021
revised: 15 11 2021
pubmed: 4 12 2021
medline: 24 2 2022
entrez: 3 12 2021
Statut: ppublish

Résumé

The oncogenic potential of the latent transcription factor signal transducer and activator of transcription (STAT)3 in many human cancers, including lung cancer, has been largely attributed to its nuclear activity as a tyrosine-phosphorylated (pY

Identifiants

pubmed: 34857889
doi: 10.1038/s41388-021-02134-4
pii: 10.1038/s41388-021-02134-4
doi:

Substances chimiques

Serine 452VLY9402

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

809-823

Informations de copyright

© 2021. The Author(s), under exclusive licence to Springer Nature Limited.

Références

Wong MCS, Lao XQ, Ho KF, Goggins WB, Tse SLA. Incidence and mortality of lung cancer: global trends and association with socioeconomic status. Sci Rep. 2017;7:14300–9.
pubmed: 29085026 pmcid: 5662733 doi: 10.1038/s41598-017-14513-7
Hirsch FR, Scagliotti GV, Mulshine JL, Kwon R, Curran WJ Jr, Wu YL, et al. Lung cancer: current therapies and new targeted treatments. Lancet. 2017;389:299–311.
pubmed: 27574741 doi: 10.1016/S0140-6736(16)30958-8
Wang X, Goldstein D, Crowe PJ, Yang JL. Next-generation EGFR/HER tyrosine kinase inhibitors for the treatment of patients with non-small-cell lung cancer harboring EGFR mutations: a review of the evidence. Onco Targets Ther. 2016;9:5461–73.
pubmed: 27660463 pmcid: 5021053 doi: 10.2147/OTT.S94745
Noonan KL, Ho C, Laskin J, Murray N. The influence of the evolution of first-line chemotherapy on steadily improving survival in advanced non-small-cell lung cancer clinical trials. J Thorac Oncol. 2015;10:1523–31.
pubmed: 26536194 doi: 10.1097/JTO.0000000000000667
Walser T, Cui X, Yanagawa J, Lee JM, Heinrich E, Lee G, et al. Smoking and lung cancer: the role of inflammation. Proc Am Thorac Soc. 2008;5:811–5.
pubmed: 19017734 pmcid: 4080902 doi: 10.1513/pats.200809-100TH
Ahrendt S, Decker PA, Alawi EA, Zhu YR, Sanchez-Cespedes M, Yang SC, et al. Cigarette smoking is strongly associated with mutation of the K-ras gene in patients with primary adenocarcinoma of the lung. Cancer. 2001;92:1525–30.
pubmed: 11745231 doi: 10.1002/1097-0142(20010915)92:6<1525::AID-CNCR1478>3.0.CO;2-H
Westra WH, Slebos RJ, Offerhaus GJ, Goodman SN, Evers SG, Kensler TW, et al. K-ras oncogene activation in lung adenocarcinomas from former smokers: evidence that K-ras mutations are an early and irreversible event in the development of adenocarcinoma of the lung. Cancer. 1993;72:432–8.
pubmed: 8319174 doi: 10.1002/1097-0142(19930715)72:2<432::AID-CNCR2820720219>3.0.CO;2-#
Román M, Baraibar I, López I, Nadal E, Rolfo C, Vicent S, et al. KRAS oncogene in non-small cell lung cancer: clinical perspectives on the treatment of an old target. Mol Cancer. 2018;17:33.
pubmed: 29455666 pmcid: 5817724 doi: 10.1186/s12943-018-0789-x
Jackson E, Willis N, Mercer K, Bronson RT, Crowley D, Montoya R, et al. DA. Analysis of lung tumor initiation and progression using conditional expression of oncogenic K-ras. Genes Dev. 2001;15:3243–8.
