A high-protein total diet replacement alters the regulation of food intake and energy homeostasis in healthy, normal-weight adults.


Journal

European journal of nutrition
ISSN: 1436-6215
Titre abrégé: Eur J Nutr
Pays: Germany
ID NLM: 100888704

Informations de publication

Date de publication:
Jun 2022
Historique:
received: 26 02 2021
accepted: 16 11 2021
pubmed: 21 12 2021
medline: 18 5 2022
entrez: 20 12 2021
Statut: ppublish

Résumé

Dietary intake can affect energy homeostasis and influence body weight control. The aim of this study was to compare the impact of high-protein total diet replacement (HP-TDR) versus a control (CON) diet in the regulation of food intake and energy homeostasis in healthy, normal-weight adults. In this acute randomized controlled, cross-over study, participants completed two isocaloric arms: a) HP-TDR: 35% carbohydrate, 40% protein, and 25% fat; b) CON: 55% carbohydrate, 15% protein, and 30% fat. The diets were provided for 32 h while inside a whole-body calorimetry unit. Appetite sensations, appetite-related hormones, and energy metabolism were assessed. Forty-three healthy, normal-weight adults (19 females) participated. Appetite sensations did not differ between diets (all p > 0.05). Compared to the CON diet, the change in fasting blood markers during the HP-TDR intervention was smaller for peptide tyrosine-tyrosine (PYY; - 18.9 ± 7.9 pg/mL, p = 0.02) and greater for leptin (1859 ± 652 pg/mL, p = 0.007). Moreover, postprandial levels of glucagon-like peptide 1 (1.62 ± 0.36 pM, p < 0.001) and PYY (31.37 ± 8.05 pg/mL, p < 0.001) were higher in the HP-TDR. Significant correlations were observed between energy balance and satiety (r = - 0.41, p = 0.007), and energy balance and PFC (r = 0.33, p = 0.033) in the HP-TDR. Compared to the CON diet, the HP-TDR increased blood levels of anorexigenic hormones. Moreover, females and males responded differently to the intervention in terms of appetite sensations and appetite-related hormones. NCT02811276 (retrospectively registered on 16 June 2016) and NCT03565510 (retrospectively registered on 11 June 2018).

Identifiants

pubmed: 34928408
doi: 10.1007/s00394-021-02747-1
pii: 10.1007/s00394-021-02747-1
pmc: PMC9106637
doi:

Substances chimiques

Carbohydrates 0
Ghrelin 0
Peptide YY 106388-42-5

Banques de données

ClinicalTrials.gov
['NCT02811276', 'NCT03565510']

Types de publication

Journal Article Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

1849-1861

Informations de copyright

© 2021. The Author(s).

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Auteurs

Camila L P Oliveira (CLP)

Human Nutrition Research Unit, Department of Agricultural, Food and Nutritional Science, University of Alberta, Edmonton, AB, Canada.
Alberta Diabetes Institute, University of Alberta, Edmonton, AB, Canada.

Normand G Boulé (NG)

Alberta Diabetes Institute, University of Alberta, Edmonton, AB, Canada.
Faculty of Kinesiology, Sport, and Recreation, University of Alberta, Edmonton, AB, Canada.

Sarah A Elliott (SA)

Human Nutrition Research Unit, Department of Agricultural, Food and Nutritional Science, University of Alberta, Edmonton, AB, Canada.
Alberta Research Centre for Health Evidence, University of Alberta, Edmonton, AB, Canada.

Arya M Sharma (AM)

Division of Endocrinology and Metabolism, Department of Medicine, University of Alberta, Edmonton, AB, Canada.

Mario Siervo (M)

School of Life Sciences, Division of Physiology, Pharmacology and Neuroscience, University of Nottingham, Nottingham, England, UK.

Aloys Berg (A)

Faculty of Medicine, University of Freiburg, Freiburg, Germany.

Sunita Ghosh (S)

Department of Medical Oncology, University of Alberta, Edmonton, AB, Canada.

Carla M Prado (CM)

Human Nutrition Research Unit, Department of Agricultural, Food and Nutritional Science, University of Alberta, Edmonton, AB, Canada. cprado@ualberta.ca.
Alberta Diabetes Institute, University of Alberta, Edmonton, AB, Canada. cprado@ualberta.ca.

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