Risk factors for developing pacing induced LV dysfunction: Experience from a tertiary center in the UK.


Journal

Pacing and clinical electrophysiology : PACE
ISSN: 1540-8159
Titre abrégé: Pacing Clin Electrophysiol
Pays: United States
ID NLM: 7803944

Informations de publication

Date de publication:
Mar 2022
Historique:
revised: 04 12 2021
received: 03 10 2021
accepted: 02 01 2022
pubmed: 14 1 2022
medline: 22 4 2022
entrez: 13 1 2022
Statut: ppublish

Résumé

The risk factors for developing pacing induced left ventricular dysfunction (LVD) in patients with high burden of right ventricular pacing (RVP) is poorly understood. Therefore, in the present study, we aimed to assess the determinants of pacing induced LVD. Our data were retrospectively collected from 146 patients with RVP > 40% who underwent generator change (GC) or cardiac resynchronization therapy (CRT) upgrade between 2016 and 2019 who had left ventricular ejection fraction (EF) ≥50% at initial implant. A total of 75 patients had CRT upgrade due to pacing induced LVD (EF < 50%) and 71 patients with preserved LV function (EF ≥ 50%) had a GC. Primary indication for pacing in both groups was complete heart block. Male predominance (p = .008), prior myocardial infarction (MI) (p = .001), atrial fibrillation (AF) (p = .009), chronic kidney disease (CKD) (p = .005), and borderline low systolic function (BLSF) (EF 50%-55%) (p = .04) were more prevalent in the CRT upgrade group. Presence of AF (odds ratio [OR] = 3.05, 95% confidence interval [CI] 1.42-6.58; p = .004), BLSF (OR = 3.8, 95% CI 1.22-11.8; p = .02), and male gender (OR = 2.41, 95% CI 1.14-5.08; p = .02) were independent predictors for RVP induced LVD. Age (OR = 1.08, 95% CI 1.02-1.14; p = .005) and BLSF (OR = 5.33, 95% CI 1.26-22.5; p = .023) were independent predictors of earlier development of LVD after implant. Our results suggested that AF, BLSF, and male gender are predictors for development of pacing induced LVD in patients with high RVP burden. LVD can occur at any time after pacemaker implant with BLSF and increasing age associated with earlier development of LVD.

Sections du résumé

BACKGROUND BACKGROUND
The risk factors for developing pacing induced left ventricular dysfunction (LVD) in patients with high burden of right ventricular pacing (RVP) is poorly understood. Therefore, in the present study, we aimed to assess the determinants of pacing induced LVD.
METHODS METHODS
Our data were retrospectively collected from 146 patients with RVP > 40% who underwent generator change (GC) or cardiac resynchronization therapy (CRT) upgrade between 2016 and 2019 who had left ventricular ejection fraction (EF) ≥50% at initial implant.
RESULTS RESULTS
A total of 75 patients had CRT upgrade due to pacing induced LVD (EF < 50%) and 71 patients with preserved LV function (EF ≥ 50%) had a GC. Primary indication for pacing in both groups was complete heart block. Male predominance (p = .008), prior myocardial infarction (MI) (p = .001), atrial fibrillation (AF) (p = .009), chronic kidney disease (CKD) (p = .005), and borderline low systolic function (BLSF) (EF 50%-55%) (p = .04) were more prevalent in the CRT upgrade group. Presence of AF (odds ratio [OR] = 3.05, 95% confidence interval [CI] 1.42-6.58; p = .004), BLSF (OR = 3.8, 95% CI 1.22-11.8; p = .02), and male gender (OR = 2.41, 95% CI 1.14-5.08; p = .02) were independent predictors for RVP induced LVD. Age (OR = 1.08, 95% CI 1.02-1.14; p = .005) and BLSF (OR = 5.33, 95% CI 1.26-22.5; p = .023) were independent predictors of earlier development of LVD after implant.
CONCLUSIONS CONCLUSIONS
Our results suggested that AF, BLSF, and male gender are predictors for development of pacing induced LVD in patients with high RVP burden. LVD can occur at any time after pacemaker implant with BLSF and increasing age associated with earlier development of LVD.

Identifiants

pubmed: 35023176
doi: 10.1111/pace.14442
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

365-373

Informations de copyright

© 2022 Wiley Periodicals LLC.

Références

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Auteurs

Vijayabharathy Kanthasamy (V)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

Nikolaos Papageorgiou (N)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.
Institute of Cardiovascular Science, University College London, London, UK.

Tomi Bajomo (T)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

Christopher Monkhouse (C)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

Antonio Creta (A)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

Malcolm Finlay (M)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

Pier D Lambiase (PD)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.
Institute of Cardiovascular Science, University College London, London, UK.

Phil Moore (P)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

Simon Sporton (S)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

Mark J Earley (MJ)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

Richard J Schilling (RJ)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

Carl Hayward (C)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

Rui Providência (R)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

Ross J Hunter (RJ)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

Anthony W Chow (AW)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

Amal Muthumala (A)

Barts Heart Centre, St Bartholomew's Hospital, West Smithfield, London, UK.

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