pubmed: 11751630 pmcid: 312845 doi: 10.1101/gad.943001
DuPage M, Dooley AL, Jacks T. Conditional mouse lung cancer models using adenoviral or lentiviral delivery of Cre recombinase. Nat Protoc. 2009;4:1064–72.
pubmed: 19561589 pmcid: 2757265 doi: 10.1038/nprot.2009.95
Cox AD, Fesik SW, Kimmelman AC, Luo J, Der CJ. Drugging the undruggable RAS: Mission possible? Nat Rev Drug Discov. 2014;13:828–51.
pubmed: 25323927 pmcid: 4355017 doi: 10.1038/nrd4389
Janes MR, Zhang J, Li LS, Hansen R, Peters U, Guo X, et al. Targeting KRAS mutant cancers with a covalent G12C-specific inhibitor. Cell. 2018;172:578–89.
pubmed: 29373830 doi: 10.1016/j.cell.2018.01.006
Zhu Z, Aref A, Cohoon TJ, Barbie TU, Imamura Y, Yang S, et al. Inhibition of KRAS-driven tumorigenicity by interruption of an autocrine cytokine circuit. Cancer Discov. 2014;4:452–65.
pubmed: 24444711 pmcid: 3980023 doi: 10.1158/2159-8290.CD-13-0646
Brooks GD, McLeod L, Alhayyani S, Miller A, Russell PA, Ferlin W, et al. IL6 Trans-signaling promotes KRAS-driven lung carcinogenesis. Cancer Res. 2016;76:866–76.
pubmed: 26744530 doi: 10.1158/0008-5472.CAN-15-2388
Saad MI, Alhayyani S, McLeod L, Yu L, Alanazi M, Deswaerte V, et al. ADAM17 selectively activates the IL-6 trans-signaling/ERK MAPK axis in KRAS-addicted lung cancer. EMBO Mol Med. 2019;11:e9976.
pubmed: 30833304 pmcid: 6460353 doi: 10.15252/emmm.201809976
Kim HS, Park YH, Lee J, Ahn JS, Kim J, Shim YM, et al. Clinical impact of phosphorylated signal transducer and activator of transcription 3, epidermal growth factor receptor, p53, and vascular endothelial growth factor receptor 1 expression in resected adenocarcinoma of lung by using tissue microarray. Cancer. 2010;116:676–85.
pubmed: 20052735 doi: 10.1002/cncr.24748
Tong M, Wang J, Jiang N, Pan H, Li D. Correlation between p-STAT3 overexpression and prognosis in lung cancer: a systematic review and meta-analysis. PLoS One. 2017;12:e0182282.
pubmed: 28797050 pmcid: 5552221 doi: 10.1371/journal.pone.0182282
Yang Q, Shen S, Zhou S, Ni J, Chen D, Wang G, et al. STAT3 activation and aberrant ligand-dependent sonic hedgehog signaling in human pulmonary adenocarcinoma. Exp Mol Pathol. 2012;93:227–36.
pubmed: 22554932 doi: 10.1016/j.yexmp.2012.04.009
Tran P, Fan AC, Bendapudi PK, Koh S, Komatsubara K, Chen J, et al. Combined Inactivation of MYC and K-Ras oncogenes reverses tumorigenesis in lung adenocarcinomas and lymphomas. PLoS One. 2008;3:e2125.
pubmed: 18461184 pmcid: 2365560 doi: 10.1371/journal.pone.0002125
Li Y, Du H, Qin Y, Roberts J, Cummings OW, Yan C. Activation of the signal transducers and activators of the transcription 3 pathway in alveolar epithelial cells induces inflammation and adenocarcinomas in mouse lung. Cancer Res. 2007;67:8494–503.
pubmed: 17875688 doi: 10.1158/0008-5472.CAN-07-0647
Yu H, Lee H, Herrmann A, Buettner R, Jove R. Revisiting STAT3 signalling in cancer: new and unexpected biological functions. Nat Rev Cancer. 2014;14:736–46.
