Myocardial oedema: pathophysiological basis and implications for the failing heart.
Cardiac lymphatics
Cardiac microcirculation
Cardiac pericytes
Extracellular matrix
Heart failure
Myocardial interstitium
Myocardial oedema
Journal
ESC heart failure
ISSN: 2055-5822
Titre abrégé: ESC Heart Fail
Pays: England
ID NLM: 101669191
Informations de publication
Date de publication:
04 2022
04 2022
Historique:
revised:
27
10
2021
received:
12
07
2021
accepted:
02
12
2021
pubmed:
13
2
2022
medline:
26
3
2022
entrez:
12
2
2022
Statut:
ppublish
Résumé
Myocardial fluid homeostasis relies on a complex interplay between microvascular filtration, interstitial hydration, cardiomyocyte water uptake and lymphatic removal. Dysregulation of one or more of these mechanisms may result in myocardial oedema. Interstitial and intracellular fluid accumulation disrupts myocardial architecture, intercellular communication, and metabolic pathways, decreasing contractility and increasing myocardial stiffness. The widespread use of cardiac magnetic resonance enabled the identification of myocardial oedema as a clinically relevant imaging finding with prognostic implications in several types of heart failure. Furthermore, growing experimental evidence has contributed to a better understanding of the physical and molecular interactions in the microvascular barrier, myocardial interstitium and lymphatics and how they might be disrupted in heart failure. In this review, we summarize current knowledge on the factors controlling myocardial water balance in the healthy and failing heart and pinpoint the new potential therapeutic avenues.
Identifiants
pubmed: 35150087
doi: 10.1002/ehf2.13775
pmc: PMC8934951
doi:
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
958-976Informations de copyright
© 2022 The Authors. ESC Heart Failure published by John Wiley & Sons Ltd on behalf of European Society of Cardiology.
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