Association of cabozantinib pharmacokinetics, progression and toxicity in metastatic renal cell carcinoma patients: results from a pharmacokinetics/pharmacodynamics study.


Journal

ESMO open
ISSN: 2059-7029
Titre abrégé: ESMO Open
Pays: England
ID NLM: 101690685

Informations de publication

Date de publication:
12 2021
Historique:
received: 20 07 2021
revised: 13 10 2021
accepted: 25 10 2021
pubmed: 6 12 2021
medline: 25 3 2022
entrez: 5 12 2021
Statut: ppublish

Résumé

Cabozantinib is a tyrosine kinase inhibitor with a substantial efficacy in metastatic renal cell carcinoma, and is associated with a challenging toxicity profile leading to frequent drug discontinuations. Whereas an exposure/safety relationship was demonstrated for this drug, an exposure/efficacy relationship is still unknown. We carried out a monocentric, observational, pharmacokinetics/pharmacodynamics (PK/PD) study in patients with metastatic renal cell carcinoma (INDS MR 5612140520). We used measured blood concentrations of cabozantinib (C We enrolled 76 patients, including 35 who experienced disease progression and 30 with grade 3-4 toxicity. Patients with progressive disease had a significantly lower median C We first demonstrate the PK/PD relationship for cabozantinib. Severe toxicities are associated with a higher drug exposure, whereas inefficacy is associated with a lower drug exposure. Cabozantinib plasma drug monitoring may be useful to optimize clinical practice.

Sections du résumé

BACKGROUND
Cabozantinib is a tyrosine kinase inhibitor with a substantial efficacy in metastatic renal cell carcinoma, and is associated with a challenging toxicity profile leading to frequent drug discontinuations. Whereas an exposure/safety relationship was demonstrated for this drug, an exposure/efficacy relationship is still unknown.
PATIENTS AND METHODS
We carried out a monocentric, observational, pharmacokinetics/pharmacodynamics (PK/PD) study in patients with metastatic renal cell carcinoma (INDS MR 5612140520). We used measured blood concentrations of cabozantinib (C
RESULTS
We enrolled 76 patients, including 35 who experienced disease progression and 30 with grade 3-4 toxicity. Patients with progressive disease had a significantly lower median C
CONCLUSION
We first demonstrate the PK/PD relationship for cabozantinib. Severe toxicities are associated with a higher drug exposure, whereas inefficacy is associated with a lower drug exposure. Cabozantinib plasma drug monitoring may be useful to optimize clinical practice.

Identifiants

pubmed: 34864351
pii: S2059-7029(21)00274-X
doi: 10.1016/j.esmoop.2021.100312
pmc: PMC8645912
pii:
doi:

Substances chimiques

Anilides 0
Pyridines 0
cabozantinib 1C39JW444G

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

100312

Informations de copyright

Copyright © 2021 The Authors. Published by Elsevier Ltd.. All rights reserved.

Déclaration de conflit d'intérêts

Disclosures EA reports personal financial interest from Genzyme and Mundipharma, outside the submitted work. EC reports personal financial interest from Bristol Myers Squibb (BMS), Brazil Clovis Oncology, GlaxoSmithKline, Ipsen, Merck, Pfizer outside the submitted work. OM reports personal financial interest from Bayer, Blueprint Medicines, BMS, Eli Lilly, Ipsen, Merck Sharp & Dohme (MSD), Pfizer, Roche, Servier and institutional financial interest from Bayer, Blueprint Medicines, Eli Lilly and Epizyme outside the submitted work. BE reports personal financial interest from AVEO, BMS, EUSA Pharma, Ipsen, MSD, Novartis, Oncorena, Pfizer; Roche/Genentech and institutional consulting fees compensated to their institution from BMS France, all outside the submitted work. LA reports consulting fees compensated to their institution from Amgen, Astellas, AstraZeneca, BMS, Corvus Pharmaceuticals, Exelixis, Ipsen, Merck KGaA, Merck & Co., Novartis, Peloton Therapeutics, Roche and Pfizer outside the submitted work. All other authors have declared no conflicts of interest.

Auteurs

L Cerbone (L)

Department of Oncological Medicine, Gustave Roussy, Villejuif, France.

D Combarel (D)

Department of Medical Biology and Pathology, Gustave Roussy, Villejuif, France; Medical School, University of Paris XI Sacly, Saclay, France.

A Geraud (A)

Medical School, University of Paris XI Sacly, Saclay, France; Department of Therapeutic Innovations and Early Trials, Gustave Roussy, Villejuif, France.

E Auclin (E)

Medical Oncology Hopital Europeen Georges Pompidou, Paris, France.

S Foulon (S)

Department of Biostatistics and Epidemiology, Gustave Roussy, Paris, France.

C Alves Costa Silva (C)

Department of Oncological Medicine, Gustave Roussy, Villejuif, France.

E Colomba (E)

Department of Oncological Medicine, Gustave Roussy, Villejuif, France.

L Carril (L)

Department of Oncological Medicine, Gustave Roussy, Villejuif, France.

L Derosa (L)

Department of Oncological Medicine, Gustave Roussy, Villejuif, France.

R Flippot (R)

Department of Oncological Medicine, Gustave Roussy, Villejuif, France.

O Mir (O)

Department of Oncological Medicine, Gustave Roussy, Villejuif, France.

N Khoudour (N)

Functional Unity (UF) Drug Biology and Toxicology Department, Hopital Cochin, Paris, France.

B Blanchet (B)

Functional Unity (UF) Drug Biology and Toxicology Department, Hopital Cochin, Paris, France; UMR8038 CNRS, U1268 INSERM, Faculty of Pharmacy, University of Paris, Paris, France.

B Escudier (B)

Department of Oncological Medicine, Gustave Roussy, Villejuif, France.

A Paci (A)

Department of Medical Biology and Pathology, Gustave Roussy, Villejuif, France; Medical School, University of Paris XI Sacly, Saclay, France.

L Albiges (L)

Department of Oncological Medicine, Gustave Roussy, Villejuif, France; Medical School, University of Paris XI Sacly, Saclay, France. Electronic address: Laurence.albiges@gustaveroussy.fr.

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Classifications MeSH