pubmed: 25342631 doi: 10.1038/nrc3818
Poli V, Camporeale A. STAT3-mediated metabolic reprograming in cellular transformation and implications for drug resistance. Front Oncol. 2015;5:121.
pubmed: 26106584 pmcid: 4459099 doi: 10.3389/fonc.2015.00121
Jones SA, Jenkins BJ. Recent insights into targeting the IL-6 cytokine family in inflammatory diseases and cancer. Nat Rev Immunol. 2018;18:773–89.
pubmed: 30254251 doi: 10.1038/s41577-018-0066-7
Gough D, Corlett A, Schlessinger K, Wegrzyn J, Larner AC, Levy DE. Mitochondrial STAT3 supports Ras-dependent oncogenic transformation. Science. 2009;324:1713–6.
pubmed: 19556508 pmcid: 2840701 doi: 10.1126/science.1171721
Wegrzyn J, Potla R, Chwae YJ, Sepuri NB, Zhang Q, Koeck T, et al. Function of mitochondrial Stat3 in cellular respiration. Science. 2009;323:793–7.
pubmed: 19131594 pmcid: 2758306 doi: 10.1126/science.1164551
Gough DJ, Marie I, Lobry C, Aifantis I, Levy DE. STAT3 supports experimental K-RasG12D–induced murine myeloproliferative neoplasms dependent on serine phosphorylation. Blood. 2014;124:2252–61.
pubmed: 25150294 pmcid: 4183984 doi: 10.1182/blood-2013-02-484196
Garama DJ, White CL, Balic JJ, Gough DJ. Mitochondrial STAT3: powering up a potent factor. Cytokine. 2016;87:20–25.
pubmed: 27269970 doi: 10.1016/j.cyto.2016.05.019
Balic JJ, Garama DJ, Saad M, Yu L, West A, West AJ, et al. Serine-phosphorylated STAT3 promotes tumorigenesis via modulation of RNA polymerase transcriptional activity. Cancer Res. 2019;79:5272–87.
pubmed: 31481496 doi: 10.1158/0008-5472.CAN-19-0974
Shen Y, Schlessinger K, Zhu X, Meffre E, Quimby F, Levy DE, et al. Essential role of STAT3 in postnatal survival and growth revealed by mice lacking STAT3 serine 727 phosphorylation. Mol Cell Biol. 2004;24:407–19.
pubmed: 14673173 pmcid: 303338 doi: 10.1128/MCB.24.1.407-419.2004
Wen Z, Zhong Z, Darnell JE Jr. Maximal activation of transcription by Stat1 and Stat3 requires both tyrosine and serine phosphorylation. Cell. 1995;82:241–50.
pubmed: 7543024 doi: 10.1016/0092-8674(95)90311-9
Zouein FA, Zgheib C, Hamza S, Fuseler JW, Hall JE, Soljancic A, et al. Role of STAT3 in angiotensin II-induced hypertension and cardiac remodeling revealed by mice lacking STAT3 serine 727 phosphorylation. Hypertens Res. 2013;36:496–503.
pubmed: 23364341 pmcid: 3674130 doi: 10.1038/hr.2012.223
Johnson L, Mercer K, Greenbaum D, Bronson RT, Crowley D, Tuveson DA, et al. Somatic activation of the K-ras oncogene causes early onset lung cancer in mice. Nature. 2001;410:1111–6.
pubmed: 11323676 doi: 10.1038/35074129
Jerome-Marson V, Mazieres J, Groussard O, Garcia O, Berjaud J, Dahan M, et al. Expression of TTF-1 and cytokeratins in primary and secondary epithelial lung tumours: correlation with histological type and grade. Histopathology. 2004;45:125–34.
pubmed: 15279630 doi: 10.1111/j.1365-2559.2004.01893.x
