The proprotein convertase furin in cancer: more than an oncogene.


Journal

Oncogene
ISSN: 1476-5594
Titre abrégé: Oncogene
Pays: England
ID NLM: 8711562

Informations de publication

Date de publication:
02 2022
Historique:
received: 31 08 2021
accepted: 30 12 2021
revised: 13 12 2021
pubmed: 9 1 2022
medline: 11 3 2022
entrez: 8 1 2022
Statut: ppublish

Résumé

Furin is the first discovered proprotein convertase member and is present in almost all mammalian cells. Therefore, by regulating the maturation of a wide range of proproteins, Furin expression and/or activity is involved in various physiological and pathophysiological processes ranging from embryonic development to carcinogenesis. Since many of these protein precursors are involved in initiating and maintaining the hallmarks of cancer, Furin has been proposed as a potential target for treating several human cancers. In contrast, other studies have revealed that some types of cancer do not benefit from Furin inhibition. Therefore, understanding the heterogeneous functions of Furin in cancer will provide important insights into the design of effective strategies targeting Furin in cancer treatment. Here, we present recent advances in understanding how Furin expression and activity are regulated in cancer cells and their influences on the activity of Furin substrates in carcinogenesis. Furthermore, we discuss how Furin represses tumorigenic properties of several cancer cells and why Furin inhibition leads to aggressive phenotypes in other tumors. Finally, we summarize the clinical applications of Furin inhibition in treating human cancers.

Identifiants

pubmed: 34997216
doi: 10.1038/s41388-021-02175-9
pii: 10.1038/s41388-021-02175-9
doi:

Substances chimiques

FURIN protein, human EC 3.4.21.75
Furin EC 3.4.21.75

Types de publication

Journal Article Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

1252-1262

Informations de copyright

© 2022. The Author(s), under exclusive licence to Springer Nature Limited.

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Auteurs

Zongsheng He (Z)

Department of Gastroenterology, Daping Hospital, Army Medical University, Chongqing, China.
Laboratory of Biochemical Neuroendocrinology, Department of Human Genetics, KU Leuven, Leuven, Belgium.

Abdel-Majid Khatib (AM)

INSERM, LAMC, UMR 1029, Allée Geoffroy St Hilaire, Pessac, France. majid.khatib@inserm.fr.
Institut Bergoinié, Bordeaux, France. majid.khatib@inserm.fr.

John W M Creemers (JWM)

Laboratory of Biochemical Neuroendocrinology, Department of Human Genetics, KU Leuven, Leuven, Belgium. john.creemers@kuleuven.be.

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