Resolving inflammatory links between myocardial infarction and vascular dementia.
Myocardial infarction
Vascular dementia
Journal
Seminars in immunology
ISSN: 1096-3618
Titre abrégé: Semin Immunol
Pays: England
ID NLM: 9009458
Informations de publication
Date de publication:
01 2022
01 2022
Historique:
received:
22
10
2021
revised:
07
02
2022
accepted:
15
02
2022
pubmed:
2
3
2022
medline:
17
12
2022
entrez:
1
3
2022
Statut:
ppublish
Résumé
Myocardial infarction is associated with increased risk for vascular dementia. In both myocardial infarction and vascular dementia, there is evidence that elevated inflammatory biomarkers are associated with worsened clinical outcomes. Myocardial infarction leads to a systemic inflammatory response, which may contribute to recruitment or activation of myeloid cells, including monocytes, microglia, and perivascular macrophages, within the central nervous system. However, our understanding of the causative roles for these cells linking cardiac injury to the development and progression of dementia is incomplete. Herein, we provide an overview of inflammatory cellular and molecular links between myocardial infarction and vascular dementia and discuss strategies to resolve inflammation after myocardial infarction to limit neurovascular injury.
Identifiants
pubmed: 35227567
pii: S1044-5323(22)00017-3
doi: 10.1016/j.smim.2022.101600
pmc: PMC10234261
mid: NIHMS1784249
pii:
doi:
Types de publication
Journal Article
Review
Research Support, N.I.H., Extramural
Langues
eng
Sous-ensembles de citation
IM
Pagination
101600Subventions
Organisme : NHLBI NIH HHS
ID : R01 HL122309
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL159964
Pays : United States
Organisme : NIA NIH HHS
ID : RF1 AG072080
Pays : United States
Informations de copyright
Copyright © 2022 Elsevier Ltd. All rights reserved.
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