Kortlever RM, Sodir N, Wilson CH, Burkhart DL, Pellegrinet L, Brown Swigart L, et al. Myc cooperates with Ras by programming inflammation and immune suppression. Cell. 2017;171:1301–15.
pubmed: 29195074 pmcid: 5720393 doi: 10.1016/j.cell.2017.11.013
Dragoj M, Milosevic Z, Bankovic J, Dinic J, Pesic M, Tanic N, et al. Association of CCND1 overexpression with KRAS and PTEN alterations in specific subtypes of non-small cell lung carcinoma and its influence on patients’ outcome. Tumour Biol. 2015;36:8773–80.
pubmed: 26055143 doi: 10.1007/s13277-015-3620-y
Soucek L, Whitfield J, Martins CP, Finch AJ, Murphy DJ, Sodir NM, et al. Modelling Myc inhibition as a cancer therapy. Nature. 2008;455:679–83.
pubmed: 18716624 pmcid: 4485609 doi: 10.1038/nature07260
Murphy DJ, Junttila MR, Pouyet L, Karnezis A, Shchors K, Bui DA, et al. Distinct thresholds govern Myc’s biological output in vivo. Cancer Cell. 2008;14:447–57.
pubmed: 19061836 doi: 10.1016/j.ccr.2008.10.018
Dutta P, Zhang L, Zhang H, Peng Q, Montgrain PR, Wang Y, et al. Unphosphorylated STAT3 in heterochromatin formation and tumor suppression in lung cancer. BMC Cancer. 2020;20:145.
pubmed: 32087696 pmcid: 7036253 doi: 10.1186/s12885-020-6649-2
Ji H, Houghton A, Mariani TJ, Perera S, Kim CB, Padera R, et al. K-ras activation generates an inflammatory response in lung tumors. Oncogene. 2006;25:2105–12.
pubmed: 16288213 doi: 10.1038/sj.onc.1209237
McClelland MR, Carskadon SL, Zhao L, White ES, Beer DG, Orringer MB, et al. Diversity of the angiogenic phenotype in non–small cell lung cancer. Am J Respir Cell Mol Biol. 2007;36:343–50.
pubmed: 17079777 doi: 10.1165/rcmb.2006-0311OC
Liou GY, Doppler H, DelGiorno KE, Zhang L, Leitges M, Crawford HC, et al. Mutant KRas-induced mitochondrial oxidative stress in acinar cells upregulates EGFR signaling to drive formation of pancreatic precancerous lesions. Cell Rep. 2016;14:2325–36.
pubmed: 26947075 pmcid: 4794374 doi: 10.1016/j.celrep.2016.02.029
Weinberg F, Hamanaka R, Wheaton WW, Weinberg S, Joseph J, Lopez M, et al. Mitochondrial metabolism and ROS generation are essential for Kras-mediated tumorigenicity. Proc Natl Acad Sci USA. 2010;107:8788–93.
pubmed: 20421486 pmcid: 2889315 doi: 10.1073/pnas.1003428107
Vander Heiden MG, Cantley LC, Thompson CB. Understanding the Warburg effect: the metabolic requirements of cell proliferation. Science. 2009;324:1029–33.
doi: 10.1126/science.1160809
Stine ZE, Walton Z, Altman BJ, Hsieh AL, Dang CV. MYC, metabolism, and cancer. Cancer Discov. 2015;5:1024–39.
pubmed: 26382145 pmcid: 4592441 doi: 10.1158/2159-8290.CD-15-0507
Bowman T, Broome MA, Sinibaldi D, Wharton W, Pledger WJ, Sedivy JM, et al. Stat3-mediated Myc expression is required for Src transformation and PDGF-induced mitogenesis. Proc Natl Acad Sci USA. 2001;98:7319–24.
pubmed: 11404481 pmcid: 34666 doi: 10.1073/pnas.131568898
Yu L, Wu D, Gao H, Balic J, Tsykin A, Han T-S, et al. Clinical utility of a STAT3-regulated microRNA-200 family signature with prognostic potential in early gastric cancer. Clin Cancer Res. 2018;24:1459–72.
