ExoU Induces Lung Endothelial Cell Damage and Activates Pro-Inflammatory Caspase-1 during
Bacterial Proteins
/ toxicity
Caspase 1
/ drug effects
Cell Death
/ drug effects
Chemical and Drug Induced Liver Injury
/ physiopathology
Endothelial Cells
/ drug effects
Genetic Variation
Genotype
Humans
Inflammation
/ chemically induced
Pseudomonas Infections
/ genetics
Pseudomonas aeruginosa
/ genetics
ExoU
Pseudomonas aeruginosa
caspase-1
mitochondria
pneumonia
pulmonary endothelial cells
reactive oxygen species
sepsis
stress responses
Journal
Toxins
ISSN: 2072-6651
Titre abrégé: Toxins (Basel)
Pays: Switzerland
ID NLM: 101530765
Informations de publication
Date de publication:
18 02 2022
18 02 2022
Historique:
received:
13
01
2022
revised:
07
02
2022
accepted:
11
02
2022
entrez:
24
2
2022
pubmed:
25
2
2022
medline:
19
3
2022
Statut:
epublish
Résumé
The Gram-negative, opportunistic pathogen
Identifiants
pubmed: 35202178
pii: toxins14020152
doi: 10.3390/toxins14020152
pmc: PMC8878379
pii:
doi:
Substances chimiques
Bacterial Proteins
0
Caspase 1
EC 3.4.22.36
Types de publication
Comparative Study
Journal Article
Research Support, N.I.H., Extramural
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : NHLBI NIH HHS
ID : P01 HL066299
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL118334
Pays : United States
Organisme : NIH HHS
ID : R01 OD010944
Pays : United States
Organisme : NIH HHS
ID : S10 OD025089
Pays : United States
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