Targeting Hemoglobin to Reduce Delayed Cerebral Ischemia After Subarachnoid Hemorrhage.
Cerebrospinal fluid filtration
Delayed cerebral ischemia
Subarachnoid hemorrhage
Vasospasm
Journal
Translational stroke research
ISSN: 1868-601X
Titre abrégé: Transl Stroke Res
Pays: United States
ID NLM: 101517297
Informations de publication
Date de publication:
10 2022
10 2022
Historique:
received:
03
12
2021
accepted:
01
02
2022
revised:
28
01
2022
pubmed:
15
2
2022
medline:
24
8
2022
entrez:
14
2
2022
Statut:
ppublish
Résumé
Delayed cerebral ischemia (DCI) continues to be a sequela of aneurysmal subarachnoid hemorrhage (aSAH) that carries significant morbidity and mortality. Aside from nimodipine, no therapeutic agents are available to reduce the incidence of DCI. Pathophysiologic mechanisms contributing to DCI are poorly understood, but accumulating evidence over the years implicates several factors. Those have included microvessel vasoconstriction, microthrombosis, oxidative tissue damage, and cortical spreading depolarization as well as large vessel vasospasm. Common to these processes is red blood cell leakage into the cerebrospinal fluids (CSF) and subsequent lysis which releases hemoglobin, a central instigator in these events. This has led to the hypothesis that early blood removal may improve clinical outcome and reduce DCI. This paper will provide a narrative review of the evidence of hemoglobin as an instigator of DCI. It will also elaborate on available human data that discuss blood clearance and CSF drainage as a treatment of DCI. Finally, we will address a recent novel device that is currently being tested, the Neurapheresis CSF Management System™. This is an automated dual-lumen lumbar drainage system that has an option to filter CSF and return it to the patient.
Identifiants
pubmed: 35157256
doi: 10.1007/s12975-022-00995-9
pii: 10.1007/s12975-022-00995-9
pmc: PMC9375776
mid: NIHMS1782685
doi:
Substances chimiques
Hemoglobins
0
Types de publication
Journal Article
Review
Research Support, Non-U.S. Gov't
Research Support, N.I.H., Extramural
Langues
eng
Sous-ensembles de citation
IM
Pagination
725-735Subventions
Organisme : NINDS NIH HHS
ID : K23 NS106054
Pays : United States
Organisme : NINDS NIH HHS
ID : R01 NS115887
Pays : United States
Organisme : NINDS NIH HHS
ID : R01 NS110779
Pays : United States
Organisme : NINDS NIH HHS
ID : U01 NS100699
Pays : United States
Organisme : NINDS NIH HHS
ID : R44 NS110247
Pays : United States
Organisme : NINDS NIH HHS
ID : U01 NS110772
Pays : United States
Organisme : NINDS NIH HHS
ID : R01 NS121339
Pays : United States
Informations de copyright
© 2022. The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature.
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