pubmed: 29330205 doi: 10.1158/1078-0432.CCR-17-2485
Niu G, Briggs J, Deng J, Ma Y, Lee H, Kortylewski M, et al. Signal transducer and activator of transcription 3 is required for hypoxia-inducible factor-1alpha RNA expression in both tumor cells and tumor-associated myeloid cells. Mol Cancer Res. 2008;6:1099–105.
pubmed: 18644974 pmcid: 2775817 doi: 10.1158/1541-7786.MCR-07-2177
Nagao A, Kobayashi M, Koyasu S, Chow CCT, Harada H. HIF-1-dependent reprogramming of glucose metabolic pathway of cancer cells and its therapeutic significance. Int J Mol Sci. 2019;20:E238.
pubmed: 30634433 doi: 10.3390/ijms20020238
Hirahara K, Onodera A, Villarino AV, Bonelli M, Sciumè G, Laurence A, et al. Asymmetric action of STAT transcription factors drives transcriptional outputs and cytokine specificity. Immunity. 2015;42:877–89.
pubmed: 25992861 doi: 10.1016/j.immuni.2015.04.014
Costa-Pereira AP, Tininini S, Strobl B, Alonzi T, Schlaak JF, Is’harc H, et al. Mutational switch of an IL-6 response to an interferon-gamma-like response. Proc Natl Acad Sci USA. 2002;99:8043–7.
pubmed: 12060750 pmcid: 123017 doi: 10.1073/pnas.122236099
Yu C, Huo X, Agoston AT, Zhang X, Theiss AL, Cheng E, et al. Mitochondrial STAT3 contributes to transformation of Barrett’s epithelial cells that express oncogenic Ras in a p53-independent fashion. Am J Physiol Gastrointest Liver Physiol. 2015;309:G146–161.
pubmed: 26045618 pmcid: 4525109 doi: 10.1152/ajpgi.00462.2014
Song L, Turkson J, Karras JG, Jove R, Haura EB. Activation of Stat3 by receptor tyrosine kinases and cytokines regulates survival in human non-small cell carcinoma cells. Oncogene. 2003;22:4150–65.
pubmed: 12833138 doi: 10.1038/sj.onc.1206479
Zhou J, Qu Z, Yan S, Sun F, Whitsett JA, Shapiro SD, et al. Differential roles of STAT3 in the initiation and growth of lung cancer. Oncogene. 2015;34:3804–14.
pubmed: 25284582 doi: 10.1038/onc.2014.318
Grabner B, Schramek D, Mueller KM, Moll HP, Svinka J, Hoffmann T, et al. Disruption of STAT3 signalling promotes KRAS-induced lung tumorigenesis. Nat Commun. 2015;6:6285–98.
pubmed: 25734337 doi: 10.1038/ncomms7285
Caetano MS, Hassane M, Van HT, Bugarin E, Cumpian AM, McDowell CL, et al. Sex specific function of epithelial STAT3 signaling in pathogenesis of K-ras mutant lung cancer. Nat Commun. 2018;9:4589.
pubmed: 30389925 pmcid: 6214980 doi: 10.1038/s41467-018-07042-y
Meissl K, Macho-Maschler S, Müller M, Strobl B. The good and the bad faces of STAT1 in solid tumours. Cytokine. 2017;89:12–20.
pubmed: 26631912 doi: 10.1016/j.cyto.2015.11.011
Takeda K, Clausen BE, Kaisho T, Tsujimura T, Terada N, Förster I, et al. Enhanced Th1 activity and development of chronic enterocolitis in mice devoid of Stat3 in macrophages and neutrophils. Immunity. 1999;10:39–49.
pubmed: 10023769 doi: 10.1016/S1074-7613(00)80005-9
Sano S, Itami S, Takeda K, Tarutani M, Yamaguchi Y, Miura H, et al. Keratinocyte-specific ablation of Stat3 exhibits impaired skin remodeling, but does not affect skin morphogenesis. EMBO J. 1999;4657–68.
