In silico evolution of nucleic acid-binding proteins from a nonfunctional scaffold.


Journal

Nature chemical biology
ISSN: 1552-4469
Titre abrégé: Nat Chem Biol
Pays: United States
ID NLM: 101231976

Informations de publication

Date de publication:
04 2022
Historique:
received: 30 03 2021
accepted: 04 01 2022
pubmed: 26 2 2022
medline: 27 4 2022
entrez: 25 2 2022
Statut: ppublish

Résumé

Directed evolution emulates the process of natural selection to produce proteins with improved or altered functions. These approaches have proven to be very powerful but are technically challenging and particularly time and resource intensive. To bypass these limitations, we constructed a system to perform the entire process of directed evolution in silico. We employed iterative computational cycles of mutation and evaluation to predict mutations that confer high-affinity binding activities for DNA and RNA to an initial de novo designed protein with no inherent function. Beneficial mutations revealed modes of nucleic acid recognition not previously observed in natural proteins, highlighting the ability of computational directed evolution to access new molecular functions. Furthermore, the process by which new functions were obtained closely resembles natural evolution and can provide insights into the contributions of mutation rate, population size and selective pressure on functionalization of macromolecules in nature.

Identifiants

pubmed: 35210620
doi: 10.1038/s41589-022-00967-y
pii: 10.1038/s41589-022-00967-y
doi:

Substances chimiques

Nucleic Acids 0
Proteins 0
RNA 63231-63-0
DNA 9007-49-2

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

403-411

Informations de copyright

© 2022. The Author(s), under exclusive licence to Springer Nature America, Inc.

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Auteurs

Samuel A Raven (SA)

Harry Perkins Institute of Medical Research, Nedlands, Western Australia, Australia.
University of Western Australia Centre for Medical Research, Nedlands, Western Australia, Australia.

Blake Payne (B)

Harry Perkins Institute of Medical Research, Nedlands, Western Australia, Australia.
University of Western Australia Centre for Medical Research, Nedlands, Western Australia, Australia.

Mitchell Bruce (M)

Curtin Medical School, Curtin University, Bentley, Western Australia, Australia.

Aleksandra Filipovska (A)

Harry Perkins Institute of Medical Research, Nedlands, Western Australia, Australia.
University of Western Australia Centre for Medical Research, Nedlands, Western Australia, Australia.
School of Molecular Sciences, The University of Western Australia, Crawley, Western Australia, Australia.
Telethon Kids Institute, Northern Entrance, Perth Children's Hospital, Nedlands, Western Australia, Australia.

Oliver Rackham (O)

Harry Perkins Institute of Medical Research, Nedlands, Western Australia, Australia. oliver.rackham@curtin.edu.au.
Curtin Medical School, Curtin University, Bentley, Western Australia, Australia. oliver.rackham@curtin.edu.au.
Telethon Kids Institute, Northern Entrance, Perth Children's Hospital, Nedlands, Western Australia, Australia. oliver.rackham@curtin.edu.au.
Curtin Health Innovation Research Institute, Curtin University, Bentley, Western Australia, Australia. oliver.rackham@curtin.edu.au.

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