Zhu S, Phatarpekar P, Denman CJ, Senyukov VV, Somanchi SS, Nguyen-Jackson HT, et al. Transcription of the activating receptor NKG2D in natural killer cells is regulated by STAT3 tyrosine phosphorylation. Blood. 2014;124:403–11.
pubmed: 24891320 pmcid: 4102712 doi: 10.1182/blood-2013-05-499707
D’Amico S, Shi J, Martin BL, Crawford HC, Petrenko O, Reich NC. STAT3 is a master regulator of epithelial identity and KRAS-driven tumorigenesis. Genes Dev. 2018;32:1175–87.
pubmed: 30135074 pmcid: 6120712 doi: 10.1101/gad.311852.118
Oft M. IL-10: master switch from tumor-promoting inflammation to antitumor immunity. Cancer Immunol Res. 2014;2:194–9.
pubmed: 24778315 doi: 10.1158/2326-6066.CIR-13-0214
Li F, Wang Y, Zeller KI, Potter JJ, Wonsey DR, O’Donnell KA, et al. Myc stimulates nuclearly encoded mitochondrial genes and mitochondrial biogenesis. Mol Cell Biol. 2005;25:6225–34.
pubmed: 15988031 pmcid: 1168798 doi: 10.1128/MCB.25.14.6225-6234.2005
Han YH, Sim HK, Kim SZ, Park WH. Antimycin A as a mitochondrial electron transport inhibitor prevents the growth of human lung cancer A549 cells. Oncol Rep. 2008;20:689–93.
pubmed: 18695925
Bretones G, Delgado M, León J. Myc and cell cycle control. Biochim Biophys Acta. 2015;1849:506–16.
pubmed: 24704206 doi: 10.1016/j.bbagrm.2014.03.013
Macias E, Rao D, Carbajal S, Kiguchi K, DiGiovanni J. Stat3 binds to mtDNA and regulates mitochondrial gene expression in keratinocytes. J Invest Dermatol. 2014;134:1971–80.
pubmed: 24496235 pmcid: 4057971 doi: 10.1038/jid.2014.68
Mao YQ, Houry WA. The role of pontin and reptin in cellular physiology and cancer etiology. Front Mol Biosci. 2017;4:58.
pubmed: 28884116 pmcid: 5573869 doi: 10.3389/fmolb.2017.00058
Weng MT, Luo J. The enigmatic ERH protein: its role in cell cycle, RNA splicing and cancer. Protein Cell. 2013;4:807–12.
pubmed: 24078386 pmcid: 4875447 doi: 10.1007/s13238-013-3056-3
Kilkenny C, Browne W, Cuthill IC, Emerson M, Altman DG. Improving bioscience research reporting: the ARRIVE guidelines for reporting animal research. PLoS Biol. 2010;8:e1000412.
pubmed: 20613859 pmcid: 2893951 doi: 10.1371/journal.pbio.1000412
Garassino MC, Marabese M, Rusconi P, Rulli E, Martelli O, Farina G, et al. Different types of K-Ras mutations could affect drug sensitivity and tumour behaviour in non-small-cell lung cancer. Ann Oncol. 2011;22:235–7.
pubmed: 21169473 doi: 10.1093/annonc/mdq680
Ruwanpura SM, McLeod L, Miller A, Jones J, Bozinovski S, Vlahos R, et al. Interleukin-6 promotes pulmonary emphysema associated with apoptosis in mice. Am J Respir Cell Mol Biol. 2011;45:720–30.
pubmed: 21297079 doi: 10.1165/rcmb.2010-0462OC
Liu YD, Yu L, Ying L, Balic J, Gao H, Deng NT, et al. Toll-like receptor 2 regulates metabolic reprogramming in gastric cancer via superoxide dismutase 2. Int J Cancer. 2019;144:3056–69.
pubmed: 30536754 pmcid: 6590666 doi: 10.1002/ijc.32060

Auteurs

Sultan Alhayyani (S)

Centre for Innate Immunity and Infectious Diseases, Hudson Institute of Medical Research, Clayton, Victoria, 3168, Australia.
Department of Molecular and Translational Science, School of Clinical Sciences, Monash University, Clayton, Victoria, 3168, Australia.
Department of Chemistry, College of Sciences and Arts, King Abdulaziz University, Rabigh, Saudi Arabia.

Louise McLeod (L)

Centre for Innate Immunity and Infectious Diseases, Hudson Institute of Medical Research, Clayton, Victoria, 3168, Australia.
Department of Molecular and Translational Science, School of Clinical Sciences, Monash University, Clayton, Victoria, 3168, Australia.

Alison C West (AC)

Centre for Innate Immunity and Infectious Diseases, Hudson Institute of Medical Research, Clayton, Victoria, 3168, Australia.
Department of Molecular and Translational Science, School of Clinical Sciences, Monash University, Clayton, Victoria, 3168, Australia.

Jesse J Balic (JJ)

Centre for Innate Immunity and Infectious Diseases, Hudson Institute of Medical Research, Clayton, Victoria, 3168, Australia.
Department of Molecular and Translational Science, School of Clinical Sciences, Monash University, Clayton, Victoria, 3168, Australia.

Christopher Hodges (C)

Centre for Innate Immunity and Infectious Diseases, Hudson Institute of Medical Research, Clayton, Victoria, 3168, Australia.
Department of Molecular and Translational Science, School of Clinical Sciences, Monash University, Clayton, Victoria, 3168, Australia.

Liang Yu (L)

Centre for Innate Immunity and Infectious Diseases, Hudson Institute of Medical Research, Clayton, Victoria, 3168, Australia.
Department of Molecular and Translational Science, School of Clinical Sciences, Monash University, Clayton, Victoria, 3168, Australia.

Julian A Smith (JA)

Department of Surgery, School of Clinical Sciences at Monash Health, Monash University, Clayton, Victoria, 3168, Australia.
Department of Cardiothoracic Surgery, Monash Health, Clayton, Victoria, 3168, Australia.

Zdenka Prodanovic (Z)

Monash Biobank, Monash Health, Clayton, Victoria, 3168, Australia.

Steven Bozinovski (S)

School of Health and Biomedical Sciences, RMIT University, Bundoora, Victoria, 3082, Australia.

Beena Kumar (B)

Department of Anatomical Pathology, Monash Health, Clayton, Victoria, 3168, Australia.

Saleela M Ruwanpura (SM)

Centre for Innate Immunity and Infectious Diseases, Hudson Institute of Medical Research, Clayton, Victoria, 3168, Australia.
Department of Molecular and Translational Science, School of Clinical Sciences, Monash University, Clayton, Victoria, 3168, Australia.

Mohamed I Saad (MI)

Centre for Innate Immunity and Infectious Diseases, Hudson Institute of Medical Research, Clayton, Victoria, 3168, Australia.
Department of Molecular and Translational Science, School of Clinical Sciences, Monash University, Clayton, Victoria, 3168, Australia.

Brendan J Jenkins (BJ)

Centre for Innate Immunity and Infectious Diseases, Hudson Institute of Medical Research, Clayton, Victoria, 3168, Australia. brendan.jenkins@hudson.org.au.
Department of Molecular and Translational Science, School of Clinical Sciences, Monash University, Clayton, Victoria, 3168, Australia. brendan.jenkins@hudson.org.au.

Articles similaires

Aminoacid functionalised magnetite nanoparticles Fe

Spoială Angela, Motelica Ludmila, Ilie Cornelia-Ioana et al.
1.00
Magnetite Nanoparticles Tryptophan Biocompatible Materials Microbial Sensitivity Tests Humans
Animals Guinea Pigs Humans KCNQ1 Potassium Channel Endocannabinoids
Arabidopsis Arabidopsis Proteins Serine Ubiquitin-Specific Proteases Phylogeny
Humans Cell Proliferation Phosphorylation HSP90 Heat-Shock Proteins Molecular Chaperones

Classifications MeSH